Rapid mitochondrial dysfunction mediates TNF-alpha-induced neurotoxicity
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Lipopolysaccharide exacerbates infarct size and results in worsened post-stroke behavioral outcomes.Mitochondria-Derived Damage-Associated Molecular Patterns in Neurodegeneration.Degeneration in Arousal Neurons in Chronic Sleep Disruption Modeling Sleep Apnea.Neuroprotective effect of activated 5'-adenosine monophosphate-activated protein kinase on cone system function during retinal inflammation.The protective mechanism of Ginkgolides and Ginkgo flavonoids on the TNF-α induced apoptosis of rat hippocampal neurons and its mechanisms in vitroInterleukin-6 is increased in plasma and cerebrospinal fluid of community-dwelling domestic dogs with acute ischaemic stroke.Stereotactic Administration of Edaravone Ameliorates Collagenase-Induced Intracerebral Hemorrhage in RatTNF-α and Beyond: Rapid Mitochondrial Dysfunction Mediates TNF-α-Induced Neurotoxicity.Mitochondrial crisis in cerebrovascular endothelial cells opens the blood-brain barrier.Oscillations of ultra-weak photon emission from cancer and non-cancer cells stressed by culture medium change and TNF-α.TNFα affects CREB-mediated neuroprotective signaling pathways of synaptic plasticity in neurons as revealed by proteomics and phospho-proteomics.A critical role of TRPM2 channel in Aβ42 -induced microglial activation and generation of tumor necrosis factor-α.Naringin and Sertraline Ameliorate Doxorubicin-Induced Behavioral Deficits Through Modulation of Serotonin Level and Mitochondrial Complexes Protection Pathway in Rat Hippocampus.Panax notoginseng saponins administration modulates pro- /anti-inflammatory factor expression and improves neurologic outcome following permanent MCAO in rats.TNF-α Mediates the Intrinsic and Extrinsic Pathway in Propofol-Induced Neuronal Apoptosis Via PI3K/Akt Signaling Pathway in Rat Prefrontal Cortical Neurons.Activation of Class I histone deacetylases contributes to mitochondrial dysfunction in cardiomyocytes with altered complex activities.Recurrent Hypoglycemia Exacerbates Cerebral Ischemic Damage in Diabetic Rats via Enhanced Post-Ischemic Mitochondrial Dysfunction.The HTLV-1 gp21 fusion peptide inhibits antigen specific T-cell activation in-vitro and in mice.Putative Inflammatory Sensitive Mechanisms Underlying Risk or Resilience to Social Stress
P2860
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P2860
Rapid mitochondrial dysfunction mediates TNF-alpha-induced neurotoxicity
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Rapid mitochondrial dysfunction mediates TNF-alpha-induced neurotoxicity
@ast
Rapid mitochondrial dysfunction mediates TNF-alpha-induced neurotoxicity
@en
type
label
Rapid mitochondrial dysfunction mediates TNF-alpha-induced neurotoxicity
@ast
Rapid mitochondrial dysfunction mediates TNF-alpha-induced neurotoxicity
@en
prefLabel
Rapid mitochondrial dysfunction mediates TNF-alpha-induced neurotoxicity
@ast
Rapid mitochondrial dysfunction mediates TNF-alpha-induced neurotoxicity
@en
P2093
P2860
P356
P1476
Rapid mitochondrial dysfunction mediates TNF-alpha-induced neurotoxicity
@en
P2093
Danielle N Doll
James W Simpkins
Stephanie L Rellick
Taura L Barr
Xuefang Ren
P2860
P304
P356
10.1111/JNC.13008
P407
P577
2015-01-29T00:00:00Z