Mpv17l protects against mitochondrial oxidative stress and apoptosis by activation of Omi/HtrA2 protease.
about
MPV17L2 is required for ribosome assembly in mitochondriaCOQ6 mutations in human patients produce nephrotic syndrome with sensorineural deafness.MicroRNAs as potential therapeutic targets in kidney diseaseAnti-microRNA-21 oligonucleotides prevent Alport nephropathy progression by stimulating metabolic pathwaysNetwork-based integration of molecular and physiological data elucidates regulatory mechanisms underlying adaptation to high-fat diet.A novel transcriptional repressor, Rhit, is involved in heat-inducible and age-dependent expression of Mpv17-like protein, a participant in reactive oxygen species metabolism.MicroRNAs are potential therapeutic targets in fibrosing kidney disease: lessons from animal models.The myriad essential roles of microRNAs in cardiovascular homeostasis and disease.The Human Mitochondrial DNA Depletion Syndrome Gene MPV17 Encodes a Non-selective Channel That Modulates Membrane Potential.Modulation of mitochondrial function and morphology by interaction of Omi/HtrA2 with the mitochondrial fusion factor OPA1.Renal gene and protein expression signatures for prediction of kidney disease progressionEffects of Low-Dose Diethylstilbestrol Exposure on DNA Methylation in Mouse SpermatocytesDistinct 3D Architecture and Dynamics of the Human HtrA2(Omi) Protease and Its Mutated Variants.Canine MPV17 truncation without clinical manifestations.Transforming growth factor-β, bioenergetics, and mitochondria in renal diseaseTemperature-induced changes of HtrA2(Omi) protease activity and structureMicroRNA-21 promotes fibrosis of the kidney by silencing metabolic pathwaysCardiac Specific Overexpression of Mitochondrial Omi/HtrA2 Induces Myocardial Apoptosis and Cardiac Dysfunction.Role of Bcl-2 family proteins and caspases in the regulation of apoptosis.Mitochondrial channels: ion fluxes and more.Computational genomic analysis of PARK7 interactome reveals high BBS1 gene expression as a prognostic factor favoring survival in malignant pleural mesothelioma.Mitochondrial Pathology and Glycolytic Shift during Proximal Tubule Atrophy after Ischemic AKI.Multiplicity-dependent activation of a serine protease-dependent cytomegalovirus-associated programmed cell death pathway.Mitochondria Damage and Kidney Disease.TrkAIII signals endoplasmic reticulum stress to the mitochondria in neuroblastoma cells, resulting in glycolytic metabolic adaptation.Missing mitochondrial Mpv17 gene function induces tissue-specific cell-death pathway in the degenerating inner ear.
P2860
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P2860
Mpv17l protects against mitochondrial oxidative stress and apoptosis by activation of Omi/HtrA2 protease.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Mpv17l protects against mitoch ...... ivation of Omi/HtrA2 protease.
@en
type
label
Mpv17l protects against mitoch ...... ivation of Omi/HtrA2 protease.
@en
prefLabel
Mpv17l protects against mitoch ...... ivation of Omi/HtrA2 protease.
@en
P2093
P2860
P356
P1476
Mpv17l protects against mitoch ...... tivation of Omi/HtrA2 protease
@en
P2093
Christian Faul
Erwin P Böttinger
Peter Mundel
Shaolin Shi
Stefanie Krick
P2860
P304
14106-14111
P356
10.1073/PNAS.0801146105
P407
P50
P577
2008-09-04T00:00:00Z