HIV infection is active and progressive in lymphoid tissue during the clinically latent stage of disease
about
CD4+ T cell depletion during all stages of HIV disease occurs predominantly in the gastrointestinal tractIncreased mortality and AIDS-like immunopathology in wild chimpanzees infected with SIVcpzCharacterization of a family of related cellular transcription factors which can modulate human immunodeficiency virus type 1 transcription in vitroAbsolute dependence on kappa B responsive elements for initiation and Tat-mediated amplification of HIV transcription in blood CD4 T lymphocytesHuman transcription factor YY1 represses human immunodeficiency virus type 1 transcription and virion productionTemporal association of cellular immune responses with the initial control of viremia in primary human immunodeficiency virus type 1 syndromeHuman immunodeficiency virus type 1 Tat upregulates interleukin-2 secretion in activated T cellsHIV type-1 infection of the cotton rat (Sigmodon fulviventer and S. hispidus)Regulation of human immunodeficiency virus type 1 and cytokine gene expression in myeloid cells by NF-kappa B/Rel transcription factorsNuclear localization signal of HIV-1 as a novel target for therapeutic interventionActivation of transcription factor NF-kappaB by the Tat protein of human immunodeficiency virus type 1Human herpesvirus 6Human immunodeficiency virus type 1 Vpr arrests the cell cycle in G2 by inhibiting the activation of p34cdc2-cyclin BApoptosis of uninfected cells induced by HIV envelope glycoproteinsAcute-phase protein α1-anti-trypsin: diverting injurious innate and adaptive immune responses from non-authentic threatsThe immunological synapse: the gateway to the HIV reservoirNatural SIV hosts: showing AIDS the doorAlpha-1-antitrypsin interacts with gp41 to block HIV-1 entry into CD4+ T lymphocytes.Release of virus from lymphoid tissue affects human immunodeficiency virus type 1 and hepatitis C virus kinetics in the blood.ABT-538 is a potent inhibitor of human immunodeficiency virus protease and has high oral bioavailability in humansThe Dynamics of T and B Cells in Lymph Node during Chronic HIV Infection: TFH and HIV, Unhappy Dance Partners?Griffithsin, a potent HIV entry inhibitor, is an excellent candidate for anti-HIV microbicideDissecting How CD4 T Cells Are Lost During HIV InfectionCharacterizing follicular dendritic cells: A progress reportMultiple effects of an anti-human immunodeficiency virus nucleocapsid inhibitor on virus morphology and replication.Impaired T cell proliferation, increased soluble death-inducing receptors and activation-induced T cell death in patients undergoing haemodialysisNevirapine resistance mutations of human immunodeficiency virus type 1 selected during therapyPhosphatidylazidothymidine and phosphatidyl-ddC: assessment of uptake in mouse lymphoid tissues and antiviral activities in human immunodeficiency virus-infected cells and in Rauscher leukemia virus-infected miceSynergistic anti-human immunodeficiency virus type 1 effect of hydroxamate compounds with 2',3'-dideoxyinosine in infected resting human lymphocytesDMP 323, a nonpeptide cyclic urea inhibitor of human immunodeficiency virus (HIV) protease, specifically and persistently blocks intracellular processing of HIV gag polyproteinCompounds that target novel cellular components involved in HIV-1 transcriptionDrug resistance during indinavir therapy is caused by mutations in the protease gene and in its Gag substrate cleavage sitesNovel inhibitory effects of gamma-glutamylcysteine ethyl ester against human immunodeficiency virus type 1 production and propagation.Grifonin-1: a small HIV-1 entry inhibitor derived from the algal lectin, GriffithsinPresence of an inducible HIV-1 latent reservoir during highly active antiretroviral therapyHIV population dynamics in vivo: implications for genetic variation, pathogenesis, and therapyPreclinical pharmacokinetics and distribution to tissue of AG1343, an inhibitor of human immunodeficiency virus type 1 proteaseWhy can't cytotoxic T cells handle HIV?Phosphatidylcholine-specific phospholipase C activation is required for CCR5-dependent, NF-kB-driven CCL2 secretion elicited in response to HIV-1 gp120 in human primary macrophages.Characterization of intestinal disease associated with human immunodeficiency virus infection and response to antiretroviral therapy.
P2860
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P2860
HIV infection is active and progressive in lymphoid tissue during the clinically latent stage of disease
description
1993 nî lūn-bûn
@nan
1993 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
1993 թվականի մարտին հրատարակված գիտական հոդված
@hy
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
name
HIV infection is active and pr ...... ically latent stage of disease
@ast
HIV infection is active and pr ...... ically latent stage of disease
@en
HIV infection is active and pr ...... ically latent stage of disease
@nl
type
label
HIV infection is active and pr ...... ically latent stage of disease
@ast
HIV infection is active and pr ...... ically latent stage of disease
@en
HIV infection is active and pr ...... ically latent stage of disease
@nl
prefLabel
HIV infection is active and pr ...... ically latent stage of disease
@ast
HIV infection is active and pr ...... ically latent stage of disease
@en
HIV infection is active and pr ...... ically latent stage of disease
@nl
P2093
P3181
P356
P1433
P1476
HIV infection is active and pr ...... ically latent stage of disease
@en
P2093
C Graziosi
D P Kotler
G Pantaleo
J F Demarest
J M Orenstein
M Montroni
P2888
P3181
P356
10.1038/362355A0
P407
P577
1993-03-25T00:00:00Z
P6179
1041590783