Mitochondrial oxidative stress and epilepsy in SOD2 deficient mice: attenuation by a lipophilic metalloporphyrin.
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Mitochondrial involvement and oxidative stress in temporal lobe epilepsyTemporal and spatial increase of reactive nitrogen species in the kainate model of temporal lobe epilepsy.Exploratory Metabolomics Profiling in the Kainic Acid Rat Model Reveals Depletion of 25-Hydroxyvitamin D3 during Epileptogenesis.Seizure-induced oxidative stress in temporal lobe epilepsyParadoxical Roles of Antioxidant Enzymes: Basic Mechanisms and Health ImplicationsPost-translational oxidative modification and inactivation of mitochondrial complex I in epileptogenesisMitochondrial respiration deficits driven by reactive oxygen species in experimental temporal lobe epilepsy.Reactive oxygen species mediate cognitive deficits in experimental temporal lobe epilepsy.SIRT5 Deficiency Enhances Susceptibility to Kainate-Induced Seizures and Exacerbates Hippocampal Neurodegeneration not through Mitochondrial Antioxidant Enzyme SOD2.Antioxidant therapeutics: Pandora's box.Stress during first pregnancy increases seizure threshold in adult male offspring.Design, mechanism of action, bioavailability and therapeutic effects of mn porphyrin-based redox modulators.A Molecular Approach to Epilepsy Management: from Current Therapeutic Methods to Preconditioning Efforts.Oxidative Stress Contributes to Status Epilepticus Associated Mortality.The role of oxidative stress in organophosphate and nerve agent toxicity.Effect of Oxidative Stress on ABC Transporters: Contribution to Epilepsy Pharmacoresistance.Altered Glycolysis and Mitochondrial Respiration in a Zebrafish Model of Dravet SyndromeA novel SOD mimic with a redox-modulating mn (II) complex, ML1 attenuates high glucose-induced abnormalities in intracellular Ca2+ transients and prevents cardiac cell death through restoration of mitochondrial functionInhibition of autophagy and glycolysis by nitric oxide during hypoxia-reoxygenation impairs cellular bioenergetics and promotes cell death in primary neuronsBrain-Specific Superoxide Dismutase 2 Deficiency Causes Perinatal Death with Spongiform Encephalopathy in Mice.Metabolic Dysfunction and Oxidative Stress in Epilepsy.Scavenging reactive oxygen species inhibits status epilepticus-induced neuroinflammation.α-Ketoadipic Acid and α-Aminoadipic Acid Cause Disturbance of Glutamatergic Neurotransmission and Induction of Oxidative Stress In Vitro in Brain of Adolescent Rats.PQQ ameliorates D-galactose induced cognitive impairments by reducing glutamate neurotoxicity via the GSK-3β/Akt signaling pathway in mouse.
P2860
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P2860
Mitochondrial oxidative stress and epilepsy in SOD2 deficient mice: attenuation by a lipophilic metalloporphyrin.
description
2011 nî lūn-bûn
@nan
2011 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Mitochondrial oxidative stress ...... a lipophilic metalloporphyrin.
@ast
Mitochondrial oxidative stress ...... a lipophilic metalloporphyrin.
@en
type
label
Mitochondrial oxidative stress ...... a lipophilic metalloporphyrin.
@ast
Mitochondrial oxidative stress ...... a lipophilic metalloporphyrin.
@en
prefLabel
Mitochondrial oxidative stress ...... a lipophilic metalloporphyrin.
@ast
Mitochondrial oxidative stress ...... a lipophilic metalloporphyrin.
@en
P2093
P2860
P1476
Mitochondrial oxidative stress ...... a lipophilic metalloporphyrin.
@en
P2093
Brian J Day
Li-Ping Liang
Manisha Patel
Shane Rowley
Simon Waldbaum
Ting-Ting Huang
P2860
P304
P356
10.1016/J.NBD.2011.12.025
P577
2011-12-16T00:00:00Z