Induction of CHOP expression by amino acid limitation requires both ATF4 expression and ATF2 phosphorylation.
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TRB3, a novel ER stress-inducible gene, is induced via ATF4-CHOP pathway and is involved in cell deathATF4 protects against neuronal death in cellular Parkinson's disease models by maintaining levels of parkinC/EBP homology protein (CHOP) interacts with activating transcription factor 4 (ATF4) and negatively regulates the stress-dependent induction of the asparagine synthetase geneCharacterization of the amino acid response element within the human sodium-coupled neutral amino acid transporter 2 (SNAT2) System A transporter geneThe p300/CBP-associated factor (PCAF) is a cofactor of ATF4 for amino acid-regulated transcription of CHOPCommon features and interesting differences in transcriptional responses to secretion stress in the fungi Trichoderma reesei and Saccharomyces cerevisiaeUnfolded Protein Response and Macroautophagy in Alzheimer's, Parkinson's and Prion DiseasesRole of the repressor JDP2 in the amino acid-regulated transcription of CHOPUpregulating Noxa by ER stress, celastrol exerts synergistic anti-cancer activity in combination with ABT-737 in human hepatocellular carcinoma cellsExpression of hereditary hemochromatosis C282Y HFE protein in HEK293 cells activates specific endoplasmic reticulum stress responses.Transcriptional regulation of the human growth hormone receptor (hGHR) gene V2 promoter by transcriptional activators and repressor.Decreased ATF4 expression as a mechanism of acquired resistance to long-term amino acid limitation in cancer cellsRestoration of visual function in P23H rhodopsin transgenic rats by gene delivery of BiP/Grp78Amino acid availability controls TRB3 transcription in liver through the GCN2/eIF2α/ATF4 pathwayFamilial CJD associated PrP mutants within transmembrane region induced Ctm-PrP retention in ER and triggered apoptosis by ER stress in SH-SY5Y cells.Role of Endoplasmic Reticulum Stress in Learning and Memory Impairment and Alzheimer's Disease-Like Neuropathology in the PS19 and APPSwe Mouse Models of Tauopathy and Amyloidosis.Regulation of CCAAT/enhancer-binding protein homologous protein (CHOP) expression by interleukin-1 beta in pancreatic beta cellsThe unfolded protein response and the phosphorylations of activating transcription factor 2 in the trans-activation of il23a promoter produced by β-glucansRole of endoplasmic reticulum stress in acrolein-induced endothelial activationERK/ribosomal S6 kinase (RSK) signaling positively regulates death receptor 5 expression through co-activation of CHOP and Elk1Stress-induced translation of ATF5 mRNA is regulated by the 5'-untranslated region.Recruitment of SWI/SNF complex is required for transcriptional activation of the SLC11A1 gene during macrophage differentiation of HL-60 cellsMAPK signaling triggers transcriptional induction of cFOS during amino acid limitation of HepG2 cells.Abnormal chondrocyte apoptosis in the cartilage growth plate is influenced by genetic background and deletion of CHOP in a targeted mouse model of pseudoachondroplasiaAuto-activation of c-JUN gene by amino acid deprivation of hepatocellular carcinoma cells reveals a novel c-JUN-mediated signaling pathway.DDIT3 and KAT2A Proteins Regulate TNFRSF10A and TNFRSF10B Expression in Endoplasmic Reticulum Stress-mediated Apoptosis in Human Lung Cancer Cells(Ctm)PrP and ER stress: a neurotoxic mechanism of some special PrP mutants.ATF2 is required for amino acid-regulated transcription by orchestrating specific histone acetylation.Differential regulation of CHOP-10/GADD153 gene expression by MAPK signaling in pancreatic beta-cells.Lysine and Leucine Deficiencies Affect Myocytes Development and IGF Signaling in Gilthead Sea Bream (Sparus aurata).The Chondroprotective Role of TMF in PGE2-Induced Apoptosis Associating with Endoplasmic Reticulum Stress.Nutritional control of gene expression: how mammalian cells respond to amino acid limitation.ATF2 - at the crossroad of nuclear and cytosolic functions.The LMP1 oncogene of EBV activates PERK and the unfolded protein response to drive its own synthesis.ATF4 is an oxidative stress-inducible, prodeath transcription factor in neurons in vitro and in vivo.ATF4-dependent transcription mediates signaling of amino acid limitation.Specificity of amino acid regulated gene expression: analysis of genes subjected to either complete or single amino acid deprivationDifferential control of the CCAAT/enhancer-binding protein beta (C/EBPbeta) products liver-enriched transcriptional activating protein (LAP) and liver-enriched transcriptional inhibitory protein (LIP) and the regulation of gene expression during theThe transcription factor network associated with the amino acid response in mammalian cells.Eukaryotic initiation factor 2 phosphorylation and translational control in metabolism.
P2860
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P2860
Induction of CHOP expression by amino acid limitation requires both ATF4 expression and ATF2 phosphorylation.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Induction of CHOP expression b ...... sion and ATF2 phosphorylation.
@en
type
label
Induction of CHOP expression b ...... sion and ATF2 phosphorylation.
@en
prefLabel
Induction of CHOP expression b ...... sion and ATF2 phosphorylation.
@en
P2093
P2860
P356
P1476
Induction of CHOP expression b ...... sion and ATF2 phosphorylation.
@en
P2093
Alain Bruhat
Céline Jousse
Gerald Thiel
Julien Averous
Pierre Fafournoux
Valérie Carraro
P2860
P304
P356
10.1074/JBC.M311862200
P407
P577
2003-12-01T00:00:00Z