IgA nephropathy, the most common cause of glomerulonephritis, is linked to 6q22-23.
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The IgA nephropathy Biobank. An important starting point for the genetic dissection of a complex trait.Recent advances in the understanding and management of IgA nephropathyNew developments in the genetics, pathogenesis, and therapy of IgA nephropathyGenetic studies of IgA nephropathy: what have we learned from genome-wide association studiesThe Genetics of IgA Nephropathy: An Overview from Western CountriesThe Genetics of IgA Nephropathy: An Overview from ChinaA genome-wide association study in Han Chinese identifies multiple susceptibility loci for IgA nephropathyA panoramic view of gene expression in the human kidneyDo the mutations of C1GALT1C1 gene play important roles in the genetic susceptibility to Chinese IgA nephropathy?Application of Proteomic Analysis to Renal Disease in the Clinic.The genetics and immunobiology of IgA nephropathy.Familial IgA nephropathy in southeastern Kentucky.Genetic basis of nondiabetic end-stage renal diseaseGenetic studies of IgA nephropathy: past, present, and future.Genetics and immunopathogenesis of IgA nephropathy.Genome-wide association study identifies susceptibility loci for IgA nephropathy.The genetic variants at the HLA-DRB1 gene are associated with primary IgA nephropathy in Han Chinese.Geographic differences in genetic susceptibility to IgA nephropathy: GWAS replication study and geospatial risk analysis.Finding genetic contributions to sporadic disease: a recessive locus at 12q24 commonly contributes to patent ductus arteriosus.HLA has strongest association with IgA nephropathy in genome-wide analysisUMOD polymorphism rs12917707 is not associated with severe or stable IgA nephropathy in a large Caucasian cohort.Association between family members of dialysis patients and chronic kidney disease: a multicenter study in China.Increased plasma sVCAM-1 is associated with severity in IgA nephropathy.Aberrantly glycosylated IgA1 as a factor in the pathogenesis of IgA nephropathyAberrant IgA1 glycosylation is inherited in familial and sporadic IgA nephropathy.Elucidating heterogeneity of IgA1 hinge-region O-glycosylation by use of MALDI-TOF/TOF mass spectrometry: role of cysteine alkylation during sample processing.Efficacy and safety of Shenyankangfu tablets for primary glomerulonephritis: study protocol for a randomized controlled trialUncoupling of glomerular IgA deposition and disease progression in alymphoplasia mice with IgA nephropathyGenetics of kidney failure and the evolving story of APOL1.Genetic heterogeneity in Italian families with IgA nephropathy: suggestive linkage for two novel IgA nephropathy lociAssociation of the CD14 gene -159C polymorphism with progression of IgA nephropathy.Genome-wide scan identifies a copy number variable region at 3p21.1 that influences the TLR9 expression levels in IgA nephropathy patientsGalectin-8 in IgA nephritis: decreased binding of IgA by galectin-8 affinity chromatography and associated increased binding in non-IgA serum glycoproteins.Development of a model of early-onset IgA nephropathyAssociation of a polymorphism of BTN2A1 with chronic kidney disease in individuals with or without hypertension or diabetes mellitusAssociation of genetic variants with chronic kidney disease in Japanese individuals with or without hypertension or diabetes mellitus.BCL2 family of apoptosis-related genes: functions and clinical implications in cancer.IL5RA and TNFRSF6B gene variants are associated with sporadic IgA nephropathyA SPRY2 mutation leading to MAPK/ERK pathway inhibition is associated with an autosomal dominant form of IgA nephropathyThe pathogenic role of IgA1 O-linked glycosylation in the pathogenesis of IgA nephropathy.
P2860
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P2860
IgA nephropathy, the most common cause of glomerulonephritis, is linked to 6q22-23.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年学术文章
@wuu
2000年学术文章
@zh-cn
2000年学术文章
@zh-hans
2000年学术文章
@zh-my
2000年学术文章
@zh-sg
2000年學術文章
@yue
2000年學術文章
@zh
2000年學術文章
@zh-hant
name
IgA nephropathy, the most common cause of glomerulonephritis, is linked to 6q22-23.
@en
IgA nephropathy, the most common cause of glomerulonephritis, is linked to 6q22-23.
@nl
type
label
IgA nephropathy, the most common cause of glomerulonephritis, is linked to 6q22-23.
@en
IgA nephropathy, the most common cause of glomerulonephritis, is linked to 6q22-23.
@nl
prefLabel
IgA nephropathy, the most common cause of glomerulonephritis, is linked to 6q22-23.
@en
IgA nephropathy, the most common cause of glomerulonephritis, is linked to 6q22-23.
@nl
P2093
P50
P356
P1433
P1476
IgA nephropathy, the most common cause of glomerulonephritis, is linked to 6q22-23.
@en
P2093
Gharavi AG
Nelson-Williams C
P2888
P304
P356
10.1038/81677
P407
P577
2000-11-01T00:00:00Z