Hepatic loss of Lissencephaly 1 (Lis1) induces fatty liver and accelerates liver tumorigenesis in mice.

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Hepatic loss of Lissencephaly 1 (Lis1) induces fatty liver and accelerates liver tumorigenesis in mice.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

@zh-cn

2018年学术文章

@zh-hans

2018年学术文章

@zh-my

2018年学术文章

@zh-sg

2018年學術文章

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2018年學術文章

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name

Hepatic loss of Lissencephaly ...... s liver tumorigenesis in mice.

@en

Hepatic loss of Lissencephaly 1

@nl

type

Item

label

Hepatic loss of Lissencephaly ...... s liver tumorigenesis in mice.

@en

Hepatic loss of Lissencephaly 1

@nl

prefLabel

Hepatic loss of Lissencephaly ...... s liver tumorigenesis in mice.

@en

Hepatic loss of Lissencephaly 1

@nl

P1476

Hepatic loss of Lissencephaly ...... s liver tumorigenesis in mice.

@en

P2093

Ailing Wu

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Hai Song

http://www.w3.org/2001/XMLSchema#string

Jinqiu Lu

http://www.w3.org/2001/XMLSchema#string

Junling Jia

http://www.w3.org/2001/XMLSchema#string

Liansheng Liu

http://www.w3.org/2001/XMLSchema#string

Mujun Zhao

http://www.w3.org/2001/XMLSchema#string

Qingzhe Wu

http://www.w3.org/2001/XMLSchema#string

Ran Li

http://www.w3.org/2001/XMLSchema#string

Xiaoling Li

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P2860

Coupling of stress in the ER to activation of JNK protein kinases by transmembrane protein kinase IRE1

Identification and characterization of pancreatic eukaryotic initiation factor 2 alpha-subunit kinase, PEK, involved in translational control

Endoplasmic reticulum stress and the inflammatory basis of metabolic disease

Brefeldin A: insights into the control of membrane traffic and organelle structure

Nudel functions in membrane traffic mainly through association with Lis1 and cytoplasmic dynein

Lis1 acts as a "clutch" between the ATPase and microtubule-binding domains of the dynein motor.

Isolation of a Miller-Dieker lissencephaly gene containing G protein beta-subunit-like repeats

Deletion of the developmentally essential gene ATR in adult mice leads to age-related phenotypes and stem cell loss

Protein translation and folding are coupled by an endoplasmic-reticulum-resident kinase

Disregulated RhoGTPases and actin cytoskeleton contribute to the migration defect in Lis1-deficient neurons

P304

5160-5171

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P31

scholarly article

P356

10.1074/JBC.RA117.001474

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P407

English

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P478

293

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P577

2018-02-23T00:00:00Z

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P698

29475944

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P921

lissencephaly

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5892582

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