A mathematical model of electrotonic interactions between ventricular myocytes and fibroblasts.
about
Computational Representations of Myocardial Infarct Scars and Implications for ArrhythmogenesisComputational Approaches to Understanding the Role of Fibroblast-Myocyte Interactions in Cardiac ArrhythmogenesisModeling Electrophysiological Coupling and Fusion between Human Mesenchymal Stem Cells and CardiomyocytesCross talk between cardiac myocytes and fibroblasts: from multiscale investigative approaches to mechanisms and functional consequencesSpiral-wave dynamics in a mathematical model of human ventricular tissue with myocytes and fibroblasts.OptoDyCE as an automated system for high-throughput all-optical dynamic cardiac electrophysiology.Electrophysiological and structural remodeling in heart failure modulate arrhythmogenesis. 2D simulation studyNonequilibrium arrhythmic states and transitions in a mathematical model for diffuse fibrosis in human cardiac tissueA Computational Study of the Factors Influencing the PVC-Triggering Ability of a Cluster of Early Afterdepolarization-Capable MyocytesConnexin43 contributes to electrotonic conduction across scar tissue in the intact heart.A Mathematical Model of Neonatal Rat Atrial Monolayers with Constitutively Active Acetylcholine-Mediated K+ CurrentComputational modeling of cardiac fibroblasts and fibrosisElectrotonic myofibroblast-to-myocyte coupling increases propensity to reentrant arrhythmias in two-dimensional cardiac monolayers.Size and ionic currents of unexcitable cells coupled to cardiomyocytes distinctly modulate cardiac action potential shape and pacemaking activity in micropatterned cell pairsMathematical approaches to understanding and imaging atrial fibrillation: significance for mechanisms and managementSlow Conduction in the Border Zones of Patchy Fibrosis Stabilizes the Drivers for Atrial Fibrillation: Insights from Multi-Scale Human Atrial Modeling.Local Gradients in Electrotonic Loading Modulate the Local Effective Refractory Period: Implications for Arrhythmogenesis in the Infarct Border Zone.Mkk4 is a negative regulator of the transforming growth factor beta 1 signaling associated with atrial remodeling and arrhythmogenesis with age.Novel therapeutic strategies targeting fibroblasts and fibrosis in heart disease.Fibroblast electrical remodeling in heart failure and potential effects on atrial fibrillationMolecular determinants of cardiac fibroblast electrical function and therapeutic implications for atrial fibrillation.Susceptibility to arrhythmia in the infarcted heart depends on myofibroblast density.Electrophysiological and structural remodeling in heart failure modulate arrhythmogenesis. 1D simulation study.Arrhythmogenic consequences of myofibroblast-myocyte couplingLoading effect of fibroblast-myocyte coupling on resting potential, impulse propagation, and repolarization: insights from a microstructure model.The role of fibroblasts in complex fractionated electrograms during persistent/permanent atrial fibrillation: implications for electrogram-based catheter ablation.Enhanced sensitivity of aged fibrotic hearts to angiotensin II- and hypokalemia-induced early afterdepolarization-mediated ventricular arrhythmias.Field and action potential recordings in heart slices: correlation with established in vitro and in vivo models.Modelling cardiac fibroblasts: interactions with myocytes and their impact on impulse propagationIslands of spatially discordant APD alternans underlie arrhythmogenesis by promoting electrotonic dyssynchrony in models of fibrotic rat ventricular myocardium.Fibroblast KATP currents modulate myocyte electrophysiology in infarcted hearts.Myocyte-fibroblast communication in cardiac fibrosis and arrhythmias: Mechanisms and model systems.Stem cell therapies for heart disease: why do we need bioengineers?Mechanistic inquiry into the role of tissue remodeling in fibrotic lesions in human atrial fibrillation.Natriuretic peptide C receptor signalling in the heart and vasculature.Excitation-contraction coupling between human atrial myocytes with fibroblasts and stretch activated channel current: a simulation studyDynamical characterization of inactivation path in voltage-gated Na(+) ion channel by non-equilibrium response spectroscopy.Electrotonic loading of anisotropic cardiac monolayers by unexcitable cells depends on connexin type and expression levelCardiac alternans induced by fibroblast-myocyte coupling: mechanistic insights from computational models.Myofibroblasts Electrotonically Coupled to Cardiomyocytes Alter Conduction: Insights at the Cellular Level from a Detailed In silico Tissue Structure Model.
P2860
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P2860
A mathematical model of electrotonic interactions between ventricular myocytes and fibroblasts.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
A mathematical model of electr ...... ular myocytes and fibroblasts.
@en
A mathematical model of electr ...... ular myocytes and fibroblasts.
@nl
type
label
A mathematical model of electr ...... ular myocytes and fibroblasts.
@en
A mathematical model of electr ...... ular myocytes and fibroblasts.
@nl
prefLabel
A mathematical model of electr ...... ular myocytes and fibroblasts.
@en
A mathematical model of electr ...... ular myocytes and fibroblasts.
@nl
P2093
P2860
P1433
P1476
A mathematical model of electr ...... ular myocytes and fibroblasts.
@en
P2093
Hojjat Bazzazi
K Andrew MacCannell
Robert B Clark
Wayne R Giles
Yoshiyuki Shibukawa
P2860
P304
P356
10.1529/BIOPHYSJ.106.101410
P407
P50
P577
2007-02-16T00:00:00Z