Persistent cognitive dysfunction after traumatic brain injury: A dopamine hypothesis
about
Traumatic brain injury-induced cognitive and histological deficits are attenuated by delayed and chronic treatment with the 5-HT1A-receptor agonist buspironeCatecholamines and cognition after traumatic brain injuryMaking Waves in the Brain: What Are Oscillations, and Why Modulating Them Makes Sense for Brain InjuryMusic in Research and Rehabilitation of Disorders of Consciousness: Psychological and Neurophysiological FoundationsIllicit dopamine transients: reconciling actions of abused drugsAll's well that transcribes well: non-coding RNAs and post-stroke brain damageAmantadine ameliorates dopamine-releasing deficits and behavioral deficits in rats after fluid percussion injuryCB1 and CB2 Cannabinoid Receptor Antagonists Prevent Minocycline-Induced Neuroprotection Following Traumatic Brain Injury in MiceMolecular mechanisms of cognitive dysfunction following traumatic brain injuryFound in translation: Understanding the biology and behavior of experimental traumatic brain injuryCombination therapies for neurobehavioral and cognitive recovery after experimental traumatic brain injury: Is more better?A combined therapeutic regimen of buspirone and environmental enrichment is more efficacious than either alone in enhancing spatial learning in brain-injured pediatric ratsAmphetamine elicits opposing actions on readily releasable and reserve pools for dopamineEnvironmental enrichment as a viable neurorehabilitation strategy for experimental traumatic brain injury.Music listening after stroke: beneficial effects and potential neural mechanisms.Network dysfunction after traumatic brain injury.Hyperbaric oxygen therapy improves cognitive functioning after brain injury.Traumatic brain injury: an overview of pathobiology with emphasis on military populations.Methamphetamine-induced neurotoxicity disrupts pharmacologically evoked dopamine transients in the dorsomedial and dorsolateral striatumEmpirical comparison of typical and atypical environmental enrichment paradigms on functional and histological outcome after experimental traumatic brain injury.Role of Akt and mammalian target of rapamycin in functional outcome after concussive brain injury in miceMidlife migraine and late-life parkinsonism: AGES-Reykjavik study.Peroxisome proliferator activated receptor-γ and traumatic brain injury.Evaluation of a combined therapeutic regimen of 8-OH-DPAT and environmental enrichment after experimental traumatic brain injury.Negative neuroplasticity in chronic traumatic brain injury and implications for neurorehabilitationAltered regulation of protein kinase a activity in the medial prefrontal cortex of normal and brain-injured animals actively engaged in a working memory task.Biologic and plastic effects of experimental traumatic brain injury treatment paradigms and their relevance to clinical rehabilitation.Traumatic brain injury in adult rats causes progressive nigrostriatal dopaminergic cell loss and enhanced vulnerability to the pesticide paraquat.Addressing neuropsychiatric disturbances during rehabilitation after traumatic brain injury: current and future methods.A mouse model of human repetitive mild traumatic brain injuryAlterations of A-type potassium channels in hippocampal neurons after traumatic brain injuryEvaluation of a combined treatment paradigm consisting of environmental enrichment and the 5-HT1A receptor agonist buspirone after experimental traumatic brain injury.Protein Citrullination: A Proposed Mechanism for Pathology in Traumatic Brain Injury.Posttraumatic Brain Injury Cognitive Performance Is Moderated by Variation Within ANKK1 and DRD2 Genes.Role of bromocriptine in multi-spectral manifestations of traumatic brain injury.Influence of Catechol-O-methyltransferase on Executive Functioning Longitudinally After Early Childhood Traumatic Brain Injury: Preliminary FindingsThe effect of environmental enrichment on substantia nigra gene expression after traumatic brain injury in rats.Increased seizure susceptibility in mice 30 days after fluid percussion injuryDonepezil is ineffective in promoting motor and cognitive benefits after controlled cortical impact injury in male ratsJuvenile Traumatic Brain Injury Increases Alcohol Consumption and Reward in Female Mice
P2860
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P2860
Persistent cognitive dysfunction after traumatic brain injury: A dopamine hypothesis
description
2009 nî lūn-bûn
@nan
2009 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Persistent cognitive dysfunction after traumatic brain injury: A dopamine hypothesis
@ast
Persistent cognitive dysfunction after traumatic brain injury: A dopamine hypothesis
@en
Persistent cognitive dysfunction after traumatic brain injury: A dopamine hypothesis.
@nl
type
label
Persistent cognitive dysfunction after traumatic brain injury: A dopamine hypothesis
@ast
Persistent cognitive dysfunction after traumatic brain injury: A dopamine hypothesis
@en
Persistent cognitive dysfunction after traumatic brain injury: A dopamine hypothesis.
@nl
prefLabel
Persistent cognitive dysfunction after traumatic brain injury: A dopamine hypothesis
@ast
Persistent cognitive dysfunction after traumatic brain injury: A dopamine hypothesis
@en
Persistent cognitive dysfunction after traumatic brain injury: A dopamine hypothesis.
@nl
P2093
P2860
P1476
Persistent cognitive dysfunction after traumatic brain injury: A dopamine hypothesis
@en
P2093
Amy K Wagner
Anthony E Kline
C Edward Dixon
James W Bales
P2860
P304
P356
10.1016/J.NEUBIOREV.2009.03.011
P577
2009-04-01T00:00:00Z