about
The AP-1 transcription factor Batf controls T(H)17 differentiationTransforming growth factor-beta induces development of the T(H)17 lineageReciprocal interactions of the intestinal microbiota and immune system.The Th17 family: flexibility follows function.Notch simultaneously orchestrates multiple helper T cell programs independently of cytokine signals.Developmental plasticity of Th17 and Treg cells.Interplay between the TH17 and TReg cell lineages: a (co-)evolutionary perspective.Regulation of the Ifng locus in the context of T-lineage specification and plasticity.IL-1 signaling modulates activation of STAT transcription factors to antagonize retinoic acid signaling and control the TH17 cell-iTreg cell balanceTh22 cells are an important source of IL-22 for host protection against enteropathogenic bacteriaGeneration of antigen-specific, Foxp3-expressing CD4+ regulatory T cells by inhibition of APC proteosome function.A distal conserved sequence element controls Ifng gene expression by T cells and NK cells.Differential IL-2 expression defines developmental fates of follicular versus nonfollicular helper T cells.Immunology. T-bet or not T-betIL-1R signaling promotes STAT3 and NF-κB factor recruitment to distal cis-regulatory elements that regulate Il17a/f transcription
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description
investigador
@es
researcher
@en
wetenschapper
@nl
name
Robin D Hatton
@en
Robin D Hatton
@nl
type
label
Robin D Hatton
@en
Robin D Hatton
@nl
prefLabel
Robin D Hatton
@en
Robin D Hatton
@nl
P31
P496
0000-0002-4797-9899