AGE/RAGE-Induced EMP Release via the NOX-Derived ROS Pathway.

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AGE/RAGE-Induced EMP Release via the NOX-Derived ROS Pathway.

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2018 nî lūn-bûn

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2018年の論文

@ja

2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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AGE/RAGE-Induced EMP Release via the NOX-Derived ROS Pathway.

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AGE/RAGE-Induced EMP Release via the NOX-Derived ROS Pathway.

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Item

label

AGE/RAGE-Induced EMP Release via the NOX-Derived ROS Pathway.

@en

AGE/RAGE-Induced EMP Release via the NOX-Derived ROS Pathway.

@nl

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AGE/RAGE-Induced EMP Release via the NOX-Derived ROS Pathway.

@en

AGE/RAGE-Induced EMP Release via the NOX-Derived ROS Pathway.

@nl

P1476

AGE/RAGE-Induced EMP Release via the NOX-Derived ROS Pathway

@en

P2093

Hong-Mei Li

http://www.w3.org/2001/XMLSchema#string

Ying-Hua Chen

http://www.w3.org/2001/XMLSchema#string

Zhang-Wei Chen

http://www.w3.org/2001/XMLSchema#string

P2860

Linking RAGE and Nox in diabetic micro- and macrovascular complications

Biochemistry, physiology, and pathophysiology of NADPH oxidases in the cardiovascular system

Role of advanced glycation end products in cellular signaling

Advanced glycation end products and diabetic complications

Benfotiamine prevents macro- and microvascular endothelial dysfunction and oxidative stress following a meal rich in advanced glycation end products in individuals with type 2 diabetes

Serum concentrations of advanced glycation endproducts are associated with the development of atherosclerosis as well as diabetic microangiopathy in patients with type 2 diabetes.

Association of circulating endothelial microparticles with cardiometabolic risk factors in the Framingham Heart Study.

Combined NOX1/4 inhibition with GKT137831 in mice provides dose-dependent reno- and atheroprotection even in established micro- and macrovascular disease.

Treatment-dependent and treatment-independent risk factors associated with the risk of diabetes-related events: a retrospective analysis based on 229,042 patients with type 2 diabetes mellitus.

Advanced glycation end products and oxidative stress in type 2 diabetes mellitus.

P304

6823058

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1155/2018/6823058

http://www.w3.org/2001/XMLSchema#string

P478

2018

http://www.w3.org/2001/XMLSchema#string

P50

Xin-Feng Yan

Bo Feng

P577

2018-03-18T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

29744367

http://www.w3.org/2001/XMLSchema#string

P932

5878883

http://www.w3.org/2001/XMLSchema#string