Inflammatory components in human Alzheimer's disease and after active amyloid-β42 immunization. - Wikidata - awgldk - TriplyDB

Inflammatory components in human Alzheimer's disease and after active amyloid-β42 immunization.

Inflammatory components in human Alzheimer's disease and after active amyloid-β42 immunization.

http://www.wikidata.org/entity/Q44833066

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Fluid Biomarkers in Clinical Trials of Alzheimer's Disease Therapeutics Immune phenotypes of microglia in human neurodegenerative disease: challenges to detecting microglial polarization in human brains Neuroinflammation in Alzheimer's disease R47H Variant of TREM2 Associated With Alzheimer Disease in a Large Late-Onset Family: Clinical, Genetic, and Neuropathological Study Targeting neuroinflammation in Alzheimer's disease Neuroinflammation in Alzheimer's disease Interactions between inflammation, sex steroids, and Alzheimer's disease risk factors Reprogramming of HUVECs into induced pluripotent stem cells (HiPSCs), generation and characterization of HiPSC-derived neurons and astrocytes Receptor for advanced glycation endproduct modulators: a new therapeutic target in Alzheimer's disease.Microglia of prefrontal white matter in suicide Amyloid β and Tau Alzheimer's disease related pathology is reduced by Toll-like receptor 9 stimulation.A critique of the drug discovery and phase 3 clinical programs targeting the amyloid hypothesis for Alzheimer disease.Non-steroidal anti-inflammatory drugs as a treatment for Alzheimer's disease: a systematic review and meta-analysis of treatment effect.A retrospective analysis of the Alzheimer's disease vaccine progress - The critical need for new development strategies.Association of CD33 polymorphism rs3865444 with Alzheimer's disease pathology and CD33 expression in human cerebral cortex Post-mortem analysis of neuroinflammatory changes in human Alzheimer's disease.RNA-sequencing reveals transcriptional up-regulation of Trem2 in response to bexarotene treatment.APOE Stabilization by Exercise Prevents Aging Neurovascular Dysfunction and Complement Induction.Young microglia restore amyloid plaque clearance of aged microglia.Progesterone treatment reduces neuroinflammation, oxidative stress and brain damage and improves long-term outcomes in a rat model of repeated mild traumatic brain injury.Plaque-Associated Local Toxicity Increases over the Clinical Course of Alzheimer Disease.Microglial immunophenotype in dementia with Alzheimer's pathology.The mixed-lineage kinase 3 inhibitor URMC-099 facilitates microglial amyloid-β degradation APOE Genotype Differentially Modulates Effects of Anti-Aβ, Passive Immunization in APP Transgenic Mice.Innate Immunity Stimulation via Toll-Like Receptor 9 Ameliorates Vascular Amyloid Pathology in Tg-SwDI Mice with Associated Cognitive Benefits.Biomarker modelling of early molecular changes in Alzheimer's disease.Alzheimer disease and cellular mechanisms of memory storage.New roles for Fc receptors in neurodegeneration-the impact on Immunotherapy for Alzheimer's Disease.Adaptive lymphocyte profiles correlate to brain Aβ burden in patients with mild cognitive impairment.Central nervous system myeloid cells as drug targets: current status and translational challenges.Does the Interplay Between Aging and Neuroinflammation Modulate Alzheimer's Disease Clinical Phenotypes? A Clinico-Pathological Perspective.Amylin Treatment Reduces Neuroinflammation and Ameliorates Abnormal Patterns of Gene Expression in the Cerebral Cortex of an Alzheimer's Disease Mouse Model.Hippocampal T cell infiltration promotes neuroinflammation and cognitive decline in a mouse model of tauopathy Hippocampal proteomics defines pathways associated with memory decline and resilience in normal aging and Alzheimer's disease mouse models.Innate immunity in Alzheimer's disease: the relevance of animal models?Amylin and its G-protein-coupled receptor: A probable pathological process and drug target for Alzheimer's disease.Therapeutic Potential of Intravenous Immunoglobulin in Acute Brain Injury.Ginsenoside Rg1 protects against neuronal degeneration induced by chronic dexamethasone treatment by inhibiting NLRP-1 inflammasomes in mice.Targeting Apolipoprotein E/Amyloid β Binding by Peptoid CPO_Aβ17-21 P Ameliorates Alzheimer's Disease Related Pathology and Cognitive Decline.Deficiency of TYROBP, an adapter protein for TREM2 and CR3 receptors, is neuroprotective in a mouse model of early Alzheimer's pathology.

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Inflammatory components in human Alzheimer's disease and after active amyloid-β42 immunization.

http://www.wikidata.org/entity/Q44833066

description

2013 nî lūn-bûn

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2013年の論文

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Inflammatory components in hum ...... tive amyloid-β42 immunization.

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Inflammatory components in hum ...... tive amyloid-β42 immunization.

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Inflammatory components in hum ...... tive amyloid-β42 immunization.

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Inflammatory components in hum ...... tive amyloid-β42 immunization.

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Inflammatory components in hum ...... tive amyloid-β42 immunization.

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Inflammatory components in hum ...... tive amyloid-β42 immunization.

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Inflammatory components in hum ...... tive amyloid-β42 immunization.

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Elina Zotova

http://www.w3.org/2001/XMLSchema#string

James A R Nicoll

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James W Neal

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Matthew Cheaveau

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Scott Harris

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Seth Love

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Viraj Bharambe

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William Morgan

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P2860

The Alzheimer's disease-associated amyloid beta-protein is an antimicrobial peptide

TREM2 Variants in Alzheimer's Disease

Variant of TREM2 Associated with the Risk of Alzheimer's Disease

Actin-binding proteins coronin-1a and IBA-1 are effective microglial markers for immunohistochemistry

Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease

Inflammation and Alzheimer's disease

Subacute meningoencephalitis in a subset of patients with AD after Abeta42 immunization

Iba1 is an actin-cross-linking protein in macrophages/microglia

Fcgamma receptors: old friends and new family members

Alzheimer's disease: paired helical filaments and cytomembranes

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10.1093/BRAIN/AWT210

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Pt 9

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136

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23943781

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Alzheimer's disease