PKCε phosphorylation of the sodium channel NaV1.8 increases channel function and produces mechanical hyperalgesia in mice.

PKCε phosphorylation of the sodium channel NaV1.8 increases channel function and produces mechanical hyperalgesia in mice.

Item
http://www.wikidata.org/entity/Q34262020
Sensory nerve terminal mitochondrial dysfunction induces hyperexcitability in airway nociceptors via protein kinase CTargeting ASIC3 for Relieving Mice Fibromyalgia Pain: Roles of Electroacupuncture, Opioid, and Adenosine.Transcriptional regulation of oncogenic protein kinase Cϵ (PKCϵ) by STAT1 and Sp1 proteins.Inhibition of tetrodotoxin-resistant sodium current in dorsal root ganglia neurons mediated by D1/D5 dopamine receptorsIncreased BDNF protein expression after ischemic or PKC epsilon preconditioning promotes electrophysiologic changes that lead to neuroprotection.Role for monocyte chemoattractant protein-1 in the induction of chronic muscle pain in the ratGeneration and characterization of ATP analog-specific protein kinase Cδ.Post-translational modifications of voltage-gated sodium channels in chronic pain syndromesProtein kinase Cε is required for spinal analgesic synergy between delta opioid and alpha-2A adrenergic receptor agonist pairs.Electrophysiological correlates of hyperalgesic priming in vitro and in vivoCaMKII Phosphorylation of Na(V)1.5: Novel in Vitro Sites Identified by Mass Spectrometry and Reduced S516 Phosphorylation in Human Heart Failure.Electroacupuncture Attenuates CFA-induced Inflammatory Pain by suppressing Nav1.8 through S100B, TRPV1, Opioid, and Adenosine Pathways in Mice.Criticality and degeneracy in injury-induced changes in primary afferent excitability and the implications for neuropathic pain.Role of reactive oxygen species and TRP channels in the cough reflex.PKC-NF-κB are involved in CCL2-induced Nav1.8 expression and channel function in dorsal root ganglion neuronsRoles of PKC Isoforms in PMA-Induced Modulation of the hERG Channel (Kv11.1).
P2860

PKCε phosphorylation of the sodium channel NaV1.8 increases channel function and produces mechanical hyperalgesia in mice.

Item
http://www.wikidata.org/entity/Q34262020
description
2012 nî lūn-bûn
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2012 թուականի Մարտին հրատարակուած գիտական յօդուած
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2012 թվականի մարտին հրատարակված գիտական հոդված
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2012年の論文
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2012年論文
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2012年論文
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2012年論文
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2012年論文
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2012年論文
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2012年论文
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name
PKCε phosphorylation of the so ...... chanical hyperalgesia in mice.
@ast
PKCε phosphorylation of the so ...... chanical hyperalgesia in mice.
@en
PKCε phosphorylation of the so ...... chanical hyperalgesia in mice.
@nl
type
label
PKCε phosphorylation of the so ...... chanical hyperalgesia in mice.
@ast
PKCε phosphorylation of the so ...... chanical hyperalgesia in mice.
@en
PKCε phosphorylation of the so ...... chanical hyperalgesia in mice.
@nl
prefLabel
PKCε phosphorylation of the so ...... chanical hyperalgesia in mice.
@ast
PKCε phosphorylation of the so ...... chanical hyperalgesia in mice.
@en
PKCε phosphorylation of the so ...... chanical hyperalgesia in mice.
@nl
P1433
P1476
P2093
P2860
P304
P31
P356
P407
P433
P478
P50
P577
P698
P921
P932
P356
P1433
P1476
PKCε phosphorylation of the so ...... echanical hyperalgesia in mice
@en
P2093
P2860
P304
P31
P356
P407
P433
P478
P50
P577
P698
P921
P932