Isolation of human fos-related genes and their expression during monocyte-macrophage differentiation
about
Human T-cell leukemia virus type 1 Tax activates transcription of the human fra-1 gene through multiple cis elements responsive to transmembrane signalsMultiple mitogen-activated protein kinase signaling pathways connect the cot oncoprotein to the c-jun promoter and to cellular transformationRedox activation of Fos-Jun DNA binding activity is mediated by a DNA repair enzymeTargeted disruption of the c-fos gene demonstrates c-fos-dependent and -independent pathways for gene expression stimulated by growth factors or oncogenesInteractions among LRF-1, JunB, c-Jun, and c-Fos define a regulatory program in the G1 phase of liver regenerationAlteration of a cyclic AMP-dependent protein kinase phosphorylation site in the c-Fos protein augments its transforming potentialIsolation of an AP-1 repressor by a novel method for detecting protein-protein interactionsInduction of senescence-like phenotypes by forced expression of hic-5, which encodes a novel LIM motif protein, in immortalized human fibroblastsInduction of gene expression via activator protein-1 in the ascorbate protection against UV-induced damageTissue-specific transgenic knockdown of Fos-related antigen 2 (Fra-2) expression mediated by dominant negative Fra-2The Mos/MAP kinase pathway stabilizes c-Fos by phosphorylation and augments its transforming activity in NIH 3T3 cellsIdentification of LRF-1, a leucine-zipper protein that is rapidly and highly induced in regenerating liverLigand-specific sequential regulation of transcription factors for differentiation of MCF-7 cellsThe immediate-early growth response in regenerating liver and insulin-stimulated H-35 cells: comparison with serum-stimulated 3T3 cells and identification of 41 novel immediate-early genes.A network of mitogen-activated protein kinases links G protein-coupled receptors to the c-jun promoter: a role for c-Jun NH2-terminal kinase, p38s, and extracellular signal-regulated kinase 5.Cot, a novel kinase of histone H3, induces cellular transformation through up-regulation of c-fos transcriptional activity.miR-597 inhibits breast cancer cell proliferation, migration and invasion through FOSL2.Antitumor promotion by phenolic antioxidants: inhibition of AP-1 activity through induction of Fra expression.Structure and mapping of the fosB gene. FosB downregulates the activity of the fosB promoter12-O-Tetradecanoylphorbol-13-Acetate Induces Up-Regulated Transcription of Variant 1 but Not Variant 2 of VIL2 in Esophageal Squamous Cell Carcinoma Cells via ERK1/2/AP-1/Sp1 SignalingPromoter-level expression clustering identifies time development of transcriptional regulatory cascades initiated by ErbB receptors in breast cancer cells.Quantitative Expression and Co-Localization of Wnt Signalling Related Proteins in Feline Squamous Cell Carcinoma.Jun is phosphorylated by several protein kinases at the same sites that are modified in serum-stimulated fibroblastsTwo cellular proteins bind specifically to a purine-rich sequence necessary for the destabilization function of a c-fos protein-coding region determinant of mRNA instability.Revertants of v-fos-transformed rat fibroblasts: suppression of transformation is dominant.Signal transduction and molecular targets of selected flavonoids.Induction of c-jun protooncogene expression and transcription factor AP-1 activity by the polyoma virus middle-sized tumor antigen.Sp1 and AP-1 regulate expression of the human gene VIL2 in esophageal carcinoma cells.Evaluation of potential regulatory function of breast cancer risk locus at 6q25.1.Growth and differentiation of embryonic stem cells that lack an intact c-fos gene.An alternative spliced form of FosB is a negative regulator of transcriptional activation and transformation by Fos proteinsAnalysis of AP-1 function in cellular transformation pathways.An autonomous N-terminal transactivation domain in Fos protein plays a crucial role in transformation.Analysis of fra-2 gene expression.The NFAT-1 DNA binding complex in activated T cells contains Fra-1 and JunB.Identification and characterization of Ref-1, a nuclear protein that facilitates AP-1 DNA-binding activity.Both products of the fosB gene, FosB and its short form, FosB/SF, are transcriptional activators in fibroblasts.c-Jun Is Required for Nuclear Factor-κB-Dependent, LPS-Stimulated Fos-Related Antigen-1 Transcription in Alveolar Macrophages.Loss of JunB activity enhances stromelysin 1 expression in a model of the epithelial-to-mesenchymal transition of mouse skin tumors.Fra-1 governs cell migration via modulation of CD44 expression in human mesotheliomas.
P2860
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P2860
Isolation of human fos-related genes and their expression during monocyte-macrophage differentiation
description
1990 nî lūn-bûn
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1990 թուականի Մարտին հրատարակուած գիտական յօդուած
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1990 թվականի մարտին հրատարակված գիտական հոդված
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1990年の論文
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1990年学术文章
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1990年学术文章
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1990年学术文章
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1990年学术文章
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1990年学术文章
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1990年學術文章
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name
Isolation of human fos-related ...... yte-macrophage differentiation
@ast
Isolation of human fos-related ...... yte-macrophage differentiation
@en
Isolation of human fos-related ...... yte-macrophage differentiation
@en-gb
Isolation of human fos-related ...... yte-macrophage differentiation
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type
label
Isolation of human fos-related ...... yte-macrophage differentiation
@ast
Isolation of human fos-related ...... yte-macrophage differentiation
@en
Isolation of human fos-related ...... yte-macrophage differentiation
@en-gb
Isolation of human fos-related ...... yte-macrophage differentiation
@nl
prefLabel
Isolation of human fos-related ...... yte-macrophage differentiation
@ast
Isolation of human fos-related ...... yte-macrophage differentiation
@en
Isolation of human fos-related ...... yte-macrophage differentiation
@en-gb
Isolation of human fos-related ...... yte-macrophage differentiation
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Isolation of human fos-related ...... yte-macrophage differentiation
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1990-03-01T00:00:00Z