Myocardin is sufficient for a smooth muscle-like contractile phenotype
about
Leiomodin 1, a new serum response factor-dependent target gene expressed preferentially in differentiated smooth muscle cellsmiR-145 and miR-143 regulate smooth muscle cell fate and plasticityMyocardin-dependent activation of the CArG box-rich smooth muscle gamma-actin gene: preferential utilization of a single CArG element through functional association with the NKX3.1 homeodomain proteinmiR-1/133a clusters cooperatively specify the cardiomyogenic lineage by adjustment of myocardin levels during embryonic heart developmentThe short and long of noncoding sequences in the control of vascular cell phenotypesInduction of microRNA-1 by myocardin in smooth muscle cells inhibits cell proliferationMyocardin is required for cardiomyocyte survival and maintenance of heart functionMyocardin overexpression is sufficient for promoting the development of a mature smooth muscle cell-like phenotype from human embryonic stem cellsThe smooth muscle cell-restricted KCNMB1 ion channel subunit is a direct transcriptional target of serum response factor and myocardin.Biomechanical Regulation of Endothelium-dependent Events Critical for Adaptive Remodeling.Transdifferentiation of human endothelial progenitors into smooth muscle cellsVEGF-A Stimulates STAT3 Activity via Nitrosylation of Myocardin to Regulate the Expression of Vascular Smooth Muscle Cell Differentiation Markers.Vascular smooth muscle phenotypic diversity and functionDirect evidence for functional smooth muscle myosin II in the 10S self-inhibited monomeric conformation in airway smooth muscle cellsSmad3-mediated myocardin silencing: a novel mechanism governing the initiation of smooth muscle differentiation.down-regulation of Kruppel-like factor-4 (KLF4) by microRNA-143/145 is critical for modulation of vascular smooth muscle cell phenotype by transforming growth factor-beta and bone morphogenetic protein 4Smooth muscle calponin: an unconventional CArG-dependent gene that antagonizes neointimal formationTransforming growth factor-beta1 (TGF-beta1) utilizes distinct pathways for the transcriptional activation of microRNA 143/145 in human coronary artery smooth muscle cells.Transforming growth factor-β and smooth muscle differentiationSTAT3 Protein Regulates Vascular Smooth Muscle Cell Phenotypic Switch by Interaction with Myocardin.Epigenetic regulation of smooth muscle cell plasticity.Loss of the mechanotransducer zyxin promotes a synthetic phenotype of vascular smooth muscle cells.Expression and promoter analysis of a highly restricted integrin alpha gene in vascular smooth muscle.Development and characterization of a 3D multicell microtissue culture model of airway smooth muscle.Autoregulatory Control of Smooth Muscle Myosin Light Chain Kinase Promoter by Notch Signaling.Ca2+/calmodulin-dependent protein kinase II-γ (CaMKIIγ) negatively regulates vascular smooth muscle cell proliferation and vascular remodelingA novel in vitro model system for smooth muscle differentiation from human embryonic stem cell-derived mesenchymal cellsNoncanonical matrix metalloprotease-1-protease-activated receptor-1 signaling triggers vascular smooth muscle cell dedifferentiation and arterial stenosis.Cell division cycle 7 mediates transforming growth factor-β-induced smooth muscle maturation through activation of myocardin gene transcription.Set7 mediated interactions regulate transcriptional networks in embryonic stem cells.Circulating miRNAs: reflecting or affecting cardiovascular disease?Vascular smooth muscle cell differentiation-2010.Extracellular matrix synthesis in vascular disease: hypertension, and atherosclerosis.Myocardin in biology and disease.Regulation of cardiac myocyte cell death and differentiation by myocardin.Envisioning metastasis as a transdifferentiation phenomenon clarifies discordant results on cancer.Noncoding RNAs in smooth muscle cell homeostasis: implications in phenotypic switch and vascular disorders.Mechanisms of Smooth Muscle Cell Differentiation Are Distinctly Altered in Thoracic Aortic Aneurysms Associated with Bicuspid or Tricuspid Aortic ValvesSMAD Signaling Is Required for Structural Integrity of the Female Reproductive Tract and Uterine Function During Early Pregnancy in Mice.IQGAP1 promotes the phenotypic switch of vascular smooth muscle by myocardin pathway: a potential target for varicose vein.
P2860
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P2860
Myocardin is sufficient for a smooth muscle-like contractile phenotype
description
2008 nî lūn-bûn
@nan
2008 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Myocardin is sufficient for a smooth muscle-like contractile phenotype
@ast
Myocardin is sufficient for a smooth muscle-like contractile phenotype
@en
Myocardin is sufficient for a smooth muscle-like contractile phenotype
@en-gb
Myocardin is sufficient for a smooth muscle-like contractile phenotype
@nl
type
label
Myocardin is sufficient for a smooth muscle-like contractile phenotype
@ast
Myocardin is sufficient for a smooth muscle-like contractile phenotype
@en
Myocardin is sufficient for a smooth muscle-like contractile phenotype
@en-gb
Myocardin is sufficient for a smooth muscle-like contractile phenotype
@nl
prefLabel
Myocardin is sufficient for a smooth muscle-like contractile phenotype
@ast
Myocardin is sufficient for a smooth muscle-like contractile phenotype
@en
Myocardin is sufficient for a smooth muscle-like contractile phenotype
@en-gb
Myocardin is sufficient for a smooth muscle-like contractile phenotype
@nl
P2093
P2860
P3181
P1476
Myocardin is sufficient for a smooth muscle-like contractile phenotype
@en
P2093
Berislav V Zlokovic
Joseph M Miano
Robert D Bell
Xiaochun Long
P2860
P304
P3181
P356
10.1161/ATVBAHA.108.166066
P407
P577
2008-08-01T00:00:00Z