α2δ-3 Is Required for Rapid Transsynaptic Homeostatic Signaling.
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Rab Interacting Molecules 2 and 3 Directly Interact with the Pore-Forming CaV1.3 Ca2+ Channel Subunit and Promote Its Membrane Expression.Injury-induced maladaptation and dysregulation of calcium channel α2 δ subunit proteins and its contribution to neuropathic pain development.Synaptic Homeostasis and Its Immunological Disturbance in Neuromuscular Junction Disorders.LRP1 influences trafficking of N-type calcium channels via interaction with the auxiliary α2δ-1 subunit.Retrograde semaphorin-plexin signalling drives homeostatic synaptic plasticity.The Maintenance of Synaptic Homeostasis at the Drosophila Neuromuscular Junction Is Reversible and Sensitive to High Temperature.A postsynaptic Pi3K-CII dependent signaling controller for presynaptic homeostatic plasticity.Drosophila Studies on Autism Spectrum Disorders.Thrombospondin-4 acts on pre-synaptic calcium channel alpha-2-delta-1 subunit to induce synaptogenesis between sensory and spinal cord neurons through a T-type calcium channel dependent pathway that is blocked by early, low-dose gabapentin treatmentSynapse-specific and compartmentalized expression of presynaptic homeostatic potentiation.Coupling the Structural and Functional Assembly of Synaptic Release Sites
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α2δ-3 Is Required for Rapid Transsynaptic Homeostatic Signaling.
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2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年学术文章
@wuu
2016年学术文章
@zh-cn
2016年学术文章
@zh-hans
2016年学术文章
@zh-my
2016年学术文章
@zh-sg
2016年學術文章
@yue
2016年學術文章
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2016年學術文章
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name
α2δ-3 Is Required for Rapid Transsynaptic Homeostatic Signaling.
@en
type
label
α2δ-3 Is Required for Rapid Transsynaptic Homeostatic Signaling.
@en
prefLabel
α2δ-3 Is Required for Rapid Transsynaptic Homeostatic Signaling.
@en
P2093
P2860
P1433
P1476
α2δ-3 Is Required for Rapid Transsynaptic Homeostatic Signaling.
@en
P2093
Graeme W Davis
Jenna M Whippen
Ryan T Jones
Tingting Wang
P2860
P304
P356
10.1016/J.CELREP.2016.08.030
P577
2016-09-01T00:00:00Z