Effect of cytokine interplay on macrophage polarization during chronic pulmonary infection with Cryptococcus neoformans.
about
Cryptococcus and Phagocytes: Complex Interactions that Influence Disease OutcomeElucidating the immunological function of the Cryptococcus neoformans capsuleTH1/TH2 paradigm extended: macrophage polarization as an unappreciated pathogen-driven escape mechanism?Immunoregulation in Fungal DiseasesLack of IL-4 receptor expression on T helper cells reduces T helper 2 cell polyfunctionality and confers resistance in allergic bronchopulmonary mycosis"Of mice and men": arginine metabolism in macrophagesScavenger receptor A modulates the immune response to pulmonary Cryptococcus neoformans infection.Development of optical probes for in vivo imaging of polarized macrophages during foreign body reactions.Macrophage activation state determines the response to rhinovirus infection in a mouse model of allergic asthma.PI3K/Akt signaling pathway modulates influenza virus induced mouse alveolar macrophage polarization to M1/M2b.STAT1 signaling is essential for protection against Cryptococcus neoformans infection in mice.Cryptococcus interactions with macrophages: evasion and manipulation of the phagosome by a fungal pathogen.Immune modulation mediated by cryptococcal laccase promotes pulmonary growth and brain dissemination of virulent Cryptococcus neoformans in mice.Macrophage mitochondrial and stress response to ingestion of Cryptococcus neoformans.Cerebrospinal fluid cytokine profiles predict risk of early mortality and immune reconstitution inflammatory syndrome in HIV-associated cryptococcal meningitisParadoxical Immune Responses in Non-HIV Cryptococcal MeningitisRepeated exposure to Aspergillus fumigatus conidia results in CD4+ T cell-dependent and -independent pulmonary arterial remodeling in a mixed Th1/Th2/Th17 microenvironment that requires interleukin-4 (IL-4) and IL-10.Cryptococcal heat shock protein 70 homolog Ssa1 contributes to pulmonary expansion of Cryptococcus neoformans during the afferent phase of the immune response by promoting macrophage M2 polarizationCryptococcus neoformans Infection in Mice Lacking Type I Interferon Signaling Leads to Increased Fungal Clearance and IL-4-Dependent Mucin Production in the Lungs.IL-23 dampens the allergic response to Cryptococcus neoformans through IL-17-independent and -dependent mechanisms.Vaccine and immunotherapeutic approaches for the prevention of cryptococcosis: lessons learned from animal models.Dectin-3 Is Not Required for Protection against Cryptococcus neoformans Infection.STAT1 signaling within macrophages is required for antifungal activity against Cryptococcus neoformansThe Cnes2 locus on mouse chromosome 17 regulates host defense against cryptococcal infection through pleiotropic effects on host immunityVirulence factors identified by Cryptococcus neoformans mutant screen differentially modulate lung immune responses and brain dissemination.Protective immunity against pulmonary cryptococcosis is associated with STAT1-mediated classical macrophage activation.Cryptococcus neoformans growth and protection from innate immunity are dependent on expression of a virulence-associated DEAD-box protein, Vad1.The phenotype of the Cryptococcus-specific CD4+ memory T-cell response is associated with disease severity and outcome in HIV-associated cryptococcal meningitis.Macrophage M1/M2 polarization dynamically adapts to changes in cytokine microenvironments in Cryptococcus neoformans infection.Protective phenotypes of club cells and alveolar macrophages are favored as part of endotoxin-mediated prevention of asthma.Induction of Protective Immunity to Cryptococcal Infection in Mice by a Heat-Killed, Chitosan-Deficient Strain of Cryptococcus neoformans.Antifungal Activity of Plasmacytoid Dendritic Cells against Cryptococcus neoformans In Vitro Requires Expression of Dectin-3 (CLEC4D) and Reactive Oxygen Species.The intracellular life of Cryptococcus neoformans.Utilizing Viral Nanoparticle/Dendron Hybrid Conjugates in Photodynamic Therapy for Dual Delivery to Macrophages and Cancer Cells.Interleukin-17A enhances host defense against cryptococcal lung infection through effects mediated by leukocyte recruitment, activation, and gamma interferon productionInduction of protective immunity against cryptococcosis.Acute lung injury: how macrophages orchestrate resolution of inflammation and tissue repairClassical versus alternative macrophage activation: the Ying and the Yang in host defense against pulmonary fungal infections.Local GM-CSF-Dependent Differentiation and Activation of Pulmonary Dendritic Cells and Macrophages Protect against Progressive Cryptococcal Lung Infection in Mice.Immunity to Cryptococcus neoformans and C. gattii during cryptococcosis
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P2860
Effect of cytokine interplay on macrophage polarization during chronic pulmonary infection with Cryptococcus neoformans.
description
2011 nî lūn-bûn
@nan
2011 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի մարտին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
name
Effect of cytokine interplay o ...... with Cryptococcus neoformans.
@ast
Effect of cytokine interplay o ...... with Cryptococcus neoformans.
@en
Effect of cytokine interplay o ...... with Cryptococcus neoformans.
@nl
type
label
Effect of cytokine interplay o ...... with Cryptococcus neoformans.
@ast
Effect of cytokine interplay o ...... with Cryptococcus neoformans.
@en
Effect of cytokine interplay o ...... with Cryptococcus neoformans.
@nl
prefLabel
Effect of cytokine interplay o ...... with Cryptococcus neoformans.
@ast
Effect of cytokine interplay o ...... with Cryptococcus neoformans.
@en
Effect of cytokine interplay o ...... with Cryptococcus neoformans.
@nl
P2093
P2860
P356
P1476
Effect of cytokine interplay o ...... with Cryptococcus neoformans.
@en
P2093
Galen B Toews
Gary B Huffnagle
Roderick A McDonald
Shikha Arora
Tiffany M Tsang
P2860
P304
P356
10.1128/IAI.01270-10
P407
P577
2011-03-07T00:00:00Z