Role of estrogen receptor signaling required for endometriosis-like lesion establishment in a mouse model. - Wikidata - awgldk - TriplyDB

Role of estrogen receptor signaling required for endometriosis-like lesion establishment in a mouse model.

Role of estrogen receptor signaling required for endometriosis-like lesion establishment in a mouse model.

http://www.wikidata.org/entity/Q36114008

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The role of lipoxin A4 in endometrial biology and endometriosis Reversal of fortune: estrogen receptor-β in endometriosis Endometriosis: where are we and where are we going?The elusive and controversial roles of estrogen and progesterone receptors in human endometriosis The dynamics of nuclear receptors and nuclear receptor coregulators in the pathogenesis of endometriosis.A novel mouse model of endometriosis mimics human phenotype and reveals insights into the inflammatory contribution of shed endometrium Relevant human tissue resources and laboratory models for use in endometriosis research.Increased susceptibility to hyperoxic lung injury and alveolar simplification in newborn rats by prenatal administration of benzo[a]pyrene.A novel role of the Sp/KLF transcription factor KLF11 in arresting progression of endometriosis.Estrogen-induced CCN1 is critical for establishment of endometriosis-like lesions in mice Inhibitor of apoptosis proteins (IAPs) may be effective therapeutic targets for treating endometriosis.Effect of exposure to atmospheric ultrafine particles on production of free fatty acids and lipid metabolites in the mouse small intestine Lipoxin A₄ prevents the progression of de novo and established endometriosis in a mouse model by attenuating prostaglandin E₂ production and estrogen signaling Exogenous activated NK cells enhance trafficking of endogenous NK cells to endometriotic lesions.The relationship among HOXA10, estrogen receptor α, progesterone receptor, and progesterone receptor B proteins in rectosigmoid endometriosis: a tissue microarray study Progesterone Alleviates Endometriosis via Inhibition of Uterine Cell Proliferation, Inflammation and Angiogenesis in an Immunocompetent Mouse Model.Estrogen Receptor β Modulates Apoptosis Complexes and the Inflammasome to Drive the Pathogenesis of Endometriosis Krüppel-Like Factor 13 Deficiency in Uterine Endometrial Cells Contributes to Defective Steroid Hormone Receptor Signaling but Not Lesion Establishment in a Mouse Model of Endometriosis The Dual Estrogen Receptor α Inhibitory Effects of the Tissue-Selective Estrogen Complex for Endometrial and Breast Safety.Multiple Beneficial Roles of Repressor of Estrogen Receptor Activity (REA) in Suppressing the Progression of Endometriosis.Dual suppression of estrogenic and inflammatory activities for targeting of endometriosis DNA methylation in endometriosis (Review).Increased Risk of Endometriosis in Patients With Lower Genital Tract Infection: A Nationwide Cohort Study.Oestrogen-induced angiogenesis and implantation contribute to the development of parasitic myomas after laparoscopic morcellation A single gestational exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin disrupts the adult uterine response to estradiol in mice.Krüppel-like factor 9 deficiency in uterine endometrial cells promotes ectopic lesion establishment associated with activated notch and hedgehog signaling in a mouse model of endometriosis.Ambient Ultrafine Particle Ingestion Alters Gut Microbiota in Association with Increased Atherogenic Lipid Metabolites.Activated AKT pathway promotes establishment of endometriosis.Self-renewal and phenotypic conversion are the main physiological responses of macrophages to the endogenous estrogen surge.Lesion development is modulated by the natural estrous cycle and mouse strain in a minimally invasive model of endometriosis.Early Endometriosis in Females Is Directed by Immune-Mediated Estrogen Receptor α and IL-6 Cross-Talk.Estrogen is essential but not sufficient to induce endometriosis.Lipopolysaccharide promotes the development of murine endometriosis-like lesions via the nuclear factor-kappa B pathway.Rodent Models of Experimental Endometriosis: Identifying Mechanisms of Disease and Therapeutic Targets.

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Role of estrogen receptor signaling required for endometriosis-like lesion establishment in a mouse model.

http://www.wikidata.org/entity/Q36114008

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2012 nî lūn-bûn

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Role of estrogen receptor sign ...... stablishment in a mouse model.

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Role of estrogen receptor sign ...... stablishment in a mouse model.

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Karina F Rodriguez

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Katherine A Burns

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Kyathanahalli S Janardhan

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Steven L Young

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P2860

A new mathematical model for relative quantification in real-time RT-PCR

Effect of single and compound knockouts of estrogen receptors alpha (ERalpha) and beta (ERbeta) on mouse reproductive phenotypes

Estrogens regulate neuroinflammatory genes via estrogen receptors α and β in the frontal cortex of middle-aged female rats

Estrogen Receptor Null Mice: What Have We Learned and Where Will They Lead Us?

Cytokine control in human endometrium.

Presence of estrogen receptor beta in the human endometrium through the cycle: expression in glandular, stromal, and vascular cells.

Prevalence of endometriosis in asymptomatic women.

Role of estrogen receptor-β in endometriosis

Insufficient luteinizing hormone-induced intracellular signaling disrupts ovulation in preovulatory follicles lacking estrogen receptor-{beta}.

Progesterone receptor isoform A but not B is expressed in endometriosis.

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3960-3971

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10.1210/EN.2012-1294

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