Upregulation of human selenoprotein H in murine hippocampal neuronal cells promotes mitochondrial biogenesis and functional performance - Wikidata - awgldk - TriplyDB

Upregulation of human selenoprotein H in murine hippocampal neuronal cells promotes mitochondrial biogenesis and functional performance

Upregulation of human selenoprotein H in murine hippocampal neuronal cells promotes mitochondrial biogenesis and functional performance

http://www.wikidata.org/entity/Q34388490

about

The dynamics of the mitochondrial organelle as a potential therapeutic target Water-soluble coenzyme q10 inhibits nuclear translocation of apoptosis inducing factor and cell death caused by mitochondrial complex I inhibition.Glutamate induces mitochondrial dynamic imbalance and autophagy activation: preventive effects of selenium.Selenium preserves mitochondrial function, stimulates mitochondrial biogenesis, and reduces infarct volume after focal cerebral ischemia.Selenoprotein H suppresses cellular senescence through genome maintenance and redox regulation Coenzyme q10 ameliorates ultraviolet B irradiation induced cell death through inhibition of mitochondrial intrinsic cell death pathway Understanding selenoprotein function and regulation through the use of rodent models.Acquired multiple acyl-CoA dehydrogenase deficiency and marked selenium deficiency causing severe rhabdomyolysis in a horse.Overexpression of human selenoprotein H in neuronal cells enhances mitochondrial biogenesis and function through activation of protein kinase A, protein kinase B, and cyclic adenosine monophosphate response element-binding protein pathway Selenium in the Therapy of Neurological Diseases. Where is it Going?Ubisol-Q10 Prevents Glutamate-Induced Cell Death by Blocking Mitochondrial Fragmentation and Permeability Transition Pore Opening Selenoprotein H is an essential regulator of redox homeostasis that cooperates with p53 in development and tumorigenesis Selenium, selenoproteins and neurodegenerative diseases.Mitochondrial biogenesis in neurodegeneration.Selenoprotein T Deficiency Leads to Neurodevelopmental Abnormalities and Hyperactive Behavior in Mice.Wolfberries potentiate mitophagy and enhance mitochondrial biogenesis leading to prevention of hepatic steatosis in obese mice: the role of AMP-activated protein kinase α2 subunit.Overexpression of selenoprotein H prevents mitochondrial dynamic imbalance induced by glutamate exposure.Selenium-Dependent Antioxidant Enzymes: Actions and Properties of Selenoproteins.

P2860

Q26853454-6CC1F289-26BA-451F-91BC-602A2E7A8128 Q34158352-967DE727-E4A6-4B1C-A9E2-7714A5BE2090 Q34314424-26FAD1CE-933B-433D-970B-85FA7EA69F70 Q34331235-23AF89B2-2440-4294-ABB5-EF83EB163193 Q34634340-19180B8F-4AAB-4971-8071-EA6FE89332CF Q35600271-43F746F6-296D-4B63-8C3F-25439FCD4B85 Q36129752-04D5FBDF-787C-4FD3-8978-9C2D0A223600 Q36170112-956F5671-080D-4D74-8F4E-FADA18782CDA Q36599604-4AAE6615-13A0-4D1E-ACC7-8C8E0A274B44 Q36872331-327B0389-8007-46ED-9BFC-5CB9CF96FAFD Q36911088-7E674555-F843-4B79-B288-83B7F24362AC Q37281599-E63CC8A2-BFD4-4567-9440-5BA7D658EB68 Q38495280-A369C4A7-4EFE-44BE-B214-C88062A62A38 Q39181903-88B69A8F-BB4E-4B56-93D6-3333E8827777 Q46468188-0D854441-C8B7-4EF4-AF46-5AB0ABAD8DC5 Q46943404-910380CD-B703-44D0-968A-BCE4DA8E50F6 Q51734185-4CF9D14C-54EA-4C07-A880-2DB8DBB75EA7 Q55082777-FE27D4DB-8969-4491-8012-DC8E6EB8481B

P2860

Upregulation of human selenoprotein H in murine hippocampal neuronal cells promotes mitochondrial biogenesis and functional performance

http://www.wikidata.org/entity/Q34388490

description

2010 nî lūn-bûn

@nan

2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հուլիսին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Upregulation of human selenopr ...... sis and functional performance

