Osteocytes use estrogen receptor alpha to respond to strain but their ERalpha content is regulated by estrogen
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The amazing osteocyteRegulation of Estrogen Receptor α Expression in the Hypothalamus by Sex Steroids: Implication in the Regulation of Energy HomeostasisLocal mechanical stimuli regulate bone formation and resorption in mice at the tissue level.Ovariectomy vs. hypofunction: their effects on rat mandibular bone.Osteocyte apoptosis and control of bone resorption following ovariectomy in miceFunctional adaptation to mechanical loading in both cortical and cancellous bone is controlled locally and is confined to the loaded bones.Osteocyte Wnt/beta-catenin signaling is required for normal bone homeostasisLoading-related regulation of gene expression in bone in the contexts of estrogen deficiency, lack of estrogen receptor alpha and disuseA review of osteocyte function and the emerging importance of sclerostinSex steroid actions in male boneLoading and skeletal development and maintenancePeripubertal estrogen levels and physical activity affect femur geometry in young adult women.Loading-related regulation of transcription factor EGR2/Krox-20 in bone cells is ERK1/2 protein-mediated and prostaglandin, Wnt signaling pathway-, and insulin-like growth factor-I axis-dependentWnt16 Is Associated with Age-Related Bone Loss and Estrogen Withdrawal in Murine Bone.Manipulation of Ovarian Function Significantly Influenced Trabecular and Cortical Bone Volume, Architecture and Density in Mice at Death.The RIP1-RIP3 Complex Mediates Osteocyte Necroptosis after Ovariectomy in Rats.Non-invasive mouse models of post-traumatic osteoarthritisEffects of 8-Prenylnaringenin and Whole-Body Vibration Therapy on a Rat Model of OsteopeniaExpression levels of estrogen receptor α mRNA in peripheral blood cells are an independent biomarker for postmenopausal osteoporosis.Vitamin K catabolite inhibition of ovariectomy-induced bone loss: structure-activity relationship considerations.Mechanical signaling for bone modeling and remodelingPhysical activity and bone development during childhood: insights from animal models.The mouse fibula as a suitable bone for the study of functional adaptation to mechanical loading.The osteocyte: an endocrine cell ... and more.Estrogen receptors' roles in the control of mechanically adaptive bone (re)modelingEstrogen receptor-β regulates mechanical signaling in primary osteoblasts.Toward mechanical systems biology in bone.Is interaction between age-dependent decline in mechanical stimulation and osteocyte-estrogen receptor levels the culprit for postmenopausal-impaired bone formation?The role of estrogen receptor α in the regulation of bone and growth plate cartilage.Treatment of post-menopausal osteoporosis: beyond bisphosphonates.Strain uses gap junctions to reverse stimulation of osteoblast proliferation by osteocytes.Regulation of ERα protein expression by 17β-estradiol in cultured neurons of hypothalamic ventromedial nucleus.The biomaterial-mediated healing of critical size bone defects in the ovariectomized rat.Mechanical loading increases detection of estrogen receptor-alpha in osteocytes and osteoblasts despite chronic energy restriction.Sclerostin serum levels in prostate cancer patients and their relationship with sex steroids.Mechanical stimulation enhanced estrogen receptor expression and callus formation in diaphyseal long bone fracture healing in ovariectomy-induced osteoporotic rats.The skeletal responsiveness to mechanical loading is enhanced in mice with a null mutation in estrogen receptor-beta.[Mechanobiology and bone metabolism: Clinical relevance for fracture treatment].Mechano-transduction in osteoblastic cells involves strain-regulated estrogen receptor alpha-mediated control of insulin-like growth factor (IGF) I receptor sensitivity to Ambient IGF, leading to phosphatidylinositol 3-kinase/AKT-dependent Wnt/LRP5Analysis of multiple bone responses to graded strains above functional levels, and to disuse, in mice in vivo show that the human Lrp5 G171V High Bone Mass mutation increases the osteogenic response to loading but that lack of Lrp5 activity reduces i
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P2860
Osteocytes use estrogen receptor alpha to respond to strain but their ERalpha content is regulated by estrogen
description
2006 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2006
@ast
im August 2006 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2006/08/01)
@sk
vědecký článek publikovaný v roce 2006
@cs
wetenschappelijk artikel (gepubliceerd op 2006/08/01)
@nl
наукова стаття, опублікована в серпні 2006
@uk
مقالة علمية (نشرت في أغسطس 2006)
@ar
name
Osteocytes use estrogen recept ...... ntent is regulated by estrogen
@ast
Osteocytes use estrogen recept ...... ntent is regulated by estrogen
@en
Osteocytes use estrogen recept ...... ntent is regulated by estrogen
@nl
type
label
Osteocytes use estrogen recept ...... ntent is regulated by estrogen
@ast
Osteocytes use estrogen recept ...... ntent is regulated by estrogen
@en
Osteocytes use estrogen recept ...... ntent is regulated by estrogen
@nl
prefLabel
Osteocytes use estrogen recept ...... ntent is regulated by estrogen
@ast
Osteocytes use estrogen recept ...... ntent is regulated by estrogen
@en
Osteocytes use estrogen recept ...... ntent is regulated by estrogen
@nl
P2093
P356
P1476
Osteocytes use estrogen recept ...... ntent is regulated by estrogen
@en
P2093
Andrew A. Pitsillides
Helen L. Jessop
Joanna S. Price
Lance L. Lanyon
Mariusz Muzylak
Roberto L. De Souza
P304
P356
10.1359/JBMR.060504
P577
2006-08-01T00:00:00Z