Prion-like Mechanism in Amyotrophic Lateral Sclerosis: are Protein Aggregates the Key?
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Confocal Spectroscopy to Study Dimerization, Oligomerization and Aggregation of Proteins: A Practical GuideParadoxical Roles of Antioxidant Enzymes: Basic Mechanisms and Health ImplicationsDifferential effects of phytotherapic preparations in the hSOD1 Drosophila melanogaster model of ALS.The Role of Proteases in Hippocampal Synaptic Plasticity: Putting Together Small Pieces of a Complex Puzzle.Spread of pathology in amyotrophic lateral sclerosis: assessment of phosphorylated TDP-43 along axonal pathways.Disease-related gene module detection based on a multi-label propagation clustering algorithm.Nucleocytoplasmic transport in C9orf72-mediated ALS/FTD.Pathological TDP-43 changes in Betz cells differ from those in bulbar and spinal α-motoneurons in sporadic amyotrophic lateral sclerosis.Old versus New Mechanisms in the Pathogenesis of ALS.Non-neuronal Cells in ALS: Role of Glial, Immune cells and Blood-CNS Barriers.Physiological functions and pathobiology of TDP-43 and FUS/TLS proteins.Oligomeropathies and pathogenesis of Alzheimer and Parkinson's diseases.Neuron-to-Neuron Transfer of FUS in Drosophila Primary Neuronal Culture Is Enhanced by ALS-Associated Mutations.Pathogenic mechanisms of prion protein, amyloid-β and α-synuclein misfolding: the prion concept and neurotoxicity of protein oligomers.Involvement of Cellular Prion Protein in α-Synuclein Transport in Neurons.Identify Huntington's disease associated genes based on restricted Boltzmann machine with RNA-seq data.Let's make microglia great again in neurodegenerative disorders.Amyotrophic lateral sclerosis as a protein level, non-genomic disease: Therapy with S2RM exosome released molecules.Amyotrophic lateral sclerosis as a protein level, non-genomic disease: Therapy with S2RM exosome released molecules.Anterior Cingulate Cortex TDP-43 Pathology in Sporadic Amyotrophic Lateral Sclerosis.A computational combinatorial approach identifies a protein inhibitor of superoxide dismutase 1 misfolding, aggregation, and cytotoxicity.A Pathogenic Mechanism Potentially Operative in Multiple Progressive Diseases and Its Therapeutic Implications.Amyotrophic lateral sclerosisMinimotifs dysfunction is pervasive in neurodegenerative disordersAggregated SOD1 causes selective death of cultured human motor neuronsAmyloid assembly and disassembly
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P2860
Prion-like Mechanism in Amyotrophic Lateral Sclerosis: are Protein Aggregates the Key?
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2015 nî lūn-bûn
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2015 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2015 թվականի մարտին հրատարակված գիտական հոդված
@hy
2015年の論文
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2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
name
Prion-like Mechanism in Amyotrophic Lateral Sclerosis: are Protein Aggregates the Key?
@ast
Prion-like Mechanism in Amyotrophic Lateral Sclerosis: are Protein Aggregates the Key?
@en
Prion-like Mechanism in Amyotrophic Lateral Sclerosis: are Protein Aggregates the Key?
@nl
type
label
Prion-like Mechanism in Amyotrophic Lateral Sclerosis: are Protein Aggregates the Key?
@ast
Prion-like Mechanism in Amyotrophic Lateral Sclerosis: are Protein Aggregates the Key?
@en
Prion-like Mechanism in Amyotrophic Lateral Sclerosis: are Protein Aggregates the Key?
@nl
prefLabel
Prion-like Mechanism in Amyotrophic Lateral Sclerosis: are Protein Aggregates the Key?
@ast
Prion-like Mechanism in Amyotrophic Lateral Sclerosis: are Protein Aggregates the Key?
@en
Prion-like Mechanism in Amyotrophic Lateral Sclerosis: are Protein Aggregates the Key?
@nl
P2860
P3181
P356
P1476
Prion-like Mechanism in Amyotrophic Lateral Sclerosis: are Protein Aggregates the Key?
@en
P2093
Hyung-Jun Kim
Shynrye Lee
P2860
P3181
P356
10.5607/EN.2015.24.1.1
P407
P577
2015-03-01T00:00:00Z