Pro-inflammatory effect of TWEAK/Fn14 interaction on human umbilical vein endothelial cells
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TWEAK binding to the Fn14 cysteine-rich domain depends on charged residues located in both the A1 and D2 modulesConcerted action of Nrf2-ARE pathway, MRN complex, HMGB1 and inflammatory cytokines - implication in modification of radiation damageTWEAK promotes peritoneal inflammationCrystal Structure of Human TWEAK in Complex with the Fab Fragment of a Neutralizing Antibody Reveals Insights into Receptor BindingThe tumor necrosis factor-like weak inducer of apoptosis (TWEAK)-fibroblast growth factor-inducible 14 (Fn14) signaling system regulates glioma cell survival via NFkappaB pathway activation and BCL-XL/BCL-W expressionTWEAK activates the non-canonical NFkappaB pathway in murine renal tubular cells: modulation of CCL21BAFF, APRIL, TWEAK, BCMA, TACI and Fn14 proteins are related to human glioma tumor grade: immunohistochemistry and public microarray data meta-analysis.TWEAK induces NF-kappaB2 p100 processing and long lasting NF-kappaB activation.TWEAK mediates inflammation in experimental atopic dermatitis and psoriasis.The interaction between tumor necrosis factor-like weak inducer of apoptosis and its receptor fibroblast growth factor-inducible 14 promotes the recruitment of neutrophils into the ischemic brain.TWEAK affects keratinocyte G2/M growth arrest and induces apoptosis through the translocation of the AIF protein to the nucleus.Regulation of tumor necrosis factor-like weak inducer of apoptosis receptor protein (TWEAKR) expression by Kaposi's sarcoma-associated herpesvirus microRNA prevents TWEAK-induced apoptosis and inflammatory cytokine expression.Tumor necrosis factor-like weak inducer of apoptosis and fibroblast growth factor-inducible 14 mediate cerebral ischemia-induced poly(ADP-ribose) polymerase-1 activation and neuronal death.Structural basis and targeting of the interaction between fibroblast growth factor-inducible 14 and tumor necrosis factor-like weak inducer of apoptosisTumor necrosis factor-like weak inducer of apoptosis or Fn14 deficiency reduce elastase perfusion-induced aortic abdominal aneurysm in mice.A soluble Fn14-Fc decoy receptor reduces infarct volume in a murine model of cerebral ischemia.TWEAK and Fn14 expression in the pathogenesis of joint inflammation and bone erosion in rheumatoid arthritis.The tumor necrosis factor superfamily of cytokines in the inflammatory myopathies: potential targets for therapy.TWEAK/Fn14 interaction regulates RANTES production, BMP-2-induced differentiation, and RANKL expression in mouse osteoblastic MC3T3-E1 cellsThe cytokine tumor necrosis factor-like weak inducer of apoptosis and its receptor fibroblast growth factor-inducible 14 have a neuroprotective effect in the central nervous system.Tweak induces proliferation in renal tubular epithelium: a role in uninephrectomy induced renal hyperplasiaBlocking TWEAK-Fn14 interaction inhibits hematopoietic stem cell transplantation-induced intestinal cell death and reduces GVHD.Therapeutic potential of the TWEAK/Fn14 pathway in intractable gastrointestinal cancer.Expression of TweakR in breast cancer and preclinical activity of enavatuzumab, a humanized anti-TweakR mAb.The cytokine TWEAK modulates renal tubulointerstitial inflammation.Amelioration of autoimmune neuroinflammation by the fusion molecule Fn14·TRAILFn14-TRAIL, a chimeric intercellular signal exchanger, attenuates experimental autoimmune encephalomyelitis.The TWEAK-Fn14 cytokine-receptor axis: discovery, biology and therapeutic targetingTWEAK and Fn14 in the Neurovascular Unit.Soluble TWEAK plasma levels as a novel biomarker of endothelial function in patients with chronic kidney disease.Urinary TWEAK as a biomarker of lupus nephritis: a multicenter cohort study.TWEAK/Fn14 Axis: A Promising Target for the Treatment of Cardiovascular Diseases.TWEAK activation of the non-canonical NF-κB signaling pathway differentially regulates melanoma and prostate cancer cell invasion.Genetic deletion or TWEAK blocking antibody administration reduce atherosclerosis and enhance plaque stability in mice.Tumor necrosis factor-like weak inducer of apoptosis and its potential roles in lupus nephritis.Role of TWEAK in lupus nephritis: a bench-to-bedside review.A further TWEAK to multiple sclerosis pathophysiology.TWEAK: A New Player in Obesity and Diabetes.Role of strain differences on host resistance and the transcriptional response of macrophages to infection with Yersinia enterocolitica.The role of tumor necrosis factor-like weak inducer of apoptosis in atherosclerosis via its two different receptors.
P2860
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P2860
Pro-inflammatory effect of TWEAK/Fn14 interaction on human umbilical vein endothelial cells
description
2002 nî lūn-bûn
@nan
2002 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Pro-inflammatory effect of TWE ...... bilical vein endothelial cells
@ast
Pro-inflammatory effect of TWE ...... bilical vein endothelial cells
@en
Pro-inflammatory effect of TWE ...... bilical vein endothelial cells
@nl
type
label
Pro-inflammatory effect of TWE ...... bilical vein endothelial cells
@ast
Pro-inflammatory effect of TWE ...... bilical vein endothelial cells
@en
Pro-inflammatory effect of TWE ...... bilical vein endothelial cells
@nl
prefLabel
Pro-inflammatory effect of TWE ...... bilical vein endothelial cells
@ast
Pro-inflammatory effect of TWE ...... bilical vein endothelial cells
@en
Pro-inflammatory effect of TWE ...... bilical vein endothelial cells
@nl
P2093
P1476
Pro-inflammatory effect of TWE ...... bilical vein endothelial cells
@en
P2093
Hideo Yagita
Ko Okumura
Masafumi Nakayama
Yoshinosuke Fukuchi
P304
P356
10.1016/S0006-291X(02)02670-0
P407
P577
2002-12-01T00:00:00Z