Bone histomorphometry in glucocorticoid-induced osteoporosis.
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Bone disease in newly diagnosed lupus nephritis patientsCurrent and potential future drug treatments for osteoporosis.Kidney transplantation with early corticosteroid withdrawal: paradoxical effects at the central and peripheral skeleton.Bone loss and glucocorticoid therapy in patients with respiratory disease.Intermittent parathyroid hormone administration counteracts the adverse effects of glucocorticoids on osteoblast and osteocyte viability, bone formation, and strength in mice.Bisphosphonates for steroid induced osteoporosis.'Down to skin and bone'.Reduced fracture risk with early corticosteroid withdrawal after kidney transplant.Prevention and management of osteoporosis: consensus statements from the Scientific Advisory Board of the Osteoporosis Society of Canada. 6. Use of bisphosphonates in the treatment of osteoporosis.An N-ethyl-N-nitrosourea induced corticotropin-releasing hormone promoter mutation provides a mouse model for endogenous glucocorticoid excessA sheep model for fracture treatment in osteoporosis: benefits of the model versus animal welfare.Should bisphosphonates be used for long-term treatment of glucocorticoid-induced osteoporosis?Promotion of osteoclast survival and antagonism of bisphosphonate-induced osteoclast apoptosis by glucocorticoids.Prevention of glucocorticoid induced-apoptosis of osteoblasts and osteocytes by protecting against endoplasmic reticulum (ER) stress in vitro and in vivo in female mice.Management of bone disease in patients on long term glucocorticoid therapy.A double blind placebo controlled study to determine the effects of intermittent cyclical etidronate on bone mineral density in patients on long-term oral corticosteroid treatment.Osteocyte viability with glucocorticoid treatment: relation to histomorphometryDisorders associated with acute rapid and severe bone loss.Glucocorticoid-treated mice have localized changes in trabecular bone material properties and osteocyte lacunar size that are not observed in placebo-treated or estrogen-deficient mice.IL-6 Contributes to the Defective Osteogenesis of Bone Marrow Stromal Cells from the Vertebral Body of the Glucocorticoid-Induced Osteoporotic Mouse.Mechanisms of action of mycophenolate mofetil.Corticosteroids and osteoporosisNew and improved glucocorticoid receptor ligands.Therapy Insight: osteoporosis and osteonecrosis in systemic lupus erythematosus.Comparative effects of teriparatide and risedronate in glucocorticoid-induced osteoporosis in men: 18-month results of the EuroGIOPs trialGlucocorticoid-induced bone loss in mice can be reversed by the actions of parathyroid hormone and risedronate on different pathways for bone formation and mineralizationGlucocorticoids and osteocyte autophagy.Management of glucocorticoids-induced osteoporosis: role of teriparatide.Glucocorticoid impairs cell-cell communication by autophagy-mediated degradation of connexin 43 in osteocytes.Inhibition of osteoblastogenesis and promotion of apoptosis of osteoblasts and osteocytes by glucocorticoids. Potential mechanisms of their deleterious effects on boneStrain dependent differences in glucocorticoid-induced bone loss between C57BL/6J and CD-1 mice.Glucocorticoid excess in mice results in early activation of osteoclastogenesis and adipogenesis and prolonged suppression of osteogenesis: a longitudinal study of gene expression in bone tissue from glucocorticoid-treated mice.Minireview: live and let die: molecular effects of glucocorticoids on bone cells.Mechanical induction of PGE2 in osteocytes blocks glucocorticoid-induced apoptosis through both the β-catenin and PKA pathways.Molecular Actions of Glucocorticoids in Cartilage and Bone During Health, Disease, and Steroid Therapy.Edaravone protects osteoblastic cells from dexamethasone through inhibiting oxidative stress and mPTP opening.Protecting Bone Health in Pediatric Rheumatic Diseases: Pharmacological Considerations.Transforming Growth Factor Beta is regulated by a Glucocorticoid-Dependent Mechanism in Denervation Mouse Bone.Glucocorticoids suppress bone formation via the osteoclast.Glucocorticoids regulate the expression of the human osteoblastic endothelin A receptor gene.
P2860
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P2860
Bone histomorphometry in glucocorticoid-induced osteoporosis.
description
1989 nî lūn-bûn
@nan
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
1989年论文
@zh
1989年论文
@zh-cn
name
Bone histomorphometry in glucocorticoid-induced osteoporosis.
@en
type
label
Bone histomorphometry in glucocorticoid-induced osteoporosis.
@en
prefLabel
Bone histomorphometry in glucocorticoid-induced osteoporosis.
@en
P356
P1476
Bone histomorphometry in glucocorticoid-induced osteoporosis.
@en
P2093
Dempster DW
P304
P356
10.1002/JBMR.5650040202
P577
1989-04-01T00:00:00Z