JunB can substitute for Jun in mouse development and cell proliferation
about
Chronic occupational exposure to arsenic induces carcinogenic gene signaling networks and neoplastic transformation in human lung epithelial cellsTargeting c-Jun and JunB proteins as potential anticancer cell therapyThe role of basic leucine zipper protein-mediated transcription in physiological and pathological myocardial hypertrophyJunB Inhibits ER Stress and Apoptosis in Pancreatic Beta CellsMice lacking JunB are osteopenic due to cell-autonomous osteoblast and osteoclast defectsDivergent functions of murine Pax3 and Pax7 in limb muscle developmentEpithelial sheet movement requires the cooperation of c-Jun and MAP3K1Cyclin A is a c-Jun target gene and is necessary for c-Jun-induced anchorage-independent growth in RAT1a cellsSuppression of AP1 transcription factor function in keratinocyte suppresses differentiationMultiple cis-elements mediate the transcriptional activation of human fra-1 by 12-O-tetradecanoylphorbol-13-acetate in bronchial epithelial cellsHelicobacter pylori and mitogen-activated protein kinases mediate activator protein-1 (AP-1) subcomponent protein expression and DNA-binding activity in gastric epithelial cells.Transcription factor AP-1 in esophageal squamous cell carcinoma: alterations in activity and expression during human Papillomavirus infection.Ras-Raf-Arf signaling critically depends on the Dmp1 transcription factor.Genetic replacement of cyclin D1 function in mouse development by cyclin D2.Molecular determinants of NOTCH4 transcription in vascular endothelium.Tumor-fibroblast interactions stimulate tumor vascularization by enhancing cytokine-driven production of MMP9 by tumor cells.Emerging roles of ATF2 and the dynamic AP1 network in cancer.Activation of AP-1 transcription factors differentiates FGF2 and vascular endothelial growth factor regulation of endothelial nitric-oxide synthase expression in placental artery endothelial cells.A novel AP-1/miR-101 regulatory feedback loop and its implication in the migration and invasion of hepatoma cellsJun is required in Isl1-expressing progenitor cells for cardiovascular developmentc-Jun/AP-1 controls liver regeneration by repressing p53/p21 and p38 MAPK activityc-Jun promotes whereas JunB inhibits epidermal neoplasiaJunB contributes to Id2 repression and the epithelial-mesenchymal transition in response to transforming growth factor-β.Interplay between TAp73 Protein and Selected Activator Protein-1 (AP-1) Family Members Promotes AP-1 Target Gene Activation and Cellular Growth.Transcription factor JunD, deprived of menin, switches from growth suppressor to growth promoter.Dietary isoflavones differentially induce gene expression changes in lymphocytes from postmenopausal women who form equol as compared with those who do not.Signal-dependent fra-2 regulation in skeletal muscle reserve and satellite cells.JUNB/AP-1 controls IFN-γ during inflammatory liver disease.The c-Jun NH2-terminal kinase is essential for epidermal growth factor expression during epidermal morphogenesis.Genes overexpressed in cerebral arteries following salt-induced hypertensive disease are regulated by angiotensin II, JunB, and CREB.MEKK1 regulates the AP-1 dimer repertoire via control of JunB transcription and Fra-2 protein stability.JUNB is a key transcriptional modulator of macrophage activationCleavage of the JunB transcription factor by caspases generates a carboxyl-terminal fragment that inhibits activator protein-1 transcriptional activity.GSK3-SCF(FBXW7) targets JunB for degradation in G2 to preserve chromatid cohesion before anaphase.AP-1 mediated relief of repressive activity of the CD30 promoter microsatellite in Hodgkin and Reed-Sternberg cells.Th2 cell-specific cytokine expression and allergen-induced airway inflammation depend on JunB.Gene and protein profiling of the response of MA-10 Leydig tumor cells to human chorionic gonadotropin.A central role for the JNK pathway in mediating the antagonistic activity of pro-inflammatory cytokines against transforming growth factor-beta-driven SMAD3/4-specific gene expression.AP1 transcription factors in epidermal differentiation and skin cancerAP1 factor inactivation in the suprabasal epidermis causes increased epidermal hyperproliferation and hyperkeratosis but reduced carcinogen-dependent tumor formation
P2860
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P2860
JunB can substitute for Jun in mouse development and cell proliferation
description
2002 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
articolo scientifico
@it
artículu científicu espublizáu en 2002
@ast
im Februar 2002 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2002/02/01)
@sk
vědecký článek publikovaný v roce 2002
@cs
wetenschappelijk artikel (gepubliceerd op 2002/02/01)
@nl
наукова стаття, опублікована в лютому 2002
@uk
name
JunB can substitute for Jun in mouse development and cell proliferation
@ast
JunB can substitute for Jun in mouse development and cell proliferation
@en
JunB can substitute for Jun in mouse development and cell proliferation
@nl
type
label
JunB can substitute for Jun in mouse development and cell proliferation
@ast
JunB can substitute for Jun in mouse development and cell proliferation
@en
JunB can substitute for Jun in mouse development and cell proliferation
@nl
prefLabel
JunB can substitute for Jun in mouse development and cell proliferation
@ast
JunB can substitute for Jun in mouse development and cell proliferation
@en
JunB can substitute for Jun in mouse development and cell proliferation
@nl
P50
P356
P1433
P1476
JunB can substitute for Jun in mouse development and cell proliferation
@en
P2093
Emmanuelle Passegué
Wolfram Jochum
P2888
P304
P356
10.1038/NG790
P407
P577
2002-02-01T00:00:00Z