@ast

Upregulation of human selenopr ...... sis and functional performance

@en

Upregulation of human selenopr ...... sis and functional performance

@nl

type

label

Upregulation of human selenopr ...... sis and functional performance

@ast

Upregulation of human selenopr ...... sis and functional performance

@en

Upregulation of human selenopr ...... sis and functional performance

@nl

prefLabel

Upregulation of human selenopr ...... sis and functional performance

@ast

Upregulation of human selenopr ...... sis and functional performance

@en

Upregulation of human selenopr ...... sis and functional performance

@nl

P1433

Q34388490-336E151A-AC39-496F-932F-1760E2572BB2

P1476

Q34388490-B412C48A-E852-43A3-9C2F-744D58C8C27D

P2093

Q34388490-12CD6A53-1635-4078-8C5F-6C35BFE08754

Q34388490-285D3555-B7EE-436E-A93B-D6181B25A775

Q34388490-4200474F-5D1A-45ED-A83E-68F4653AD30B

Q34388490-50DEA099-225E-41BD-8354-5E4606835018

Q34388490-5630ADA0-B4F2-468A-845E-9B810817A4FD

Q34388490-5AEED772-608A-404A-8286-11841FAFDEA0

P2860

Q34388490-230A6037-F703-4FCA-A101-AF4359CF419E

Q34388490-322E5398-3B2C-46C7-B427-7FD668061733

Q34388490-5629160A-89DE-4F4F-B7C2-28A3E851BB7A

Q34388490-677F743C-946F-44F9-BE0C-C9D72936D2E1

Q34388490-6877EF10-7393-4042-A5C6-2FD94C241296

Q34388490-6F1DC80F-F317-41ED-ACBC-76EFA6D17A28

Q34388490-7F1F3D84-E2B2-41D1-8993-ABE7DDD6E106

Q34388490-A7E0FA1D-0F14-44C6-A889-2B772B35689F

Q34388490-AA726FB6-5FB9-48E4-B4B0-6ED8EEADEDFC

Q34388490-CC42061C-C6B3-4E53-B822-A193382054EF

P304

Q34388490-C7C514C3-0686-4DB2-9250-23B704B10D70

P31

Q34388490-9BB1DEC4-90CC-4E2F-9D1A-99FE3774A277

P356

Q34388490-73A8BBAC-ECC0-4F69-B2E8-BEC9D7BEB1C3

P433

Q34388490-2587108C-94FD-4812-9C92-BDCFD4B38934

P478

Q34388490-E4E32ED1-D11A-471B-99EA-6096B093FE44

P577

Q34388490-FBBF5200-1A9B-4C49-BCEC-80228C1EC876

P5875

Q34388490-F94B4A48-17C1-427B-A7C2-1175C48FEF20

P698

Q34388490-7C783623-A0CE-4EE1-A228-A5F66ADF930D

P932

Q34388490-1FA30653-E0F1-43EE-8515-84A90C195E38

P356

J.MITO.2010.07.007

P1433

P1476

Upregulation of human selenopr ...... sis and functional performance

@en

P2093

Jonathan Z Sexton

http://www.w3.org/2001/XMLSchema#string

Natalia Mendelev

http://www.w3.org/2001/XMLSchema#string

P Andy Li

http://www.w3.org/2001/XMLSchema#string

Qingping He

http://www.w3.org/2001/XMLSchema#string

Sam Witherspoon

http://www.w3.org/2001/XMLSchema#string

Suresh L Mehta

http://www.w3.org/2001/XMLSchema#string

P2860

Overexpression of selenoprotein H reduces Ht22 neuronal cell death after UVB irradiation by preventing superoxide formation.

Selenoprotein H is a redox-sensing high mobility group family DNA-binding protein that up-regulates genes involved in glutathione synthesis and phase II detoxification.

Mechanisms controlling mitochondrial biogenesis and respiration through the thermogenic coactivator PGC-1

Transcriptional paradigms in mammalian mitochondrial biogenesis and function

Chronic activation of AMP kinase results in NRF-1 activation and mitochondrial biogenesis.

Transcriptional activators and coactivators in the nuclear control of mitochondrial function in mammalian cells.

Transcriptional regulatory circuits controlling mitochondrial biogenesis and function.

Overexpression of human selenoprotein H in neuronal cells ameliorates ultraviolet irradiation-induced damage by modulating cell signaling pathways.

TNF-alpha reduces PGC-1alpha expression through NF-kappaB and p38 MAPK leading to increased glucose oxidation in a human cardiac cell model.

Selenoprotein H is a nucleolar thioredoxin-like protein with a unique expression pattern.

P304

76-82

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1016/J.MITO.2010.07.007

http://www.w3.org/2001/XMLSchema#string

P433

1

http://www.w3.org/2001/XMLSchema#string

P478

11

http://www.w3.org/2001/XMLSchema#string

P577

2010-07-23T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

45287002

http://www.w3.org/2001/XMLSchema#string

P698

20656065

http://www.w3.org/2001/XMLSchema#string

P932

2998579

http://www.w3.org/2001/XMLSchema#string