RAGE expression in tumor-associated macrophages promotes angiogenesis in glioma. - Wikidata - awgldk - TriplyDB

RAGE expression in tumor-associated macrophages promotes angiogenesis in glioma.

RAGE expression in tumor-associated macrophages promotes angiogenesis in glioma.

http://www.wikidata.org/entity/Q34716082

about

Scavenger Receptors: Emerging Roles in Cancer Biology and Immunology The multiple faces of RAGE--opportunities for therapeutic intervention in aging and chronic disease Tuftsin-driven experimental autoimmune encephalomyelitis recovery requires neuropilin-1.Soy and breast cancer: focus on angiogenesis.Small Molecule Inhibition of Ligand-Stimulated RAGE-DIAPH1 Signal Transduction Characterization of Arginase Expression in Glioma-Associated Microglia and Macrophages The role of microglia and macrophages in glioma maintenance and progression Tumour-associated macrophages correlate with microvascular bed extension in colorectal cancer patients.Class A1 scavenger receptor modulates glioma progression by regulating M2-like tumor-associated macrophage polarization.M2-like tumor-associated macrophages drive vasculogenic mimicry through amplification of IL-6 expression in glioma cells.Targeting the Receptor for Advanced Glycation Endproducts (RAGE): A Medicinal Chemistry Perspective.Overexpression of receptor for advanced glycation end products (RAGE) in ovarian cancer.Targeting of RAGE-ligand signaling impairs breast cancer cell invasion and metastasis.CECR1-mediated cross talk between macrophages and vascular mural cells promotes neovascularization in malignant glioma.Plasma miR-185 is decreased in patients with esophageal squamous cell carcinoma and might suppress tumor migration and invasion by targeting RAGE.RAGE: A novel target for breast cancer growth and metastasis.Targeted delivery of nano-PTX to the brain tumor-associated macrophages Glioblastoma-associated microglia and macrophages: targets for therapies to improve prognosis.Paradoxical Role of High Mobility Group Box 1 in Glioma: A Suppressor or a Promoter?The immunological function of GABAergic system.Chronic inflammation: is it the driver or is it paving the road for malignant transformation?Elevated AEG-1 expression in macrophages promotes hypopharyngeal cancer invasion through the STAT3-MMP-9 signaling pathway.Microglia in the tumor microenvironment: taking their TOLL on glioma biology.AGER rs2070600 polymorphism elevates neutrophil-lymphocyte ratio and mortality in metastatic lung adenocarcinoma.Establishment of S100A8 Transgenic Rats to Understand Innate Property of S100A8 and Its Immunological Role.When Immune Cells Turn Bad-Tumor-Associated Microglia/Macrophages in Glioma.The Role of Microglia and Macrophages in CNS Homeostasis, Autoimmunity, and Cancer.VEGF as a modulator of the innate immune response in glioblastoma.Extracellular vesicle-transported Semaphorin3A promotes vascular permeability in glioblastoma.Role of Macrophages in Brain Tumor Growth and Progression.In vitro anticancer effects of a RAGE inhibitor discovered using a structure-based drug design system.Comparative proteomic analysis of cat eye syndrome critical region protein 1- function in tumor-associated macrophages and immune response regulation of glial tumors Microglia/Astrocytes-Glioblastoma Crosstalk: Crucial Molecular Mechanisms and Microenvironmental Factors

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P2860

RAGE expression in tumor-associated macrophages promotes angiogenesis in glioma.

http://www.wikidata.org/entity/Q34716082

description

2014 nî lūn-bûn

@nan

2014 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2014年の論文

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2014年論文

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2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

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2014年論文

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2014年论文

@wuu

name

RAGE expression in tumor-associated macrophages promotes angiogenesis in glioma.

@ast

RAGE expression in tumor-associated macrophages promotes angiogenesis in glioma.

@en

RAGE expression in tumor-associated macrophages promotes angiogenesis in glioma.

@nl

type

label

RAGE expression in tumor-associated macrophages promotes angiogenesis in glioma.

@ast

RAGE expression in tumor-associated macrophages promotes angiogenesis in glioma.

@en

RAGE expression in tumor-associated macrophages promotes angiogenesis in glioma.

@nl

prefLabel

RAGE expression in tumor-associated macrophages promotes angiogenesis in glioma.

@ast

RAGE expression in tumor-associated macrophages promotes angiogenesis in glioma.

@en

RAGE expression in tumor-associated macrophages promotes angiogenesis in glioma.

@nl

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0008-5472.CAN-14-1240

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Cancer Research

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RAGE expression in tumor-associated macrophages promotes angiogenesis in glioma

@en

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Anna Carolina Carvalho da Fonseca

http://www.w3.org/2001/XMLSchema#string

Behnam Badie

http://www.w3.org/2001/XMLSchema#string

Hiroshi Yamamoto

http://www.w3.org/2001/XMLSchema#string

Huaqing Wang

http://www.w3.org/2001/XMLSchema#string

Ian Y Zhang

http://www.w3.org/2001/XMLSchema#string

Junling Liang

http://www.w3.org/2001/XMLSchema#string

Leying Zhang

http://www.w3.org/2001/XMLSchema#string

Lihong Weng

http://www.w3.org/2001/XMLSchema#string

Mao Ouyang

http://www.w3.org/2001/XMLSchema#string

Rama Natarajan

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P2860

AIP1 functions as an endogenous inhibitor of VEGFR2-mediated signaling and inflammatory angiogenesis in mice

Immunity, Inflammation, and Cancer

The receptor for advanced glycation end products (RAGE) is a cellular binding site for amphoterin. Mediation of neurite outgrowth and co-expression of rage and amphoterin in the developing nervous system

Peroxisome proliferator-activated receptor gamma down-regulates receptor for advanced glycation end products and inhibits smooth muscle cell proliferation in a diabetic and nondiabetic rat carotid artery injury model

S100A4 and bone morphogenetic protein-2 codependently induce vascular smooth muscle cell migration via phospho-extracellular signal-regulated kinase and chloride intracellular channel 4

RAGE gene deletion inhibits the development and progression of ductal neoplasia and prolongs survival in a murine model of pancreatic cancer.

Tumor-associated macrophages are predominant carriers of cyclodextrin-based nanoparticles into gliomas.

Cathepsin cysteine proteases are effectors of invasive growth and angiogenesis during multistage tumorigenesis.

Induction of anti-glioma natural killer cell response following multiple low-dose intracerebral CpG therapy

Carbon nanotubes enhance CpG uptake and potentiate antiglioma immunity.

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7285-7297

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scholarly article

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10.1158/0008-5472.CAN-14-1240

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24

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74

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25326491

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