Selenium induces cholinergic motor neuron degeneration in Caenorhabditis elegans. - Wikidata - awgldk - TriplyDB

Selenium induces cholinergic motor neuron degeneration in Caenorhabditis elegans.

Selenium induces cholinergic motor neuron degeneration in Caenorhabditis elegans.

http://www.wikidata.org/entity/Q36243584

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Role of environmental contaminants in the etiology of Alzheimer's disease: a review Integrated control of protein degradation in C. elegans muscle Protective efficacy of selenite against lead-induced neurotoxicity in Caenorhabditis elegans.Assessing different mechanisms of toxicity in mountaintop removal/valley fill coal mining-affected watershed samples using Caenorhabditis elegans.Metal-induced neurodegeneration in C. elegans.Selenium in the Therapy of Neurological Diseases. Where is it Going?Degenerin channel activation causes caspase-mediated protein degradation and mitochondrial dysfunction in adult C. elegans muscle Exposure to environmental toxicants and pathogenesis of amyotrophic lateral sclerosis: state of the art and research perspectives.Cerebrospinal fluid of newly diagnosed amyotrophic lateral sclerosis patients exhibits abnormal levels of selenium species including elevated selenite.The neurodegenerative effects of selenium are inhibited by FOXO and PINK1/PTEN regulation of insulin/insulin-like growth factor signaling in Caenorhabditis elegans.Use of Caenorhabditis elegans as a model to study Alzheimer's disease and other neurodegenerative diseases.In vitro dopaminergic neurotoxicity of pesticides: a link with neurodegeneration?Selenium, selenoproteins and neurodegenerative diseases.Biological functions of selenium and its potential influence on Parkinson's disease.Knockdown of the C. elegans kinome identifies kinases required for normal protein homeostasis, mitochondrial network structure, and sarcomere structure in muscle.Using Multiple Phenotype Assays and Epistasis Testing to Enhance the Reliability of RNAi Screening and Identify Regulators of Muscle Protein Degradation.Protective role of DNJ-27/ERdj5 in Caenorhabditis elegans models of human neurodegenerative diseases Assessment of selenium toxicity on the life cycle of Caenorhabditis elegans.The Functions of Antioxidants and Heat Shock Proteins Are Altered in the Immune Organs of Selenium-Deficient Broiler Chickens.Toxicity evaluation of Wanzhou watershed of Yangtze Three Gorges Reservior in the flood season in Caenorhabditis elegans.

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P2860

Selenium induces cholinergic motor neuron degeneration in Caenorhabditis elegans.

http://www.wikidata.org/entity/Q36243584

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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2012年论文

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name

Selenium induces cholinergic motor neuron degeneration in Caenorhabditis elegans.

@ast

Selenium induces cholinergic motor neuron degeneration in Caenorhabditis elegans.

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type

label

Selenium induces cholinergic motor neuron degeneration in Caenorhabditis elegans.

@ast

Selenium induces cholinergic motor neuron degeneration in Caenorhabditis elegans.

@en

prefLabel

Selenium induces cholinergic motor neuron degeneration in Caenorhabditis elegans.

@ast

Selenium induces cholinergic motor neuron degeneration in Caenorhabditis elegans.

@en

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J.NEURO.2012.04.019

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NeuroToxicology

P1476

Selenium induces cholinergic motor neuron degeneration in Caenorhabditis elegans.

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Annette O Estevez

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Catherine L Mueller

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Kathleen L Morgan

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Luke Teece

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Miguel Estevez

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P2860

The Structure of the Nervous System of the Nematode Caenorhabditis elegans

Choline acetyltransferase-deficient mutants of the nematode Caenorhabditis elegans

The genetics of Caenorhabditis elegans

An ALS-linked mutant SOD1 produces a locomotor defect associated with aggregation and synaptic dysfunction when expressed in neurons of Caenorhabditis elegans

Drug resistance and neurotransmitter receptors of nematodes: recent studies on the mode of action of levamisole

The glutaredoxin GLRX-21 functions to prevent selenium-induced oxidative stress in Caenorhabditis elegans

Exon 1 of the HD gene with an expanded CAG repeat is sufficient to cause a progressive neurological phenotype in transgenic mice

The threshold for polyglutamine-expansion protein aggregation and cellular toxicity is dynamic and influenced by aging in Caenorhabditis elegans

Genetic and cellular basis for acetylcholine inhibition of Caenorhabditis elegans egg-laying behavior.

IDA-1, a Caenorhabditis elegans homolog of the diabetic autoantigens IA-2 and phogrin, is expressed in peptidergic neurons in the worm.

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1021-1032

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scholarly article

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10.1016/J.NEURO.2012.04.019

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5

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33

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Antonio Miranda-Vizuete

Nathaniel J. Szewczyk

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2012-04-25T00:00:00Z

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224913543

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22560997

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Caenorhabditis elegans

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