Hyperammonaemia in V1a vasopressin receptor knockout mice caused by the promoted proteolysis and reduced intrahepatic blood volume.
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Correction of hyponatraemia improves cognition, quality of life, and brain oedema in cirrhosis.The metabolic syndromeIndocyanine green clearance varies as a function of N-acetylcysteine treatment in a murine model of acetaminophen toxicity.A(a)LS: Ammonia-induced amyotrophic lateral sclerosis.Vasopressin: a novel target for the prevention and retardation of kidney disease?
P2860
Hyperammonaemia in V1a vasopressin receptor knockout mice caused by the promoted proteolysis and reduced intrahepatic blood volume.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
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name
Hyperammonaemia in V1a vasopre ...... ced intrahepatic blood volume.
@en
Hyperammonaemia in V1a vasopre ...... ced intrahepatic blood volume.
@nl
type
label
Hyperammonaemia in V1a vasopre ...... ced intrahepatic blood volume.
@en
Hyperammonaemia in V1a vasopre ...... ced intrahepatic blood volume.
@nl
prefLabel
Hyperammonaemia in V1a vasopre ...... ced intrahepatic blood volume.
@en
Hyperammonaemia in V1a vasopre ...... ced intrahepatic blood volume.
@nl
P2093
P2860
P921
P1476
Hyperammonaemia in V1a vasopre ...... ced intrahepatic blood volume.
@en
P2093
Akito Tanoue
Atsushi Sanbe
Fumio Endo
Masami Hiroyama
Sayuri Oshikawa
Toshinori Aoyagi
Yoko Fujiwara
P2860
P304
P356
10.1113/JPHYSIOL.2007.129569
P407
P577
2007-03-22T00:00:00Z