Natural killer cells: primary target for hepatitis C virus immune evasion strategies? - Wikidata - awgldk - TriplyDB

Natural killer cells: primary target for hepatitis C virus immune evasion strategies?

Natural killer cells: primary target for hepatitis C virus immune evasion strategies?

http://www.wikidata.org/entity/Q36405185

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Natural killer cells in hepatitis C: Current progress Emerging concepts in immunity to hepatitis C virus infection.Phenotypic and Functional Changes of Cytotoxic CD56pos Natural T Cells Determine Outcome of Acute Hepatitis C Virus Infection Hepatitis C and Innate Immunity: Recent Advances Adoptive immunotherapy with liver allograft–derived lymphocytes induces anti-HCV activity after liver transplantation in humans and humanized mice Effect of Killer Immunoglobulin-Like Receptors in the Response to Combined Treatment in Patients with Chronic Hepatitis C Virus Infection Regulation of host innate immunity by hepatitis C virus: crosstalk between hepatocyte and NK/DC Regulation of NK cell repertoire and function in the liver.Intrahepatic IL-10 maintains NKG2A+Ly49- liver NK cells in a functionally hyporesponsive state.Hepatitis C virus (HCV) evades NKG2D-dependent NK cell responses through NS5A-mediated imbalance of inflammatory cytokines.Natural killer cell function and dysfunction in hepatitis C virus infection.Innate immune function in placenta and cord blood of hepatitis C--seropositive mother-infant dyads.Human liver transplantation as a model to study hepatitis C virus pathogenesis Innate immune cell networking in hepatitis C virus infection The effect of donor race on the survival of Black Americans undergoing liver transplantation for chronic hepatitis C Assessment of immunological changes in Epstein-Barr virus co-infection in Egyptian chronic HCV patients.Innate immune genes synergize to predict increased risk of chronic disease in hepatitis C virus infection.NK cells, innate immunity and hepatitis C infection after liver transplantation.Dysfunction of Immune Systems and Host Genetic Factors in Hepatitis C Virus Infection with Persistent Normal ALT.Coinfection with HIV-1 and HCV--a one-two punch.Natural killer inhibitory receptor expression associated with treatment failure and interleukin-28B genotype in patients with chronic hepatitis C A novel approach to identify candidate prognostic factors for hepatitis C treatment response integrating clinical and viral genetic data.Glances in Immunology of HIV and HCV Infection Hepatitis viruses: live and let die.Natural killer cells: multifaceted players with key roles in hepatitis C immunity.Natural killer cell function is intact after direct exposure to infectious hepatitis C virions.KLRG1 negatively regulates natural killer cell functions through the Akt pathway in individuals with chronic hepatitis C virus infection.Liver natural killer and natural killer T cells: immunobiology and emerging roles in liver diseases.Activation of natural killer cells inhibits liver fibrosis: a novel strategy to treat liver fibrosis.Recurrent hepatitis C virus infection post liver transplantation: impact of choice of calcineurin inhibitor.Hepatitis C virus impairs natural killer cell-mediated augmentation of complement synthesis.MicroRNA-155 regulates interferon-γ production in natural killer cells via Tim-3 signalling in chronic hepatitis C virus infection.Immune modulation by the hepatitis C virus core protein.The effects of hepatitis C virus core protein on functional responses in the NK cell line YTS.Activation of natural killer cells by hepatitis C virus particles in vitro.A synergistic interferon-gamma production is induced by mouse hepatitis virus in interleukin-12 (IL-12)/IL-18-activated natural killer cells and modulated by carcinoembryonic antigen-related cell adhesion molecules (CEACAM) 1a receptor.Partial Activation of natural killer and γδ T cells by classical swine fever viruses is associated with type I interferon elicited from plasmacytoid dendritic cells.Immunopathogenesis of hepatitis C virus infection and hepatic fibrosis: New insights into antifibrotic therapy in chronic hepatitis C.Compromised function of natural killer cells in acute and chronic viral hepatitis.Increased frequency of CD56Bright NK-cells, CD3-CD16+CD56- NK-cells and activated CD4+T-cells or B-cells in parallel with CD4+CDC25High T-cells control potentially viremia in blood donors with HCV.

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P2860

Natural killer cells: primary target for hepatitis C virus immune evasion strategies?

http://www.wikidata.org/entity/Q36405185

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2006 nî lūn-bûn

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2006年學術文章

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2006年學術文章

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Natural killer cells: primary target for hepatitis C virus immune evasion strategies?

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Liver Transplantation

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Natural killer cells: primary target for hepatitis C virus immune evasion strategies?

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Hugo R Rosen

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Lucy Golden-Mason

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P2860

NK cell TRAIL eliminates immature dendritic cells in vivo and limits dendritic cell vaccination efficacy.

NK cell compartments and their activation by dendritic cells.

The HLA-A2 restricted T cell epitope HCV core 35-44 stabilizes HLA-E expression and inhibits cytolysis mediated by natural killer cells

Negative regulation of NK cell activities by inhibitory receptor CD94/NKG2A leads to altered NK cell-induced modulation of dendritic cell functions in chronic hepatitis C virus infection.

Cutting edge: increased NK cell activity in HIV-1-exposed but uninfected Vietnamese intravascular drug users.

NK cells cause liver injury and facilitate the induction of T cell-mediated immunity to a viral liver infection.

Autocrine/paracrine IL-15 that is required for type I IFN-mediated dendritic cell expression of MHC class I-related chain A and B is impaired in hepatitis C virus infection.

Selective impairments in dendritic cell-associated function distinguish hepatitis C virus and HIV infection.

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363-372

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10.1002/LT.20708

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3

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12

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Lucy Golden-Mason

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2006-03-01T00:00:00Z

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7278132

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16498647

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