Cholesterol- and sphingolipid-rich microdomains are essential for microtubule-based membrane protrusions induced by Clostridium difficile transferase (CDT).
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The Regulatory Networks That Control Clostridium difficile Toxin SynthesisHighly Selective Anti-Cancer Activity of Cholesterol-Interacting Agents Methyl-β-Cyclodextrin and Ostreolysin A/Pleurotolysin B Protein Complex on Urothelial Cancer CellsEffects of plasma membrane cholesterol level and cytoskeleton F-actin on cell protrusion mechanicsSelection of nanobodies that block the enzymatic and cytotoxic activities of the binary Clostridium difficile toxin CDTAn Integrated Metabolomic and Microbiome Analysis Identified Specific Gut Microbiota Associated with Fecal Cholesterol and Coprostanol in Clostridium difficile InfectionClostridium difficile toxins: mediators of inflammation.Septins guide microtubule protrusions induced by actin-depolymerizing toxins like Clostridium difficile transferase (CDT).Clostridial binary toxins: iota and C2 family portraits.Hype or hypervirulence: a reflection on problematic C. difficile strains.De Novo sphingolipid synthesis is essential for Salmonella-induced autophagy and human beta-defensin 2 expression in intestinal epithelial cells.Perturbation to Cholesterol at the Neuromuscular Junction confers botulinum neurotoxin A sensitivity to neonatal mice.Host Cell Chaperones Hsp70/Hsp90 and Peptidyl-Prolyl Cis/Trans Isomerases Are Required for the Membrane Translocation of Bacterial ADP-Ribosylating Toxins.Overexpression of the Endosomal Anion/Proton Exchanger ClC-5 Increases Cell Susceptibility toward Clostridium difficile Toxins TcdA and TcdB.Interaction of the Clostridium difficile Binary Toxin CDT and Its Host Cell Receptor, Lipolysis-stimulated Lipoprotein Receptor (LSR).Formation of Nanotube-Like Protrusions, Regulation of Septin Organization and Re-guidance of Vesicle Traffic by Depolymerization of the Actin Cytoskeleton Induced by Binary Bacterial Protein Toxins.The role of cholesterol-sphingomyelin membrane nanodomains in the stability of intercellular membrane nanotubes.Identification of the cellular receptor of Clostridium spiroforme toxin.The Role of Sphingolipids on Innate Immunity to Intestinal Salmonella Infection.Clostridium difficile binary toxin CDT induces clustering of the lipolysis-stimulated lipoprotein receptor into lipid rafts.Loss of LSR affects epithelial barrier integrity and tumor xenograft growth of CaCo-2 cellsThe role of toxins in Clostridium difficile infection.
P2860
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P2860
Cholesterol- and sphingolipid-rich microdomains are essential for microtubule-based membrane protrusions induced by Clostridium difficile transferase (CDT).
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Cholesterol- and sphingolipid- ...... m difficile transferase (CDT).
@en
type
label
Cholesterol- and sphingolipid- ...... m difficile transferase (CDT).
@en
prefLabel
Cholesterol- and sphingolipid- ...... m difficile transferase (CDT).
@en
P2093
P2860
P356
P1476
Cholesterol- and sphingolipid- ...... m difficile transferase (CDT).
@en
P2093
Alexander E Lang
Anna S Kruppke
Carsten Schwan
Daniel M Schubert
P2860
P304
29356-29365
P356
10.1074/JBC.M111.261925
P407
P577
2011-06-25T00:00:00Z