about
Activation of Chymotrypsin-Like Activity of the Proteasome during Ischemia Induces Myocardial Dysfunction and DeathTherapeutic targeting of autophagy in myocardial infarction and heart failure.The role of PI3Kα isoform in cardioprotection.Cardioprotection mediated by exosomes is impaired in the setting of type II diabetes but can be rescued by the use of non-diabetic exosomes in vitro.Role of PI3K in myocardial ischaemic preconditioning: mapping pro-survival cascades at the trigger phase and at reperfusion.Mechanical stretch increases L-type calcium channel stability in cardiomyocytes through a polycystin-1/AKT-dependent mechanism.Plasma exosomes protect the myocardium from ischemia-reperfusion injury.High-Fat-Diet-Induced Obesity Produces Spontaneous Ventricular Arrhythmias and Increases the Activity of Ryanodine Receptors in Mice.Novel Therapies Targeting Cardioprotection and Regeneration.Dexmedetomidine protects the heart against ischemia-reperfusion injury by an endothelial eNOS/NO dependent mechanism.Autophagy mediates tumor necrosis factor-α-induced phenotype switching in vascular smooth muscle A7r5 cell line.Drp1 loss-of-function reduces cardiomyocyte oxygen dependence protecting the heart from ischemia-reperfusion injury
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description
researcher ORCID ID = 0000-0003-0517-5697
@en
wetenschapper
@nl
name
Jaime A Riquelme
@ast
Jaime A Riquelme
@en
Jaime A Riquelme
@es
Jaime A Riquelme
@nl
type
label
Jaime A Riquelme
@ast
Jaime A Riquelme
@en
Jaime A Riquelme
@es
Jaime A Riquelme
@nl
prefLabel
Jaime A Riquelme
@ast
Jaime A Riquelme
@en
Jaime A Riquelme
@es
Jaime A Riquelme
@nl
P106
P1153
56015203500
P31
P496
0000-0003-0517-5697