Ischemic preconditioning preserves connexin 43 phosphorylation during sustained ischemia in pig hearts in vivo. - Wikidata - thesis-toulmin - TriplyDB

Ischemic preconditioning preserves connexin 43 phosphorylation during sustained ischemia in pig hearts in vivo.

Ischemic preconditioning preserves connexin 43 phosphorylation during sustained ischemia in pig hearts in vivo.

http://www.wikidata.org/entity/Q31142769

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p38 MAPK in cardioprotection - are we there yet?Activator of G protein signaling 8 (AGS8) is required for hypoxia-induced apoptosis of cardiomyocytes: role of G betagamma and connexin 43 (CX43)Selective blockade of protein kinase B protects the rat and human myocardium against ischaemic injury No impact of protein phosphatases on connexin 43 phosphorylation in ischemic preconditioning.Gap junctions regulate extracellular signal-regulated kinase signaling to affect gene transcription.S-diclofenac protects against doxorubicin-induced cardiomyopathy in mice via ameliorating cardiac gap junction remodeling The p38 mitogen-activated protein kinase pathway--a potential target for intervention in infarction, hypertrophy, and heart failure.Remote ischemic conditioning.The role of mitochondria in protection of the heart by preconditioning.Remote ischemic preconditioning preserves Connexin 43 phosphorylation in the rat heart in vivo Analysis of Connexin43 phosphorylated at S325, S328 and S330 in normoxic and ischemic heart.Leptin, the obesity-associated hormone, exhibits direct cardioprotective effects Temporal regulation of connexin phosphorylation in embryonic and adult tissues.Connexin43 phosphorylation in brain, cardiac, endothelial and epithelial tissues.Role of Akt and Ca2+ on cell permeabilization via connexin43 hemichannels induced by metabolic inhibition.Role of Mitogen-Activated Protein Kinases in Myocardial Ischemia-Reperfusion Injury during Heart Transplantation Biological role of connexin intercellular channels and hemichannels Subcellular preconditioning of stem cells: mito-Cx43 gene targeting is cytoprotective via shift of mitochondrial Bak and Bcl-xL balance.Update on the Pathophysiological Role of Intracellular Signaling Pathways in Atherosclerotic Plaques and Ischemic Myocardium Connexin 43 is an emerging therapeutic target in ischemia/reperfusion injury, cardioprotection and neuroprotection Phosphorylation at S365 is a gatekeeper event that changes the structure of Cx43 and prevents down-regulation by PKC.Quantitative proteomic analysis reveals novel mitochondrial targets of estrogen deficiency in the aged female rat heart Connexin 43 signalling and cardioprotection.Cardiomyocyte-restricted overexpression of extracellular superoxide dismutase increases nitric oxide bioavailability and reduces infarct size after ischemia/reperfusion.Hypoxia-regulated activity of PKCepsilon in the lens.Connexin mimetic peptides inhibit Cx43 hemichannel opening triggered by voltage and intracellular Ca2+ elevation Regulation of gap junctions by nitric oxide influences the generation of arrhythmias resulting from acute ischemia and reperfusion in vivo.Key connexin 43 phosphorylation events regulate the gap junction life cycle Creation and implications of a phenome-genome network Connexin43 phosphorylation: structural changes and biological effects.Injury-triggered Akt phosphorylation of Cx43: a ZO-1-driven molecular switch that regulates gap junction size.Stimulation of cardiac beta-adrenoceptors targets connexin 43.Specific Cx43 phosphorylation events regulate gap junction turnover in vivo Spatio-temporal regulation of connexin43 phosphorylation and gap junction dynamics.Phosphorylation at Connexin43 Serine-368 Is Necessary for Myocardial Conduction During Metabolic Stress.Nitric oxide, PKC-ε, and connexin43 are crucial for ischemic preconditioning-induced chemical gap junction uncoupling.Protein kinase Cepsilon mediates salutary effects on electrical coupling induced by ischemic preconditioning.Protection afforded by ischemic preconditioning is not mediated by effects on cell-to-cell electrical coupling during myocardial ischemia-reperfusion.Protective role of gap junctions in preconditioning against myocardial infarction.High glucose down-regulates intercellular communication in retinal endothelial cells by enhancing degradation of connexin 43 by a proteasome-dependent mechanism.

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P2860

Ischemic preconditioning preserves connexin 43 phosphorylation during sustained ischemia in pig hearts in vivo.

http://www.wikidata.org/entity/Q31142769

description

2003 nî lūn-bûn

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2003 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2003 թվականի մայիսին հրատարակված գիտական հոդված

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年论文

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Ischemic preconditioning prese ...... schemia in pig hearts in vivo.

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Ischemic preconditioning prese ...... schemia in pig hearts in vivo.

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Ischemic preconditioning prese ...... schemia in pig hearts in vivo.

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Ischemic preconditioning prese ...... schemia in pig hearts in vivo.

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Alexej Duschin

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Andreas Skyschally

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Gerd Heusch

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Ina Konietzka

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Petra Gres

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Rainer Schulz

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Sergej Belosjorow

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1355-1357

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scholarly article

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10.1096/FJ.02-0975FJE

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10

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17

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2003-05-20T00:00:00Z

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12759340

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phosphorylation