Essential role of MD-2 in TLR4-dependent signaling during Helicobacter pylori-associated gastritis. - Wikidata - awgldk - TriplyDB

Essential role of MD-2 in TLR4-dependent signaling during Helicobacter pylori-associated gastritis.

Essential role of MD-2 in TLR4-dependent signaling during Helicobacter pylori-associated gastritis.

http://www.wikidata.org/entity/Q43450468

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A20 is an early responding negative regulator of Toll-like receptor 5 signalling in intestinal epithelial cells during inflammation Induction of microRNA-155 is TLR- and type IV secretion system-dependent in macrophages and inhibits DNA-damage induced apoptosis Pattern-recognition receptors and gastric cancer Interplay of the Gastric Pathogen Helicobacter pylori with Toll-Like Receptors Increased expression of toll-like receptors (TLR) 2, 4 and 5 in gastric dysplasia Interaction of Toll-like receptors with bacterial components induces expression of CDX2 and MUC2 in rat biliary epithelium in vivo and in culture Genetic polymorphisms in TLR1, TLR2, TLR4, and TLR10 of Helicobacter pylori-associated gastritis: a prospective cross-sectional study in Thailand.Asthma is inversely associated with Helicobacter pylori status in an urban population.Pathogen recognition receptors, cancer and inflammation in the gut.Helicobacter pylori infection of gastrointestinal epithelial cells in vitro induces mesenchymal stem cell migration through an NF-κB-dependent pathway Role of innate immunity in Helicobacter pylori-induced gastric malignancy.Bacterial interactions with cells of the intestinal mucosa: Toll-like receptors and NOD2 Helicobacter pylori regulates TLR4 and TLR9 during gastric carcinogenesis.Let-7b is involved in the inflammation and immune responses associated with Helicobacter pylori infection by targeting Toll-like receptor 4.The role of TLR2, TLR4 and CD14 genetic polymorphisms in gastric carcinogenesis: a case-control study and meta-analysis.TLR9 promoter polymorphism is associated with both an increased susceptibility to gastric carcinoma and poor prognosis.Helicobacter pylori persistence: an overview of interactions between H. pylori and host immune defenses Effect of Helicobacter pylori eradication on TLR2 and TLR4 expression in patients with gastric lesions.H. pylori exploits and manipulates innate and adaptive immune cell signaling pathways to establish persistent infection.Effects of Two Common Polymorphisms rs2910164 in miR-146a and rs11614913 in miR-196a2 on Gastric Cancer Susceptibility.Probiotic BIFICO cocktail ameliorates Helicobacter pylori induced gastritis.Deficiencies of myeloid differentiation factor 88, Toll-like receptor 2 (TLR2), or TLR4 produce specific defects in macrophage cytokine secretion induced by Helicobacter pylori Ceramide and Toll-like receptor 4 are mobilized into membrane rafts in response to Helicobacter pylori infection in gastric epithelial cells.Compartmentalizing intestinal epithelial cell toll-like receptors for immune surveillance The influence of cytokine gene polymorphisms on the risk of developing gastric cancer in patients with Helicobacter pylori infection.Implication of antigenic conversion of Helicobacter pylori lipopolysaccharides that involve interaction with surfactant protein D.Intestinal epithelial cell signalling and host-derived negative regulators under chronic inflammation: to be or not to be activated determines the balance towards commensal bacteria.TLR4 and TLR2 expression in biopsy specimens from antral and corporal stomach zones in Helicobacter pylori infections.Suppression of Toll-like receptor 4 activation by caffeic acid phenethyl ester is mediated by interference of LPS binding to MD2.Role of Toll-like receptors in gastrointestinal malignancies.Conventional, regulatory, and unconventional T cells in the immunologic response to Helicobacter pylori.Life in the human stomach: persistence strategies of the bacterial pathogen Helicobacter pylori.Activation of TLR4 signaling promotes gastric cancer progression by inducing mitochondrial ROS production CD74 in antigen presentation, inflammation, and cancers of the gastrointestinal tract Lipopolysaccharide-squamous cell carcinoma-monocyte interactions induce cancer-supporting factors leading to rapid STAT3 activation.Should a Toll-like receptor 4 (TLR-4) agonist or antagonist be designed to treat cancer? TLR-4: its expression and effects in the ten most common cancers Toll-like receptors and their role in gastrointestinal disease.Toll-like receptors as therapeutic targets in gastrointestinal diseases.Cytotoxic T cells in H. pylori-related gastric autoimmunity and gastric lymphoma.Role of virulence factors and host cell signaling in the recognition of Helicobacter pylori and the generation of immune responses.

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Essential role of MD-2 in TLR4-dependent signaling during Helicobacter pylori-associated gastritis.

http://www.wikidata.org/entity/Q43450468

description

2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年學術文章

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2004年學術文章

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name

Essential role of MD-2 in TLR4 ...... r pylori-associated gastritis.

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Essential role of MD-2 in TLR4 ...... r pylori-associated gastritis.

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type

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Essential role of MD-2 in TLR4 ...... r pylori-associated gastritis.

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Essential role of MD-2 in TLR4 ...... r pylori-associated gastritis.

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prefLabel

Essential role of MD-2 in TLR4 ...... r pylori-associated gastritis.

@en

Essential role of MD-2 in TLR4 ...... r pylori-associated gastritis.

@nl

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JIMMUNOL.173.2.1406

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Journal of Immunology

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Essential role of MD-2 in TLR4 ...... r pylori-associated gastritis.

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Cesar Francisco Ortega-Cava

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Hideaki Kazumori

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Mohammad Azharul Karim Rumi

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Nagisa Yoshino

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Norihisa Ishimura

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Shunji Ishihara

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Takafumi Yuki

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Yasunori Kadowaki

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Yoshikazu Kinoshita

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Youichi Miyaoka

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1406-1416

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scholarly article

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10.4049/JIMMUNOL.173.2.1406

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2004-07-01T00:00:00Z

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Helicobacter pylori

Toll-like receptor