Monotypic low-level HIV viremias during ART are associated with disproportionate production of X4 virions and systemic immune activation.

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Monotypic low-level HIV viremias during ART are associated with disproportionate production of X4 virions and systemic immune activation.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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2018年學術文章

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Monotypic low-level HIV viremi ...... nd systemic immune activation.

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Monotypic low-level HIV viremi ...... nd systemic immune activation.

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Item

label

Monotypic low-level HIV viremi ...... nd systemic immune activation.

@en

Monotypic low-level HIV viremi ...... nd systemic immune activation.

@nl

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Monotypic low-level HIV viremi ...... and systemic immune activation

@en

prefLabel

Monotypic low-level HIV viremi ...... nd systemic immune activation.

@en

Monotypic low-level HIV viremi ...... nd systemic immune activation.

@nl

P1476

Monotypic low-level HIV viremi ...... and systemic immune activation

@en

P2093

Alberto La Rosa

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Caroline Mitchell

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Corey Williams-Wietzikoski

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Eduardo Ticona

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Edward P Acosta

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Frankline Onchiri

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Jennifer McKernan-Mullin

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Jillian Legard

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Joshua Stern

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Kelli Kraft

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P2860

Microbial translocation is a cause of systemic immune activation in chronic HIV infection

Residual human immunodeficiency virus type 1 viremia in some patients on antiretroviral therapy is dominated by a small number of invariant clones rarely found in circulating CD4+ T cells

ART suppresses plasma HIV-1 RNA to a stable set point predicted by pretherapy viremia

A modified Bonferroni method for discrete data.

Influence of membrane CD25 stability on T lymphocyte activity: implications for immunoregulation.

HIV-1 residual viremia correlates with persistent T-cell activation in poor immunological responders to combination antiretroviral therapy.

Characterization of HIV-1 RNA forms in the plasma of patients undergoing successful HAART.

Evidence that low-level viremias during effective highly active antiretroviral therapy result from two processes: expression of archival virus and replication of virus.

Protease inhibitor monotherapy is associated with a higher level of monocyte activation, bacterial translocation and inflammation.

Soluble markers of inflammation and coagulation but not T-cell activation predict non-AIDS-defining morbid events during suppressive antiretroviral treatment.

P304

1389-1401

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P31

scholarly article

P356

10.1097/QAD.0000000000001824

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P407

English

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P478

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P50

Jaime Soria

P577

2018-07-01T00:00:00Z

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P698

29683841

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P932

6039404

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