about
Effect of transient and permanent permeability transition pore opening on NAD(P)H localization in intact cells.Hormonal regulation of mitochondrial energy production.Implication of liver cardiolipins in mitochondrial energy metabolism disorder in cancer cachexia.Reduced cardiolipin content decreases respiratory chain capacities and increases ATP synthesis yield in the human HepaRG cells.Regulation of hepatic cardiolipin metabolism by TNFα: Implication in cancer cachexia.Metabolic reprogramming in cancer cells, consequences on pH and tumour progression: Integrated therapeutic perspectives with dietary lipids as adjuvant to anticancer treatment.Efficiency of oxidative phosphorylation in liver mitochondria is decreased in a rat model of peritoneal carcinosis.Cancer cachexia is associated with a decrease in skeletal muscle mitochondrial oxidative capacities without alteration of ATP production efficiency.Mitochondrial energy metabolism in a model of undernutrition induced by dexamethasone.Dexamethasone treatment specifically increases the basal proton conductance of rat liver mitochondria.Cardiolipin content is involved in liver mitochondrial energy wasting associated with cancer-induced cachexia without the involvement of adenine nucleotide translocase.Processing of vegetable-borne carotenoids in the human stomach and duodenum.Dexamethasone impairs muscle energetics, studied by (31)P NMR, in rats.Thyroxine therapy in euthyroid patients does not affect body composition or muscular function.Pharmacological inhibition of carnitine palmitoyltransferase 1 restores mitochondrial oxidative phosphorylation in human trifunctional protein deficient fibroblasts.ATP-dependent activity and mitochondrial localization of drug efflux pumps in doxorubicin-resistant breast cancer cells.Oral supplements differing in fat and carbohydrate content: effect on the appetite and food intake of undernourished elderly patientsn-3 PUFA-enriched diet delays the occurrence of cancer cachexia in rat with peritoneal carcinosis
P50
Q33426739-D3417DD1-9A6B-4276-8A81-B3CAD8F592E6Q36148089-4DDAF756-8E1A-4CE3-9B0D-23C4F9E19011Q38028227-B6916F9A-BC65-4397-987B-4C2ADE10F0D5Q38410643-3B8E80DA-465D-45C3-A4BE-31CCF6782FE6Q38412318-4DF437C9-96F3-40CE-A576-63B86D1ED1F1Q39189962-16F3D1F0-8140-416C-90EB-51D0A40B526BQ39629174-D2F09527-C24B-4504-9081-C20977DAA156Q42350705-8BE8B508-C9E9-44D5-BDD7-D17A2CA05D43Q42411402-5DEE94D6-C77F-4FE4-A1FD-0900A010BB0DQ42440098-99C4BC04-2C91-420E-8B92-A9C043969534Q44276118-74D504A7-0CD7-4749-9144-6E665CEEB5B7Q44433187-6566C01A-B62F-4678-8051-F3B2F3CB47F5Q45220595-32AC1496-7515-40BC-9F28-4E1BC16009F0Q46910924-EA4D0B6A-90EB-4CB8-AF89-925853071DEBQ51065662-43471CE7-DAF3-41E0-85F7-54A3754FBEABQ51132270-0CC348CC-654F-45DE-AC31-90983AF9958AQ80422082-22C622E2-8CF2-478B-A76B-025D1BDC50DDQ83970205-6BF36A68-71BC-4739-AAE8-BBBC58DE99A7
P50
description
researcher ORCID ID = 0000-0002-2293-6606
@en
wetenschapper
@nl
name
Jean-François Dumas
@ast
Jean-François Dumas
@en
Jean-François Dumas
@es
Jean-François Dumas
@nl
type
label
Jean-François Dumas
@ast
Jean-François Dumas
@en
Jean-François Dumas
@es
Jean-François Dumas
@nl
prefLabel
Jean-François Dumas
@ast
Jean-François Dumas
@en
Jean-François Dumas
@es
Jean-François Dumas
@nl
P31
P496
0000-0002-2293-6606