Deafness and renal tubular acidosis in mice lacking the K-Cl co-transporter Kcc4. - Wikidata - thesis-toulmin - TriplyDB

Deafness and renal tubular acidosis in mice lacking the K-Cl co-transporter Kcc4.

Deafness and renal tubular acidosis in mice lacking the K-Cl co-transporter Kcc4.

http://www.wikidata.org/entity/Q34125574

about

NH2-terminal heterogeneity in the KCC3 K+-Cl- cotransporter The thiazide-sensitive Na+-Cl- cotransporter: molecular biology, functional properties, and regulation by WNKs WNK kinases, renal ion transport and hypertension Molecular mechanisms and regulation of urinary acidification K-Cl cotransporters, cell volume homeostasis, and neurological disease The role of connexins in ear and skin physiology - functional insights from disease-associated mutations Physiology of SLC12 transporters: lessons from inherited human genetic mutations and genetically engineered mouse knockouts Deletion of Tricellulin Causes Progressive Hearing Loss Associated with Degeneration of Cochlear Hair Cells.Fibro-vascular coupling in the control of cochlear blood flow Molecular and evolutionary insights into the structural organization of cation chloride cotransporters Gap junctions and cochlear homeostasis Lead roles for supporting actors: critical functions of inner ear supporting cells The forkhead transcription factor Foxi1 directly activates the AE4 promoter Mice with altered KCNQ4 K+ channels implicate sensory outer hair cells in human progressive deafness.Immunohistochemical localization of OCT2 in the cochlea of various species.KCNK5 channels mostly expressed in cochlear outer sulcus cells are indispensable for hearing Characterizing human vestibular sensory epithelia for experimental studies: new hair bundles on old tissue and implications for therapeutic interventions in ageing.Spontaneous regeneration of cochlear supporting cells after neonatal ablation ensures hearing in the adult mouse.Potassium ion movement in the inner ear: insights from genetic disease and mouse models Genetics of auditory mechano-electrical transduction.Inner ear supporting cells: rethinking the silent majority.Changes in the adult vertebrate auditory sensory epithelium after trauma.Potassium currents in isolated deiters' cells of Guinea pig.Selective ablation of pillar and deiters' cells severely affects cochlear postnatal development and hearing in mice.A mouse model for distal renal tubular acidosis reveals a previously unrecognized role of the V-ATPase a4 subunit in the proximal tubule.Defining the cellular environment in the organ of Corti following extensive hair cell loss: a basis for future sensory cell replacement in the Cochlea.Reduced electromotility of outer hair cells associated with connexin-related forms of deafness: an in silico study of a cochlear network mechanism.The Membrane Properties of Cochlear Root Cells are Consistent with Roles in Potassium Recirculation and Spatial Buffering.Supporting cell characteristics in long-deafened aged mouse ears Three-dimensional current flow in a large-scale model of the cochlea and the mechanism of amplification of sound.Endocochlear potential depends on Cl- channels: mechanism underlying deafness in Bartter syndrome IV.Recent findings and emerging questions in cochlear noise injury.Rapid hair cell loss: a mouse model for cochlear lesions.ATP-mediated cell-cell signaling in the organ of Corti: the role of connexin channels.Orphan glutamate receptor delta1 subunit required for high-frequency hearing.The role of potassium chloride cotransporters in murine and human sperm volume regulation.A claudin-9-based ion permeability barrier is essential for hearing.Motor protein-dependent membrane trafficking of KCl cotransporter-4 is important for cancer cell invasion Chemical crosslinking studies with the mouse Kcc1 K-Cl cotransporter Cell-type specific distribution of chloride transporters in the rat suprachiasmatic nucleus.

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Deafness and renal tubular acidosis in mice lacking the K-Cl co-transporter Kcc4.

http://www.wikidata.org/entity/Q34125574

description

2002 nî lūn-bûn

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2002 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2002 թվականի ապրիլին հրատարակված գիտական հոդված

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2002年の論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年论文

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name

Deafness and renal tubular acidosis in mice lacking the K-Cl co-transporter Kcc4.

@ast

Deafness and renal tubular acidosis in mice lacking the K-Cl co-transporter Kcc4.

@en

Deafness and renal tubular acidosis in mice lacking the K-Cl co-transporter Kcc4.

@nl

type

label

Deafness and renal tubular acidosis in mice lacking the K-Cl co-transporter Kcc4.

@ast

Deafness and renal tubular acidosis in mice lacking the K-Cl co-transporter Kcc4.

@en

Deafness and renal tubular acidosis in mice lacking the K-Cl co-transporter Kcc4.

@nl

prefLabel

Deafness and renal tubular acidosis in mice lacking the K-Cl co-transporter Kcc4.

@ast

Deafness and renal tubular acidosis in mice lacking the K-Cl co-transporter Kcc4.

@en

Deafness and renal tubular acidosis in mice lacking the K-Cl co-transporter Kcc4.

@nl

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Deafness and renal tubular acidosis in mice lacking the K-Cl co-transporter Kcc4.

@en

P2093

Christian A Hübner

http://www.w3.org/2001/XMLSchema#string

Franz X Beck

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Hannes Maier

http://www.w3.org/2001/XMLSchema#string

Marco B Rust

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Thomas Boettger

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P2860

Mutations in the gene encoding B1 subunit of H+-ATPase cause renal tubular acidosis with sensorineural deafness

KCNQ4, a novel potassium channel expressed in sensory outer hair cells, is mutated in dominant deafness

Cloning and characterization of KCC3 and KCC4, new members of the cation-chloride cotransporter gene family

Familial distal renal tubular acidosis is associated with mutations in the red cell anion exchanger (Band 3, AE1) gene

Connexin 26 mutations in hereditary non-syndromic sensorineural deafness

Deafness and imbalance associated with inactivation of the secretory Na-K-2Cl co-transporter

Gap junction systems in the mammalian cochlea

Intracellular anions as the voltage sensor of prestin, the outer hair cell motor protein

Barttin is a Cl- channel beta-subunit crucial for renal Cl- reabsorption and inner ear K+ secretion

Gap junctions in the rat cochlea: immunohistochemical and ultrastructural analysis

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10.1038/416874A

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