about
HIV-associated adipose redistribution syndrome (HARS): etiology and pathophysiological mechanismsAntisense-mediated inhibition of human immunodeficiency virus (HIV) replication by use of an HIV type 1-based vector results in severely attenuated mutants incapable of developing resistanceMechanism of HIV protein induced modulation of mesenchymal stem cell osteogenic differentiation.Antiretroviral therapy-induced mitochondrial toxicity: potential mechanisms beyond polymerase-γ inhibition.A phase I study of nelfinavir concurrent with temozolomide and radiotherapy in patients with glioblastoma multiforme.Monitoring Antiretroviral Therapy in HIV-Infected Children in Resource-Limited Countries: A Tale of Two EpidemicsHIV protease inhibitors decrease VEGF/HIF-1alpha expression and angiogenesis in glioblastoma cellsMutational analysis of the mitochondrial tRNA genes and flanking regions in umbilical cord tissue from uninfected infants receiving AZT-based therapies for prophylaxis of HIV-1.The HIV proteins Tat and Nef promote human bone marrow mesenchymal stem cell senescence and alter osteoblastic differentiation.HIV-associated adipose redistribution syndrome (HARS): definition, epidemiology and clinical impact.Modulation of osteoclastogenesis induced by nucleoside reverse transcriptase inhibitors.Intracellular metabolism and persistence of the anti-human immunodeficiency virus activity of 2',3'-didehydro-3'-deoxy-4'-ethynylthymidine, a novel thymidine analog.Is PPARγ a prospective player in HIV-1-associated bone disease?Modulating the tumor microenvironment to increase radiation responsiveness.Electrocardiographic findings in a cross-sectional study of human immunodeficiency virus (HIV) patients in Enugu, south-east Nigeria.Performance of Clinical Criteria for Screening of Possible Antiretroviral Related Mitochondrial Toxicity in HIV-Infected Children in AccraUpregulation of Apoptosis Pathway Genes in Peripheral Blood Mononuclear Cells of HIV-Infected Individuals with Antiretroviral Therapy-Associated Mitochondrial Toxicity.The HIV matrix protein p17 induces hepatic lipid accumulation via modulation of nuclear receptor transcriptoma.The effects of HIV disease and older age on laboratory-based, naturalistic, and self-perceived symptoms of prospective memory: does retrieval cue type and delay interval matter?
P2860
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P2860
description
2002 nî lūn-bûn
@nan
2002 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Long-term exposure to lifelong therapies.
@ast
Long-term exposure to lifelong therapies.
@en
Long-term exposure to lifelong therapies.
@nl
type
label
Long-term exposure to lifelong therapies.
@ast
Long-term exposure to lifelong therapies.
@en
Long-term exposure to lifelong therapies.
@nl
prefLabel
Long-term exposure to lifelong therapies.
@ast
Long-term exposure to lifelong therapies.
@en
Long-term exposure to lifelong therapies.
@nl
P1476
Long-term exposure to lifelong therapies.
@en
P2093
Powderly WG
P304
P407
P478
29 Suppl 1
P577
2002-02-01T00:00:00Z