In situ hybridization for the detection of telomerase RNA in the progression from Barrett's esophagus to esophageal adenocarcinoma.
about
Variations in telomere maintenance and the role of telomerase inhibition in gastrointestinal cancerBiology of telomeres: importance in etiology of esophageal cancer and as therapeutic targetRisk factors for neoplastic progression in Barrett's esophagusMolecular changes in the progression of Barrett's oesophagusMalignant transformation of non-neoplastic Barrett's epithelial cells through well-defined genetic manipulationsMolecular biology of Barrett's adenocarcinoma.Telomere maintenance in laser capture microdissection-purified Barrett's adenocarcinoma cells and effect of telomerase inhibition in vivo.Molecular evolution of the metaplasia-dysplasia-adenocarcinoma sequence in the esophagusThe molecular basis of carcinogenesis in Barrett's esophagus.Biology of Barrett's esophagus and esophageal adenocarcinomaZoning of mucosal phenotype, dysplasia, and telomerase activity measured by telomerase repeat assay protocol in Barrett's esophagusObserver variation in the diagnosis of superficial oesophageal adenocarcinoma.Progressive transformation of immortalized esophageal epithelial cellsTelomere and telomerase in the initial stage of immortalization of esophageal epithelial cell.Esophageal adenocarcinoma arising in Barrett esophagusLong Noncoding RNAs in the Pathogenesis of Barrett's Esophagus and Esophageal Carcinoma.Quantitation of telomerase components and hTERT mRNA splicing patterns in immortal human cells.Both transcriptional and posttranscriptional mechanisms regulate human telomerase template RNA levels.Characterisation of telomerase immortalised normal human oesophageal squamous cellsThe regulation of telomerase in oncogenesis.Cellular senescence in gastrointestinal diseases: from pathogenesis to therapeutics.Epidermal growth factor receptor (EGFR) is overexpressed in high-grade dysplasia and adenocarcinoma of the esophagus and may represent a biomarker of histological progression in Barrett's esophagus (BE).
P2860
Q26775381-326AD727-4EEB-43DF-A79A-EB208658E0ADQ26829993-91CE6A66-2D5F-4BFA-854E-14DF2154A37AQ27002062-35805544-9010-4A66-94E4-DFAF9D01B72CQ28219148-717AAD1E-694C-4028-87DA-F00EFBB45482Q28749063-9F97D11E-D52A-48F5-BD82-035682D8B211Q30327935-368CD2B3-9171-4AF7-853C-B180B6E1ABF1Q33357296-5A99BB86-1DFE-47F8-9322-FC4C9FCEFAEBQ33611018-BBF54416-2394-4849-B4B4-2F7508BCDB59Q33862429-CD1E9DBD-ABE1-4D80-BFDB-8621360D262CQ34644324-46A4F3D8-28E0-4818-84FE-605FA0BC585EQ34787887-0A46CA17-57AE-41DE-B016-5FA7ED439692Q34960762-0BBBD75D-877F-4E21-9F0A-D95F02FC3EDBQ36308468-91715178-8523-42D3-8C7B-E60A9E254CADQ36316137-202BDE23-EB2B-4CEB-8CC6-93124D47D821Q37169303-A6C3ED0F-8084-4859-B034-75A25F70A1C9Q39320641-009AED5C-4DF0-4654-A827-DBF9C27DD7B5Q39511317-4CFAD19F-9739-4E79-9D4F-63FC1CF86387Q39551372-3EAEF768-F40F-4D2F-B7C4-C43C5CB86574Q40670991-BDF64D39-768C-4E0B-968E-A8226CA98DB9Q42152999-2BCD8FE2-82A1-43C6-B121-086BA78E2496Q49565626-A603F98D-7A21-4D6D-ADB3-7C98BFDCDB09Q53754804-610A6AE6-3AAE-4994-84DD-FE8CDE22C075
P2860
In situ hybridization for the detection of telomerase RNA in the progression from Barrett's esophagus to esophageal adenocarcinoma.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
1998年论文
@zh
1998年论文
@zh-cn
name
In situ hybridization for the ...... to esophageal adenocarcinoma.
@en
In situ hybridization for the ...... to esophageal adenocarcinoma.
@nl
type
label
In situ hybridization for the ...... to esophageal adenocarcinoma.
@en
In situ hybridization for the ...... to esophageal adenocarcinoma.
@nl
prefLabel
In situ hybridization for the ...... to esophageal adenocarcinoma.
@en
In situ hybridization for the ...... to esophageal adenocarcinoma.
@nl
P2093
P1433
P1476
In situ hybridization for the ...... to esophageal adenocarcinoma.
@en
P2093
P304
P356
10.1002/(SICI)1097-0142(19980815)83:4<652::AID-CNCR4>3.3.CO;2-4
P407
P577
1998-08-01T00:00:00Z