T cell pathways involving CTLA4 contribute to a model of acute lung injury. - Wikidata - awgldk - TriplyDB

T cell pathways involving CTLA4 contribute to a model of acute lung injury.

T cell pathways involving CTLA4 contribute to a model of acute lung injury.

http://www.wikidata.org/entity/Q34748704

about

Concordance of preclinical and clinical pharmacology and toxicology of therapeutic monoclonal antibodies and fusion proteins: cell surface targets T cells and lung injury. Impact of rapamycin.Active players in resolution of shock/sepsis induced indirect lung injury: immunomodulatory effects of Tregs and PD-1.Pathogenesis of indirect (secondary) acute lung injury.Critical modifier role of membrane-cystic fibrosis transmembrane conductance regulator-dependent ceramide signaling in lung injury and emphysema.The ratio of Th17/Treg cells as a risk indicator in early acute respiratory distress syndrome Unexpected Role for Adaptive αβTh17 Cells in Acute Respiratory Distress Syndrome.Blockage of thymic stromal lymphopoietin signaling improves acute lung injury in mice by regulating pulmonary dendritic cells γδ T cells protect against LPS-induced lung injury.Regulation of cytotoxic T lymphocyte antigen 4 by cyclic AMP.Imbalance of Th17/Tregs in rats with smoke inhalation-induced acute lung injury.Rapamycin attenuates acute lung injury induced by LPS through inhibition of Th17 cell proliferation in mice Plasma glutamate-modulated interaction of A2AR and mGluR5 on BMDCs aggravates traumatic brain injury-induced acute lung injury Overcoming in vivo barriers to targeted nanodelivery.Innate Lymphoid Cells Are the Predominant Source of IL-17A during the Early Pathogenesis of Acute Respiratory Distress Syndrome.α-bisabolol-loaded lipid-core nanocapsules reduce lipopolysaccharide-induced pulmonary inflammation in mice.Early infectious acute respiratory distress syndrome is characterized by activation and proliferation of alveolar T-cells.ROCK regulates the intermittent mode of interstitial T cell migration in inflamed lungs.Upregulation of CD19⁺CD24(hi)CD38(hi) regulatory B cells is associated with a reduced risk of acute lung injury in elderly pneumonia patients.

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P2860

T cell pathways involving CTLA4 contribute to a model of acute lung injury.

http://www.wikidata.org/entity/Q34748704

description

2010 nî lūn-bûn

@nan

2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

T cell pathways involving CTLA4 contribute to a model of acute lung injury.

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T cell pathways involving CTLA4 contribute to a model of acute lung injury.

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T cell pathways involving CTLA4 contribute to a model of acute lung injury.

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T cell pathways involving CTLA4 contribute to a model of acute lung injury.

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T cell pathways involving CTLA4 contribute to a model of acute lung injury.

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T cell pathways involving CTLA4 contribute to a model of acute lung injury.

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T cell pathways involving CTLA4 contribute to a model of acute lung injury.

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T cell pathways involving CTLA4 contribute to a model of acute lung injury.

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T cell pathways involving CTLA4 contribute to a model of acute lung injury.

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JIMMUNOL.0903238

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Journal of Immunology

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T cell pathways involving CTLA4 contribute to a model of acute lung injury.

@en

P2093

Carlos Jose Suarez

http://www.w3.org/2001/XMLSchema#string

David L Perkins

http://www.w3.org/2001/XMLSchema#string

Jan E Schnitzer

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Kai Yu Jen

http://www.w3.org/2001/XMLSchema#string

Ko-Wei Lin

http://www.w3.org/2001/XMLSchema#string

Nathan Parker

http://www.w3.org/2001/XMLSchema#string

Patricia W Finn

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Samir S Makani

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Takeshi Nakajima

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P2860

The B7 family revisited

IL-23 drives a pathogenic T cell population that induces autoimmune inflammation

Incidence and outcomes of acute lung injury

The acute respiratory distress syndrome

Toll-like receptors: critical proteins linking innate and acquired immunity

Requirement of interleukin 17 receptor signaling for lung CXC chemokine and granulocyte colony-stimulating factor expression, neutrophil recruitment, and host defense.

Regulatory T cells selectively express toll-like receptors and are activated by lipopolysaccharide

Control of adaptive immune responses by Toll-like receptors.

Critical role of IL-17RA in immunopathology of influenza infection.

Resolution of LPS-induced airway inflammation and goblet cell hyperplasia is independent of IL-18.

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5835-5841

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scholarly article

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10.4049/JIMMUNOL.0903238

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10

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184

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2010-04-12T00:00:00Z

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43131065

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20385880

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3068917

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