Mechanisms underlying the inability to induce area CA1 LTP in the mouse after traumatic brain injury.
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Making Waves in the Brain: What Are Oscillations, and Why Modulating Them Makes Sense for Brain InjurySensory cortex underpinnings of traumatic brain injury deficitsRole of NMDA Receptor-Mediated Glutamatergic Signaling in Chronic and Acute NeuropathologiesAugmented Inhibition from Cannabinoid-Sensitive Interneurons Diminishes CA1 Output after Traumatic Brain Injury.Blocking leukotriene synthesis attenuates the pathophysiology of traumatic brain injury and associated cognitive deficitsExperimental mild traumatic brain injury induces functional alteration of the developing hippocampusAlterations in neuronal calcium levels are associated with cognitive deficits after traumatic brain injury.Repeated mild traumatic brain injury causes chronic neuroinflammation, changes in hippocampal synaptic plasticity, and associated cognitive deficits.Traumatic brain injury-induced ependymal ciliary loss decreases cerebral spinal fluid flow.Hippocampal θ dysfunction after lateral fluid percussion injury.Excitatory synaptic transmission and network activity are depressed following mechanical injury in cortical neuronsLong-Term Potentiation at CA3-CA1 Hippocampal Synapses with Special Emphasis on Aging, Disease, and Stress.Delayed reduction in hippocampal postsynaptic density protein-95 expression temporally correlates with cognitive dysfunction following controlled cortical impact in mice.Mild traumatic brain injury and diffuse axonal injury in swine.Efficacy, dosage, and duration of action of branched chain amino Acid therapy for traumatic brain injury.Calcium/calmodulin-dependent protein kinase II (CaMKII) inhibition induces neurotoxicity via dysregulation of glutamate/calcium signaling and hyperexcitability.Altered calcium signaling following traumatic brain injuryMapping the Connectome Following Traumatic Brain Injury.Decoding hippocampal signaling deficits after traumatic brain injuryInvestigations on alterations of hippocampal circuit function following mild traumatic brain injuryPhosphodiesterase inhibition rescues chronic cognitive deficits induced by traumatic brain injuryTheta burst stimulation to characterize changes in brain plasticity following mild traumatic brain injury: A proof-of-principle studyNeurotrophin-mediated neuroprotection of hippocampal neurons following traumatic brain injury is not associated with acute recovery of hippocampal functionDeficits in ERK and CREB activation in the hippocampus after traumatic brain injury.Controlled contusion injury alters molecular systems associated with cognitive performance.Single-neuron NMDA receptor phenotype influences neuronal rewiring and reintegration following traumatic injury.Diffuse traumatic brain injury and the sensory brain.Cognitive impairment, genomic instability and trace elements.Concussion Induces Hippocampal Circuitry Disruption in Swine.Pathophysiology and Treatment of Memory Dysfunction After Traumatic Brain Injury.Monitoring hippocampus electrical activity in vitro on an elastically deformable microelectrode array.Traumatic Brain Injuries during Development: Implications for Alcohol Abuse.Inhibition of the integrated stress response reverses cognitive deficits after traumatic brain injuryPrimary blast injury causes cognitive impairments and hippocampal circuit alterations.Isolated Primary Blast Inhibits Long-Term Potentiation in Organotypic Hippocampal Slice Cultures.Calcium-permeable AMPA receptors appear in cortical neurons after traumatic mechanical injury and contribute to neuronal fate.The new pyridoindole antioxidant SMe1EC2 and its intervention in hypoxia/hypoglycemia-induced impairment of longterm potentiation in rat hippocampus.Pycnogenol protects CA3-CA1 synaptic function in a rat model of traumatic brain injuryEphB3 signaling propagates synaptic dysfunction in the traumatic injured brainXuefu zhuyu decoction improves cognitive impairment in experimental traumatic brain injury via synaptic regulation.
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Mechanisms underlying the inability to induce area CA1 LTP in the mouse after traumatic brain injury.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on January 2006
@en
vedecký článok
@sk
vetenskaplig artikel
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videnskabelig artikel
@da
vědecký článek
@cs
name
Mechanisms underlying the inab ...... after traumatic brain injury.
@en
Mechanisms underlying the inab ...... after traumatic brain injury.
@nl
type
label
Mechanisms underlying the inab ...... after traumatic brain injury.
@en
Mechanisms underlying the inab ...... after traumatic brain injury.
@nl
prefLabel
Mechanisms underlying the inab ...... after traumatic brain injury.
@en
Mechanisms underlying the inab ...... after traumatic brain injury.
@nl
P2093
P2860
P356
P1433
P1476
Mechanisms underlying the inab ...... after traumatic brain injury.
@en
P2093
D P Bonislawski
E Schwarzbach
P2860
P304
P356
10.1002/HIPO.20183
P577
2006-01-01T00:00:00Z