Molecular mechanism of cardiac cellular hypertrophy by mechanical stress.
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Effects of concentric and eccentric contractions on phosphorylation of MAPK(erk1/2) and MAPK(p38) in isolated rat skeletal muscleDisruption of integrin function in the murine myocardium leads to perinatal lethality, fibrosis, and abnormal cardiac performanceMitogen-activated protein kinases and mitogen-activated protein kinase phosphatases mediate the inhibitory effects of all-trans retinoic acid on the hypertrophic growth of cardiomyocytesEmbryonic cardiomyocyte hypoplasia and craniofacial defects in G alpha q/G alpha 11-mutant mice.Differential Signaling and Hypertrophic Responses in Cyclically Stretched vs Endothelin-1 Stimulated Neonatal Rat Cardiomyocytes.A role for the p38 mitogen-activated protein kinase pathway in myocardial cell growth, sarcomeric organization, and cardiac-specific gene expression.Electrical stimulation of neonatal cardiomyocytes results in the sequential activation of nuclear genes governing mitochondrial proliferation and differentiationInvestigation of the bnp level changes in blood stream in different modes and lead locations after pacemaker implementation.Signaling pathways mediating cardiac myocyte gene expression in physiological and stress responses.Role of mitogen-activated protein kinase in cardiac hypertrophy and heart failureEssential roles for early growth response transcription factor Egr-1 in tissue fibrosis and wound healing.Fibroblasts contracting collagen matrices form transient plasma membrane passages through which the cells take up fluorescein isothiocyanate-dextran and Ca2+Poor sleep and impaired self-care: towards a comprehensive model linking sleep, cognition, and heart failure outcomes.Role of differential signaling pathways and oxidative stress in diabetic cardiomyopathy.Mechano-sensing by actin filaments and focal adhesion proteins.Early left ventricular gene expression profile in response to increase in blood pressure.Increased c-fos mRNA expression by human fibroblasts contracting stressed collagen matrices.Signal transduction of mechanical stress in myocytes and fibroblasts derived from neonatal rat ventriclesPattern of superoxide dismutase enzymatic activity and RNA changes in rat heart ventricles after myocardial infarction.Activation of catalase by apelin prevents oxidative stress-linked cardiac hypertrophy.Phenylephrine protects neonatal rat cardiomyocytes from hypoxia and serum deprivation-induced apoptosis.Glucocorticoid regulation of angiotensin-converting enzyme in primary culture of adult cardiac fibroblasts.MKK6 activates myocardial cell NF-kappaB and inhibits apoptosis in a p38 mitogen-activated protein kinase-dependent manner.Atrial natriuretic peptide induces apoptosis in neonatal rat cardiac myocytes.Early sequence of cardiac adaptations and growth factor formation in pressure- and volume-overload hypertrophy.On-chip assessment of human primary cardiac fibroblasts proliferative responses to uniaxial cyclic mechanical strain.Microstructured cocultures of cardiac myocytes and fibroblasts: a two-dimensional in vitro model of cardiac tissue
P2860
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P2860
Molecular mechanism of cardiac cellular hypertrophy by mechanical stress.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
Molecular mechanism of cardiac cellular hypertrophy by mechanical stress.
@en
type
label
Molecular mechanism of cardiac cellular hypertrophy by mechanical stress.
@en
prefLabel
Molecular mechanism of cardiac cellular hypertrophy by mechanical stress.
@en
P2093
P1476
Molecular mechanism of cardiac cellular hypertrophy by mechanical stress.
@en
P2093
P304
P356
10.1016/S0022-2828(08)80013-2
P577
1995-01-01T00:00:00Z