Multiple sites of tumorigenesis in transgenic mice overproducing hCG.

Multiple sites of tumorigenesis in transgenic mice overproducing hCG.

Item
http://www.wikidata.org/entity/Q34412127
Genetic Models for the Study of Luteinizing Hormone Receptor FunctionSynergistic activation of innate and adaptive immune mechanisms in the treatment of gonadotropin-sensitive tumorsInfertility in Female Mice with a Gain-of-Function Mutation in the Luteinizing Hormone Receptor Is Due to Irregular Estrous Cyclicity, Anovulation, Hormonal Alterations, and Polycystic Ovaries.Luteinizing hormone promotes gonadal tumorigenesis in inhibin-deficient miceActivation of the lutropin/choriogonadotropin receptor in MA-10 cells stimulates tyrosine kinase cascades that activate ras and the extracellular signal regulated kinases (ERK1/2).Female mice expressing constitutively active mutants of FSH receptor present with a phenotype of premature follicle depletion and estrogen excessTransgenesis-mediated reproductive dysfunction and tumorigenesis: effects of immunological neutralizationLuteinizing hormone receptor deficiency increases the susceptibility to alkylating agent-induced lymphomagenesis in mice.Animal models for aberrations of gonadotropin action.Gonadotropin-mediated chemoresistance: Delineation of molecular pathways and targetsLutropin/choriogonadotropin stimulate the proliferation of primary cultures of rat Leydig cells through a pathway that involves activation of the extracellularly regulated kinase 1/2 cascade.Gonadectomy-induced adrenocortical neoplasia in the domestic ferret (Mustela putorius furo) and laboratory mouse.The mammalian ovary from genesis to revelation.TGF-β-induced hCG-β regulates redox homeostasis in glioma cells.The ERK1/2 pathway regulates testosterone synthesis by coordinately regulating the expression of steroidogenic genes in Leydig cells.The permissive role of prolactin as a regulator of luteinizing hormone action in the female mouse ovary and extragonadal tumorigenesis.
P2860

Multiple sites of tumorigenesis in transgenic mice overproducing hCG.

Item
http://www.wikidata.org/entity/Q34412127
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2005 nî lūn-bûn
@nan
2005 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Multiple sites of tumorigenesis in transgenic mice overproducing hCG.
@ast
Multiple sites of tumorigenesis in transgenic mice overproducing hCG.
@en
Multiple sites of tumorigenesis in transgenic mice overproducing hCG.
@nl
type
label
Multiple sites of tumorigenesis in transgenic mice overproducing hCG.
@ast
Multiple sites of tumorigenesis in transgenic mice overproducing hCG.
@en
Multiple sites of tumorigenesis in transgenic mice overproducing hCG.
@nl
prefLabel
Multiple sites of tumorigenesis in transgenic mice overproducing hCG.
@ast
Multiple sites of tumorigenesis in transgenic mice overproducing hCG.
@en
Multiple sites of tumorigenesis in transgenic mice overproducing hCG.
@nl
P1433
P1476
P2093
P304
P31
P356
P433
P478
P577
P698
P356
P1433
P1476
Multiple sites of tumorigenesis in transgenic mice overproducing hCG.
@en
P2093
P304
P31
P356
P433
P478
P577
P698