Activation of Gi induces mechanical hyperalgesia poststress or inflammation. - Wikidata - awgldk - TriplyDB

Activation of Gi induces mechanical hyperalgesia poststress or inflammation.

Activation of Gi induces mechanical hyperalgesia poststress or inflammation.

http://www.wikidata.org/entity/Q43140160

about

Commonalities between pain and memory mechanisms and their meaning for understanding chronic pain Enhanced cytokine-induced mechanical hyperalgesia in skeletal muscle produced by a novel mechanism in rats exposed to unpredictable sound stress.A-Kinase Anchoring Protein 79/150 Scaffolds Transient Receptor Potential A 1 Phosphorylation and Sensitization by Metabotropic Glutamate Receptor Activation.Shared mechanisms for opioid tolerance and a transition to chronic pain.Accounting for the delay in the transition from acute to chronic pain: axonal and nuclear mechanisms Mitochondrial dependence of nerve growth factor-induced mechanical hyperalgesia Distinct terminal and cell body mechanisms in the nociceptor mediate hyperalgesic priming Acidosis Mediates the Switching of Gs-PKA and Gi-PKCε Dependence in Prolonged Hyperalgesia Induced by Inflammation.Generation of a pain memory in the primary afferent nociceptor triggered by PKCε activation of CPEB.Mas-Related Gene (Mrg) C Activation Attenuates Bone Cancer Pain via Modulating Gi and NR2B Repeated Mu-Opioid Exposure Induces a Novel Form of the Hyperalgesic Priming Model for Transition to Chronic Pain.Transient decrease in nociceptor GRK2 expression produces long-term enhancement in inflammatory pain The pharmacology of nociceptor priming Serotonin Receptor 2B Mediates Mechanical Hyperalgesia by Regulating Transient Receptor Potential Vanilloid 1 Role of nociceptor αCaMKII in transition from acute to chronic pain (hyperalgesic priming) in male and female rats.Protein kinase Cε is required for spinal analgesic synergy between delta opioid and alpha-2A adrenergic receptor agonist pairs.Adenosine-A1 receptor agonist induced hyperalgesic priming type II.Electrophysiological correlates of hyperalgesic priming in vitro and in vivo The fundamental unit of pain is the cell.Critical role of nociceptor plasticity in chronic pain.Second messengers mediating the expression of neuroplasticity in a model of chronic pain in the rat.Roles of Proton-Sensing Receptors in the Transition from Acute to Chronic Pain.Gi-protein-coupled 5-HT1B/D receptor agonist sumatriptan induces type I hyperalgesic priming.Role of a novel nociceptor autocrine mechanism in chronic pain.Age-dependent sexual dimorphism in susceptibility to develop chronic pain in the rat.Regulation of Expression of Hyperalgesic Priming by Estrogen Receptor α in the Rat.An Integrative Neuroscience Framework for the Treatment of Chronic Pain: From Cellular Alterations to Behavior.

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P2860

Activation of Gi induces mechanical hyperalgesia poststress or inflammation.

http://www.wikidata.org/entity/Q43140160

description

2009 nî lūn-bûn

@nan

2009年の論文

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2009年学术文章

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2009年学术文章

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2009年学术文章

@zh-cn

2009年学术文章

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2009年学术文章

@zh-my

2009年学术文章

@zh-sg

2009年學術文章

@yue

2009年學術文章

@zh-hant

name

Activation of Gi induces mechanical hyperalgesia poststress or inflammation.

@en

Activation of Gi induces mechanical hyperalgesia poststress or inflammation.

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type

label

Activation of Gi induces mechanical hyperalgesia poststress or inflammation.

@en

Activation of Gi induces mechanical hyperalgesia poststress or inflammation.

@nl

prefLabel

Activation of Gi induces mechanical hyperalgesia poststress or inflammation.

@en

Activation of Gi induces mechanical hyperalgesia poststress or inflammation.

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J.NEUROSCIENCE.2009.03.001

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Activation of Gi induces mechanical hyperalgesia poststress or inflammation.

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J D Levine

http://www.w3.org/2001/XMLSchema#string

O A Dina

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R W Gear

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S G Khasar

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P2860

Assembly of an A kinase-anchoring protein-beta(2)-adrenergic receptor complex facilitates receptor phosphorylation and signaling

A protein kinase C translocation inhibitor as an isozyme-selective antagonist of cardiac function

Role of interleukin-6 in chronic muscle hyperalgesic priming

Arachidonic acid protects neonatal rat cardiac myocytes from ischaemic injury through epsilon protein kinase C.

The molecular basis for distinct beta-adrenergic receptor subtype actions in cardiomyocytes.

Involvement of Gs and Gi proteins in dual coupling of the luteinizing hormone receptor to adenylyl cyclase and phospholipase C.

Cardiac-specific overexpression of human beta2 adrenoceptors in mice exposes coupling to both Gs and Gi proteins

The cardiac beta2-adrenergic signalling a new role for the cPLA2.

Molecular mechanisms of neuropathic pain-phenotypic switch and initiation mechanisms.

Alterations in adrenergic receptor signaling in heart failure.

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