Hyperglycemia-induced B cell toxicity. The fate of pancreatic islets transplanted into diabetic mice is dependent on their genetic background.
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Protein Modifications as Manifestations of Hyperglycemic Glucotoxicity in Diabetes and Its ComplicationsHyperglycemic Stress and Carbon Stress in Diabetic GlucotoxicityStreptozotocin-induced type 1 diabetes in rodents as a model for studying mitochondrial mechanisms of diabetic β cell glucotoxicityBeta cell mass and growth after syngeneic islet cell transplantation in normal and streptozocin diabetic C57BL/6 mice.Engraftment and growth of transplanted pancreatic islets.Early intensive insulin treatment for induction of long-term glycaemic control in type 2 diabetes.Sustained beta-cell dysfunction but normalized islet mass in aged thrombospondin-1 deficient mice.Preservation of insulin mRNA levels and insulin secretion in HIT cells by avoidance of chronic exposure to high glucose concentrations.New perspectives on the structure and function of the normal and diabetic beta-cell.Resistance to type 2 diabetes mellitus: a matter of hormesis?β-Cell dysfunction in diabetes: a crisis of identity?Prolonged exposure of human beta cells to elevated glucose levels results in sustained cellular activation leading to a loss of glucose regulation.Prolonged exposure of human pancreatic islets to high glucose concentrations in vitro impairs the beta-cell function.Optimal elevation of β-cell 11β-hydroxysteroid dehydrogenase type 1 is a compensatory mechanism that prevents high-fat diet-induced β-cell failure.Impairment of glucose-induced insulin secretion in human pancreatic islets transplanted to diabetic nude mice.Accelerated loss of islet beta cells in sucrose-fed Goto-Kakizaki rats, a genetic model of non-insulin-dependent diabetes mellitus.Effect of rosiglitazone on the differential expression of diabetes-associated proteins in pancreatic islets of C57Bl/6 lep/lep mice.pH is decreased in transplanted rat pancreatic islets.Effect of the Diabetic State on Islet Engraftment and Function in a Large Animal Model of Islet-Kidney Transplantation.Delayed type hypersensitivity-associated cytokines in islet xenotransplantation: limited efficacy of interleukin-2- and tumor necrosis factor-alpha-blockade in interferon-gamma receptor-deficient mice.The Spleen as an Optimal Site for Islet Transplantation and a Source of Mesenchymal Stem Cells.Optimal Insulin Treatment in Syngeneic Islet TransplantationDifferent islet protein expression profiles during spontaneous diabetes development vs. allograft rejection in BB-DP rats
P2860
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P2860
Hyperglycemia-induced B cell toxicity. The fate of pancreatic islets transplanted into diabetic mice is dependent on their genetic background.
description
1990 nî lūn-bûn
@nan
1990年の論文
@ja
1990年論文
@yue
1990年論文
@zh-hant
1990年論文
@zh-hk
1990年論文
@zh-mo
1990年論文
@zh-tw
1990年论文
@wuu
1990年论文
@zh
1990年论文
@zh-cn
name
Hyperglycemia-induced B cell t ...... t on their genetic background.
@ast
Hyperglycemia-induced B cell t ...... t on their genetic background.
@en
type
label
Hyperglycemia-induced B cell t ...... t on their genetic background.
@ast
Hyperglycemia-induced B cell t ...... t on their genetic background.
@en
prefLabel
Hyperglycemia-induced B cell t ...... t on their genetic background.
@ast
Hyperglycemia-induced B cell t ...... t on their genetic background.
@en
P2860
P356
P1476
Hyperglycemia-induced B cell t ...... t on their genetic background.
@en
P2093
Andersson A
P2860
P304
P356
10.1172/JCI114955
P407
P577
1990-12-01T00:00:00Z