Downregulation of miR-16 via URGCP pathway contributes to glioma growth.

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Downregulation of miR-16 via URGCP pathway contributes to glioma growth.

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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Downregulation of miR-16 via URGCP pathway contributes to glioma growth.

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Downregulation of miR-16 via URGCP pathway contributes to glioma growth.

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Downregulation of miR-16 via URGCP pathway contributes to glioma growth.

@en

Downregulation of miR-16 via URGCP pathway contributes to glioma growth.

@nl

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Downregulation of miR-16 via URGCP pathway contributes to glioma growth.

@en

Downregulation of miR-16 via URGCP pathway contributes to glioma growth.

@nl

P1476

Downregulation of miR-16 via URGCP pathway contributes to glioma growth.

@en

P2093

Chen Ying

http://www.w3.org/2001/XMLSchema#string

Cui Hao

http://www.w3.org/2001/XMLSchema#string

Liang Hong

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Ouyang Qing

http://www.w3.org/2001/XMLSchema#string

Xu Lunshan

http://www.w3.org/2001/XMLSchema#string

Xu Minhui

http://www.w3.org/2001/XMLSchema#string

Zhang Chengqu

http://www.w3.org/2001/XMLSchema#string

Zhou Ji

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P2860

Downregulation of the tumor-suppressor miR-16 via progestin-mediated oncogenic signaling contributes to breast cancer development

Exciting new advances in neuro-oncology: the avenue to a cure for malignant glioma

Widespread microRNA repression by Myc contributes to tumorigenesis

Truncation in CCND1 mRNA alters miR-16-1 regulation in mantle cell lymphoma

Enhanced cell survival of gastric cancer cells by a novel gene URG4

Hepatitis Bx antigen stimulates expression of a novel cellular gene, URG4, that promotes hepatocellular growth and survival

c-Myc-regulated microRNAs modulate E2F1 expression

The RelA NF-kappaB subunit and the aryl hydrocarbon receptor (AhR) cooperate to transactivate the c-myc promoter in mammary cells

DLEU2, frequently deleted in malignancy, functions as a critical host gene of the cell cycle inhibitory microRNAs miR-15a and miR-16-1

Diagnosis and treatment of recurrent high-grade astrocytoma.

P2888

s41598-017-14035-2

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13470

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scholarly article

P356

10.1038/S41598-017-14035-2

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P407

English

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P478

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P577

2017-10-18T00:00:00Z

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29044221

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P932

5647380

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