Deletion of ribosomal S6 kinases does not attenuate pathological, physiological, or insulin-like growth factor 1 receptor-phosphoinositide 3-kinase-induced cardiac hypertrophy.
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Cardiac regulation by phosphoinositide 3-kinases and PTENProtein kinase cascades in the regulation of cardiac hypertrophyMolecular basis of physiological heart growth: fundamental concepts and new players17ß-Estradiol regulates mTORC2 sensitivity to rapamycin in adaptive cardiac remodelingPhosphorylated Ribosomal Protein S6 Is Required for Akt-Driven Hyperplasia and Malignant Transformation, but Not for Hypertrophy, Aneuploidy and Hyperfunction of Pancreatic β-CellsInsulin Signaling and Heart Failure.Partial restoration of cardiac function with ΔPDZ nNOS in aged mdx model of Duchenne cardiomyopathy.Akt1 in the cardiovascular system: friend or foe?MTORC1 regulates cardiac function and myocyte survival through 4E-BP1 inhibition in mice.Class IA phosphoinositide 3-kinase regulates heart size and physiological cardiac hypertrophy.Folliculin (Flcn) inactivation leads to murine cardiac hypertrophy through mTORC1 deregulationRheb (Ras homologue enriched in brain)-dependent mammalian target of rapamycin complex 1 (mTORC1) activation becomes indispensable for cardiac hypertrophic growth after early postnatal period.Myocardial loss of IRS1 and IRS2 causes heart failure and is controlled by p38α MAPK during insulin resistance.mTOR attenuates the inflammatory response in cardiomyocytes and prevents cardiac dysfunction in pathological hypertrophy.Smooth muscle protein-22-mediated deletion of Tsc1 results in cardiac hypertrophy that is mTORC1-mediated and reversed by rapamycinThe role of TGFβ1 and LRG1 in cardiac remodelling and heart failure.Cardiac aging: from molecular mechanisms to significance in human health and diseasemTOR signaling in mice with dysfunctional cardiac ryanodine receptor ion channel.p70 ribosomal S6 kinase regulates subpleural fibrosis following transforming growth factor-α expression in the lungThe mTOR pathway in the control of protein synthesis.Cardiac restricted overexpression of kinase-dead mammalian target of rapamycin (mTOR) mutant impairs the mTOR-mediated signaling and cardiac function.Ecto-5'-nucleotidase deficiency exacerbates pressure-overload-induced left ventricular hypertrophy and dysfunction.Insulin signaling pathways and cardiac growth.Myocardial AKT: the omnipresent nexus.Akt2 regulates cardiac metabolism and cardiomyocyte survival.Mammalian target of rapamycin signaling in cardiac physiology and disease.mTORC1 signaling: what we still don't know.Drosophila models of cardiac disease.Early origins of heart disease: low birth weight and the role of the insulin-like growth factor system in cardiac hypertrophy.Characterization and regulation of mechanical loading-induced compensatory muscle hypertrophy.Fuel availability and fate in cardiac metabolism: A tale of two substrates.Cardiac mTOR complex 2 preserves ventricular function in pressure-overload hypertrophy.p70 Ribosomal S6 kinase is required for airway smooth muscle cell size enlargement but not increased contractile protein expression.Transforming growth factor-beta induces airway smooth muscle hypertrophy.ZFP260 is an inducer of cardiac hypertrophy and a nuclear mediator of endothelin-1 signaling.S6 kinase 1 knockout inhibits uninephrectomy- or diabetes-induced renal hypertrophy.Disruption of coordinated cardiac hypertrophy and angiogenesis contributes to the transition to heart failure.The death effector domain-containing DEDD supports S6K1 activity via preventing Cdk1-dependent inhibitory phosphorylation.Unique and Redundant Functions of p70 Ribosomal S6 Kinase Isoforms Regulate Mesenchymal Cell Proliferation and Migration in Pulmonary Fibrosis.Selective activation of PI3Kalpha/Akt/GSK-3beta signalling and cardiac compensatory hypertrophy during recovery from heart failure.
P2860
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P2860
Deletion of ribosomal S6 kinases does not attenuate pathological, physiological, or insulin-like growth factor 1 receptor-phosphoinositide 3-kinase-induced cardiac hypertrophy.
description
2004 nî lūn-bûn
@nan
2004 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Deletion of ribosomal S6 kinas ...... e-induced cardiac hypertrophy.
@ast
Deletion of ribosomal S6 kinas ...... e-induced cardiac hypertrophy.
@en
type
label
Deletion of ribosomal S6 kinas ...... e-induced cardiac hypertrophy.
@ast
Deletion of ribosomal S6 kinas ...... e-induced cardiac hypertrophy.
@en
prefLabel
Deletion of ribosomal S6 kinas ...... e-induced cardiac hypertrophy.
@ast
Deletion of ribosomal S6 kinas ...... e-induced cardiac hypertrophy.
@en
P2093
P2860
P1476
Deletion of ribosomal S6 kinas ...... e-induced cardiac hypertrophy.
@en
P2093
Adam L Dorfman
George Thomas
John Blenis
Julie R McMullen
Kathleen A Martin
Mario Pende
Megan C Sherwood
Oleg Tarnavski
Sarah Longnus
P2860
P304
P356
10.1128/MCB.24.14.6231-6240.2004
P407
P577
2004-07-01T00:00:00Z