Does the shape of lipid A determine the interaction of LPS with Toll-like receptors?
about
Cyanobacterial lipopolysaccharides and human health - a reviewCharacterization of lipopolysaccharides present in settled house dustRegulation of aicda expression and AID activity: relevance to somatic hypermutation and class switch DNA recombinationBartonella quintana lipopolysaccharide is a natural antagonist of Toll-like receptor 4TLR-Dependent Human Mucosal Epithelial Cell Responses to Microbial PathogensSignal transduction by the lipopolysaccharide receptor, Toll-like receptor-4The effects of airway microbiome on corticosteroid responsiveness in asthmaGender-dependent effects of maternal immune activation on the behavior of mouse offspringBaicalin downregulates Porphyromonas gingivalis lipopolysaccharide-upregulated IL-6 and IL-8 expression in human oral keratinocytes by negative regulation of TLR signaling.Evidence of a specific interaction between new synthetic antisepsis agents and CD14.Pattern recognition via the toll-like receptor system in the human female genital tract.Substitution of the Bordetella pertussis lipid A phosphate groups with glucosamine is required for robust NF-kappaB activation and release of proinflammatory cytokines in cells expressing human but not murine Toll-like receptor 4-MD-2-CD14Role of the conformational rigidity in the design of biomimetic antimicrobial compounds.Effects of aging on endotoxin tolerance induced by lipopolysaccharides derived from Porphyromonas gingivalis and Escherichia coli.Molecular hydrogen reduces LPS-induced neuroinflammation and promotes recovery from sickness behaviour in miceTRPA1 channels mediate acute neurogenic inflammation and pain produced by bacterial endotoxins.Lipopolysaccharide induces a stromal-epithelial signalling axis in a rat model of chronic periodontitis.Chemo-enzymatic synthesis and in vitro cytokine profiling of tailor-made oligofructosides.Single-cell and population NF-κB dynamic responses depend on lipopolysaccharide preparation.Tetra- and penta-acylated lipid A structures of Porphyromonas gingivalis LPS differentially activate TLR4-mediated NF-κB signal transduction cascade and immuno-inflammatory response in human gingival fibroblastsPhosphoryl moieties of lipid A from Neisseria meningitidis and N. gonorrhoeae lipooligosaccharides play an important role in activation of both MyD88- and TRIF-dependent TLR4-MD-2 signaling pathwaysCardiovascular disease and the role of oral bacteria.Toll-like receptors as potential therapeutic targets for multiple diseases.Failure of TLR4-driven NF-kappa B activation to stimulate virus replication in models of HIV type 1 activation.Human gammadelta T cell recognition of lipid A is predominately presented by CD1b or CD1c on dendritic cells.Modulating LPS signal transduction at the LPS receptor complex with synthetic Lipid A analoguesExogenous α-synuclein induces toll-like receptor 4 dependent inflammatory responses in astrocytes.Microbial etiology of periodontitis.Differential Toll-Like Receptor-Signalling of Burkholderia pseudomallei Lipopolysaccharide in Murine and Human Models.Schistosome larvae stimulate macrophage cytokine production through TLR4-dependent and -independent pathways.Toll-like receptor activation of human cells by synthetic triacylated lipid A-like moleculesImplication of antigenic conversion of Helicobacter pylori lipopolysaccharides that involve interaction with surfactant protein D.Generation of inflammatory stimuli: how bacteria set up inflammatory responses in the gingiva.Neuroprotective effects of toll-like receptor 4 antagonism in spinal cord cultures and in a mouse model of motor neuron degeneration.Optimized Triton X-114 assisted lipopolysaccharide (LPS) removal method reveals the immunomodulatory effect of food proteins.Energetics of Endotoxin Recognition in the Toll-Like Receptor 4 Innate Immune Response.Characterization of htrB and msbB mutants of the light organ symbiont Vibrio fischeri.Endotoxin induces proliferation of NSCLC in vitro and in vivo: role of COX-2 and EGFR activation.Characterization of differential Toll-like receptor responses below the optical diffraction limit.Knockdown of versican V1 induces a severe inflammatory response in LPS-induced acute lung injury via the TLR2-NF-κB signaling pathway in C57BL/6J mice.
P2860
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P2860
Does the shape of lipid A determine the interaction of LPS with Toll-like receptors?
description
2002 nî lūn-bûn
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2002 թուականի Մարտին հրատարակուած գիտական յօդուած
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2002 թվականի մարտին հրատարակված գիտական հոդված
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2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Does the shape of lipid A determine the interaction of LPS with Toll-like receptors?
@ast
Does the shape of lipid A determine the interaction of LPS with Toll-like receptors?
@en
Does the shape of lipid A determine the interaction of LPS with Toll-like receptors?
@nl
type
label
Does the shape of lipid A determine the interaction of LPS with Toll-like receptors?
@ast
Does the shape of lipid A determine the interaction of LPS with Toll-like receptors?
@en
Does the shape of lipid A determine the interaction of LPS with Toll-like receptors?
@nl
prefLabel
Does the shape of lipid A determine the interaction of LPS with Toll-like receptors?
@ast
Does the shape of lipid A determine the interaction of LPS with Toll-like receptors?
@en
Does the shape of lipid A determine the interaction of LPS with Toll-like receptors?
@nl
P2093
P1433
P1476
Does the shape of lipid A determine the interaction of LPS with Toll-like receptors?
@en
P2093
Bart Jan Kullberg
Jos W M Van der Meer
Marcel van Deuren
Mihai G Netea
P304
P356
10.1016/S1471-4906(01)02169-X
P577
2002-03-01T00:00:00Z