Diet restriction inhibits apoptosis and HMGB1 oxidation and promotes inflammatory cell recruitment during acetaminophen hepatotoxicity. - Wikidata - awgldk - TriplyDB

Diet restriction inhibits apoptosis and HMGB1 oxidation and promotes inflammatory cell recruitment during acetaminophen hepatotoxicity.

Diet restriction inhibits apoptosis and HMGB1 oxidation and promotes inflammatory cell recruitment during acetaminophen hepatotoxicity.

http://www.wikidata.org/entity/Q41963390

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High mobility group box protein 1 (HMGB1)-partner molecule complexes enhance cytokine production by signaling through the partner molecule receptor Novel in vitro and mathematical models for the prediction of chemical toxicity Cell death in the pathogenesis of immune-mediated diseases: the role of HMGB1 and DAMP-PAMP complexes Ethyl pyruvate is a novel anti-inflammatory agent to treat multiple inflammatory organ injuries The HMGB1-RAGE Inflammatory Pathway: Implications for Brain Injury-Induced Pulmonary Dysfunction Patterns of acetaminophen use exceeding 4 grams daily in a hospitalized population at a tertiary care center The many faces of HMGB1: molecular structure-functional activity in inflammation, apoptosis, and chemotaxis.The role of high mobility group box 1 in innate immunity.Molecular forms of HMGB1 and keratin-18 as mechanistic biomarkers for mode of cell death and prognosis during clinical acetaminophen hepatotoxicity High mobility group box-1 (HMGB1) participates in the pathogenesis of alcoholic liver disease (ALD).Sequestering HMGB1 via DNA-conjugated beads ameliorates murine colitis High mobility group B1 impairs hepatocyte regeneration in acetaminophen hepatotoxicity Biomarkers distinguish apoptotic and necrotic cell death during hepatic ischemia/reperfusion injury in mice.HMGB1 in health and disease.Role of the Nalp3 inflammasome in acetaminophen-induced sterile inflammation and liver injury.High systemic levels of the cytokine-inducing HMGB1 isoform secreted in severe macrophage activation syndrome.Mouse strain-dependent caspase activation during acetaminophen hepatotoxicity does not result in apoptosis or modulation of inflammation.Redox modification of cysteine residues regulates the cytokine activity of high mobility group box-1 (HMGB1)Identification of a functional interaction of HMGB1 with Receptor for Advanced Glycation End-products in a model of neuropathic pain.Target biomarker profile for the clinical management of paracetamol overdose.Acetaminophen hepatotoxicity and repair: the role of sterile inflammation and innate immunity.Models of drug-induced liver injury for evaluation of phytotherapeutics and other natural products.Characterization of the Inflammatory Properties of Actively Released HMGB1 in Juvenile Idiopathic Arthritis.Fas receptor-deficient lpr mice are protected against acetaminophen hepatotoxicity due to higher glutathione synthesis and enhanced detoxification of oxidant stress.Receptor interacting protein kinase 3 is a critical early mediator of acetaminophen-induced hepatocyte necrosis in mice.Plasma biomarkers of liver injury and inflammation demonstrate a lack of apoptosis during obstructive cholestasis in mice Emerging role of high-mobility group box 1 (HMGB1) in liver diseases.A signature of renal stress resistance induced by short-term dietary restriction, fasting, and protein restriction.Character and temporal evolution of apoptosis in acetaminophen-induced acute liver failure*.Oxidant stress, mitochondria, and cell death mechanisms in drug-induced liver injury: lessons learned from acetaminophen hepatotoxicity Key Events Participating in the Pathogenesis of Alcoholic Liver Disease.HMGB1: a multifunctional alarmin driving autoimmune and inflammatory disease.High-mobility group box-1 in sterile inflammation.Nidovirus-Associated Proliferative Pneumonia in the Green Tree Python (Morelia viridis).Mechanistic Modelling of Drug-Induced Liver Injury: Investigating the Role of Innate Immune Responses.Recent advances in biomarkers and therapeutic interventions for hepatic drug safety - false dawn or new horizon?Transcriptional regulation of Mcl-1 plays an important role of cellular protective effector of vincristine-triggered autophagy in oral cancer cells.HMGB1 and Histones Play a Significant Role in Inducing Systemic Inflammation and Multiple Organ Dysfunctions in Severe Acute Pancreatitis.High mobility group box protein 1 in complex with lipopolysaccharide or IL-1 promotes an increased inflammatory phenotype in synovial fibroblasts.Macrophage-derived IL-1α promotes sterile inflammation in a mouse model of acetaminophen hepatotoxicity.

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P2860

Diet restriction inhibits apoptosis and HMGB1 oxidation and promotes inflammatory cell recruitment during acetaminophen hepatotoxicity.

http://www.wikidata.org/entity/Q41963390

description

2010 nî lūn-bûn

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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2010年论文

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2010年论文

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name

Diet restriction inhibits apop ...... acetaminophen hepatotoxicity.

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type

label

Diet restriction inhibits apop ...... acetaminophen hepatotoxicity.

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prefLabel

Diet restriction inhibits apop ...... acetaminophen hepatotoxicity.

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MOLMED.2010.00126

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Molecular Medicine

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Diet restriction inhibits apop ...... acetaminophen hepatotoxicity.

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Anja Kipar

http://www.w3.org/2001/XMLSchema#string

B Kevin Park

http://www.w3.org/2001/XMLSchema#string

Daniel James Antoine

http://www.w3.org/2001/XMLSchema#string

Dominic P Williams

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Hugh Laverty

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P2860

Induction of immunological tolerance by apoptotic cells requires caspase-dependent oxidation of high-mobility group box-1 protein

A critical cysteine is required for HMGB1 binding to Toll-like receptor 4 and activation of macrophage cytokine release

Release of chromatin protein HMGB1 by necrotic cells triggers inflammation

HMG-1 as a late mediator of endotoxin lethality in mice

N-acetyl-p-benzoquinone imine: a cytochrome P-450-mediated oxidation product of acetaminophen

The receptor for advanced glycation end products (RAGE) is a cellular binding site for amphoterin. Mediation of neurite outgrowth and co-expression of rage and amphoterin in the developing nervous system

High mobility group 1 protein (HMG-1) stimulates proinflammatory cytokine synthesis in human monocytes

How dying cells alert the immune system to danger

Blockade of high mobility group box-1 protein attenuates experimental severe acute pancreatitis.

Acetaminophen-induced hepatic necrosis. IV. Protective role of glutathione.

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