Natural killer cells trigger osteoclastogenesis and bone destruction in arthritis. - Test2 - elhamkhani - TriplyDB

Natural killer cells trigger osteoclastogenesis and bone destruction in arthritis.

Natural killer cells trigger osteoclastogenesis and bone destruction in arthritis.

http://www.wikidata.org/entity/Q34059280

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Bindarit, an inhibitor of monocyte chemotactic protein synthesis, protects against bone loss induced by chikungunya virus infection Mesenchymal Stem Cells Regulate the Innate and Adaptive Immune Responses Dampening Arthritis Progression An alternate STAT6-independent pathway promotes eosinophil influx into blood during allergic airway inflammation.NK cells are not required for spontaneous autoimmune diabetes in NOD mice.SPA0355, a thiourea analogue, inhibits inflammatory responses and joint destruction in fibroblast-like synoviocytes and mice with collagen-induced arthritis.NK cells: immune cross-talk and therapeutic implications.Positive regulators of osteoclastogenesis and bone resorption in rheumatoid arthritis Activation of resorption in fatigue-loaded bone involves both apoptosis and active pro-osteoclastogenic signaling by distinct osteocyte populations Bisphosphonate-induced differential modulation of immune cell function in gingiva and bone marrow in vivo: role in osteoclast-mediated NK cell activation.The phenotype of circulating follicular-helper T cells in patients with rheumatoid arthritis defines CD200 as a potential therapeutic target.Osteoporotic bone of miR-150-deficient mice: Possibly due to low serum OPG-mediated osteoclast activation.Organ-specific features of natural killer cells NAP-2 Secreted by Human NK Cells Can Stimulate Mesenchymal Stem/Stromal Cell Recruitment.The Interplay between the bone and the immune system Advances in rheumatoid arthritis animal models.Coupling the activities of bone formation and resorption: a multitude of signals within the basic multicellular unit Interleukin-22 Secreted by NKp44+ Natural Killer Cells Promotes Proliferation of Fibroblast-Like Synoviocytes in Rheumatoid Arthritis.Natural killer cells in human autoimmune disorders Differential chemotactic receptor requirements for NK cell subset trafficking into bone marrow.Cells of the immune system orchestrate changes in bone cell function.Immune cells and bone: coupling goes both ways.Bone and the innate immune system.Mechanisms of tissue damage in arthritis.Circulating monocytes: an appropriate model for bone-related study.IL15RA is required for osteoblast function and bone mineralization.Human CD56bright NK Cells: An Update.Arthritis models: usefulness and interpretation.Balance between activating NKG2D, DNAM-1, NKp44 and NKp46 and inhibitory CD94/NKG2A receptors determine natural killer degranulation towards rheumatoid arthritis synovial fibroblasts.Enhanced mesenchymal stromal cell recruitment via natural killer cells by incorporation of inflammatory signals in biomaterials.Interleukin-15-activated natural killer cells kill autologous osteoclasts via LFA-1, DNAM-1 and TRAIL, and inhibit osteoclast-mediated bone erosion in vitro.Bi(o)communications among peripheral blood fractions: a focus on NK and NKT cell biology in rheumatoid arthritis.Role of cytolytic impairment of natural killer and natural killer T-cell populations in rheumatoid arthritis.---Inhibition of histone H3K27 demethylase selectively modulates inflammatory phenotypes of natural killer cells.CXCR3/CXCL10 Axis Regulates Neutrophil-NK Cell Cross-Talk Determining the Severity of Experimental Osteoarthritis.Granzyme A Contributes to Inflammatory Arthritis in Mice Through Stimulation of Osteoclastogenesis.Antibiotics with Interleukin-15 inhibition reduces joint inflammation and bone erosions but not cartilage destruction in Staphylococcus aureus-induced arthritis.Molecular signatures and transcriptional regulatory networks of human immature decidual NK and mature peripheral NK cells.The Role of the Immune Cells in Fracture Healing.Absence of Fms-like tyrosine kinase 3 ligand (Flt3L) signalling protects against collagen-induced arthritis.Local gene transfer of OPG prevents joint damage and disease progression in collagen-induced arthritis.

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P2860

Natural killer cells trigger osteoclastogenesis and bone destruction in arthritis.

http://www.wikidata.org/entity/Q34059280

description

2010 nî lūn-bûn

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2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հուլիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

@wuu

name

Natural killer cells trigger osteoclastogenesis and bone destruction in arthritis.

@ast

Natural killer cells trigger osteoclastogenesis and bone destruction in arthritis.

@en

Natural killer cells trigger osteoclastogenesis and bone destruction in arthritis.

@nl

type

label

Natural killer cells trigger osteoclastogenesis and bone destruction in arthritis.

@ast

Natural killer cells trigger osteoclastogenesis and bone destruction in arthritis.

@en

Natural killer cells trigger osteoclastogenesis and bone destruction in arthritis.

@nl

prefLabel

Natural killer cells trigger osteoclastogenesis and bone destruction in arthritis.

@ast

Natural killer cells trigger osteoclastogenesis and bone destruction in arthritis.

@en

Natural killer cells trigger osteoclastogenesis and bone destruction in arthritis.

@nl

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Proceedings of the National Academy of Sciences of the United States of America

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Natural killer cells trigger osteoclastogenesis and bone destruction in arthritis.

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P2093

Cristina Teixeira de Matos

http://www.w3.org/2001/XMLSchema#string

Edgar G Engleman

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Emily Stein

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Ingo H Tarner

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Kalle Söderström

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Paula Colmenero

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Ulrich Purath

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William H Robinson

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P2860

Osteoprotegerin ligand is a cytokine that regulates osteoclast differentiation and activation

Osteoprotegerin: a novel secreted protein involved in the regulation of bone density

IFN-gamma-producing human T cells directly induce osteoclastogenesis from human monocytes via the expression of RANKL

RANK is essential for osteoclast and lymph node development

osteoprotegerin-deficient mice develop early onset osteoporosis and arterial calcification

Early rheumatoid arthritis is characterized by a distinct and transient synovial fluid cytokine profile of T cell and stromal cell origin.

Bone resorption by osteoclasts

Unique subpopulations of CD56+ NK and NK-T peripheral blood lymphocytes identified by chemokine receptor expression repertoire

OPGL is a key regulator of osteoclastogenesis, lymphocyte development and lymph-node organogenesis

NK cell recognition

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13028-13033

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10.1073/PNAS.1000546107

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29

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107

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Ulf Müller-Ladner

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2010-07-06T00:00:00Z

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45113506

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20615964

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2919936

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