Activation of Gi induces mechanical hyperalgesia poststress or inflammation.
about
Commonalities between pain and memory mechanisms and their meaning for understanding chronic painEnhanced cytokine-induced mechanical hyperalgesia in skeletal muscle produced by a novel mechanism in rats exposed to unpredictable sound stress.A-Kinase Anchoring Protein 79/150 Scaffolds Transient Receptor Potential A 1 Phosphorylation and Sensitization by Metabotropic Glutamate Receptor Activation.Shared mechanisms for opioid tolerance and a transition to chronic pain.Accounting for the delay in the transition from acute to chronic pain: axonal and nuclear mechanismsMitochondrial dependence of nerve growth factor-induced mechanical hyperalgesiaDistinct terminal and cell body mechanisms in the nociceptor mediate hyperalgesic primingAcidosis Mediates the Switching of Gs-PKA and Gi-PKCε Dependence in Prolonged Hyperalgesia Induced by Inflammation.Generation of a pain memory in the primary afferent nociceptor triggered by PKCε activation of CPEB.Mas-Related Gene (Mrg) C Activation Attenuates Bone Cancer Pain via Modulating Gi and NR2BRepeated Mu-Opioid Exposure Induces a Novel Form of the Hyperalgesic Priming Model for Transition to Chronic Pain.Transient decrease in nociceptor GRK2 expression produces long-term enhancement in inflammatory painThe pharmacology of nociceptor primingSerotonin Receptor 2B Mediates Mechanical Hyperalgesia by Regulating Transient Receptor Potential Vanilloid 1Role of nociceptor αCaMKII in transition from acute to chronic pain (hyperalgesic priming) in male and female rats.Protein kinase Cε is required for spinal analgesic synergy between delta opioid and alpha-2A adrenergic receptor agonist pairs.Adenosine-A1 receptor agonist induced hyperalgesic priming type II.Electrophysiological correlates of hyperalgesic priming in vitro and in vivoThe fundamental unit of pain is the cell.Critical role of nociceptor plasticity in chronic pain.Second messengers mediating the expression of neuroplasticity in a model of chronic pain in the rat.Roles of Proton-Sensing Receptors in the Transition from Acute to Chronic Pain.Gi-protein-coupled 5-HT1B/D receptor agonist sumatriptan induces type I hyperalgesic priming.Role of a novel nociceptor autocrine mechanism in chronic pain.Age-dependent sexual dimorphism in susceptibility to develop chronic pain in the rat.Regulation of Expression of Hyperalgesic Priming by Estrogen Receptor α in the Rat.An Integrative Neuroscience Framework for the Treatment of Chronic Pain: From Cellular Alterations to Behavior.
P2860
Q28083722-BE87D461-7FF3-4194-B36B-0700ADEFACFEQ30464076-3F2C41E6-8859-40BC-B00C-CB5E422B7498Q33686921-9D463BBF-5394-47FC-BCBB-995D73C69C10Q33803874-3FAE8689-8C5F-4B88-BF59-802A6A1A0503Q34946158-A257F31B-2FB2-4A71-A79E-80D50CE4CBA1Q35180285-2761EF38-C40C-4E32-8EEA-5F381E8D108BQ35423741-EE7C9AA7-27D2-4B49-9BC9-A43592CE906DQ35560825-AB91F2FB-F900-4D0C-A794-BCC4150E2330Q35833776-5B8564C5-A920-4068-A063-46E19E65A73CQ36010727-DCEA28F7-B833-4EE1-9CF6-916574EC5DE4Q36038255-D24CCA76-CFBA-4D4C-ADAC-1BA882F869FBQ36207533-3A1552FF-DCE3-454E-9D53-B5A5D890E25DQ36332325-89BD6489-86F3-4918-992F-A950AAECF3D3Q36853422-4E0E1F2F-19EB-447D-9829-31E13111B18EQ37032440-703EA3D5-50A4-4009-896D-3CA97C30C8D5Q37093923-5516437F-0C2C-4741-83E0-123B7E3D92DCQ37308329-96844DA2-0133-47B7-A49B-B0F013496442Q37336340-C2C1B8F0-CE84-4287-8D08-F867D5532F9DQ37381403-64A44C29-1B60-4139-9E35-2D7F31472A01Q37603560-56C2D65C-481C-4A3A-81D7-71A5DF2E0A88Q37704685-F684E93B-779D-49A5-BC5C-3D7702D836A0Q38644749-B7CB6A98-EFC4-4468-953A-CFF26E44A5AFQ41300169-C52079BC-4768-4C97-84C1-405556A2A28EQ46337882-0893F76F-F0E7-431D-8F2F-66A559C43A2CQ46485544-FA2E7793-B954-4895-A0E3-79844B82D724Q46755470-1A0F68CD-7975-46FC-B971-72EAC2EBDB3AQ55159501-0F9F75D2-3F5E-4636-BACB-B385763452EF
P2860
Activation of Gi induces mechanical hyperalgesia poststress or inflammation.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh-hant
name
Activation of Gi induces mechanical hyperalgesia poststress or inflammation.
@en
Activation of Gi induces mechanical hyperalgesia poststress or inflammation.
@nl
type
label
Activation of Gi induces mechanical hyperalgesia poststress or inflammation.
@en
Activation of Gi induces mechanical hyperalgesia poststress or inflammation.
@nl
prefLabel
Activation of Gi induces mechanical hyperalgesia poststress or inflammation.
@en
Activation of Gi induces mechanical hyperalgesia poststress or inflammation.
@nl
P2093
P2860
P1433
P1476
Activation of Gi induces mechanical hyperalgesia poststress or inflammation.
@en
P2093
P2860
P304
P356
10.1016/J.NEUROSCIENCE.2009.03.001
P407
P577
2009-03-09T00:00:00Z