Mammary-specific inactivation of E-cadherin and p53 impairs functional gland development and leads to pleomorphic invasive lobular carcinoma in mice
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A periodic table for cancerAdhesion in mammary development: novel roles for E-cadherin in individual and collective cell migrationNuclear Kaiso expression is associated with high grade and triple-negative invasive breast cancerp120-catenin prevents multinucleation through control of MKLP1-dependent RhoA activity during cytokinesis.Loss of CDH1 and Pten accelerates cellular invasiveness and angiogenesis in the mouse uterus.Lobular breast cancer: molecular basis, mouse and cellular models.Toca-1 is suppressed by p53 to limit breast cancer cell invasion and tumor metastasis.Cytosolic p120-catenin regulates growth of metastatic lobular carcinoma through Rock1-mediated anoikis resistance.Mammary gland tumor promotion by chronic administration of IGF1 and the insulin analogue AspB10 in the p53R270H/⁺WAPCre mouse modelAEG-1 promotes anoikis resistance and orientation chemotaxis in hepatocellular carcinoma cells.Choosing a mouse model: experimental biology in context--the utility and limitations of mouse models of breast cancer.Nuclear p120-catenin regulates the anoikis resistance of mouse lobular breast cancer cells through Kaiso-dependent Wnt11 expressionTranscriptional Dynamics of Immortalized Human Mesenchymal Stem Cells during Transformation.Loss of Cdh1 and Trp53 in the uterus induces chronic inflammation with modification of tumor microenvironmentMammary collective cell migration involves transient loss of epithelial features and individual cell migration within the epithelium.Loss of plakoglobin promotes decreased cell-cell contact, increased invasion, and breast cancer cell dissemination in vivoCx26 knockout predisposes the mammary gland to primary mammary tumors in a DMBA-induced mouse model of breast cancerProteomics of genetically engineered mouse mammary tumors identifies fatty acid metabolism members as potential predictive markers for cisplatin resistance.Defined lipid analogues induce transient channels to facilitate drug-membrane traversal and circumvent cancer therapy resistanceModeling invasive lobular breast carcinoma by CRISPR/Cas9-mediated somatic genome editing of the mammary gland.PTEN Loss in E-Cadherin-Deficient Mouse Mammary Epithelial Cells Rescues Apoptosis and Results in Development of Classical Invasive Lobular Carcinoma.p120-Catenin Is Critical for the Development of Invasive Lobular Carcinoma in MiceLobular breast cancer: pathology, biology, and options for clinical intervention.Beyond E-cadherin: roles of other cadherin superfamily members in cancer.E-cadherin's role in development, tissue homeostasis and disease: Insights from mouse models: Tissue-specific inactivation of the adhesion protein E-cadherin in mice reveals its functions in health and disease.The relevance of EMT in breast cancer metastasis: Correlation or causality?Overview of Genetically Engineered Mouse Models of Breast Cancer Used in Translational Biology and Drug Development.Genetic Dissection of Cancer Development, Therapy Response, and Resistance in Mouse Models of Breast Cancer.IPH-926 lobular breast cancer cells harbor a p53 mutant with temperature-sensitive functional activity and allow for profiling of p53-responsive genes.Acquisition of anoikis resistance through CD147 upregulation: A new mechanism underlying metastasis of hepatocellular carcinoma cells.Restraining FOXO3-dependent transcriptional BMF activation underpins tumour growth and metastasis of E-cadherin-negative breast cancerIntraductal cisplatin treatment in a BRCA-associated breast cancer mouse model attenuates tumor development but leads to systemic tumors in aged female miceBreak Breast Cancer Addiction by CRISPR/Cas9 Genome Editing.Lobular carcinoma in situ and invasive lobular breast cancer are characterized by enhanced expression of transcription factor AP-2β.Correlation between E-cadherin and p120 expression in invasive ductal breast cancer with a lobular component and MRI findings.Using the GEMM-ESC strategy to study gene function in mouse models.Insertional mutagenesis identifies drivers of a novel oncogenic pathway in invasive lobular breast carcinoma.Pulling cells out of tumours.Aging Mouse Models Reveal Complex Tumor-Microenvironment Interactions in Cancer Progression.IRS2 mutations linked to invasion in pleomorphic invasive lobular carcinoma.
P2860
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P2860
Mammary-specific inactivation of E-cadherin and p53 impairs functional gland development and leads to pleomorphic invasive lobular carcinoma in mice
description
2011 nî lūn-bûn
@nan
2011 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Mammary-specific inactivation ...... sive lobular carcinoma in mice
@ast
Mammary-specific inactivation ...... sive lobular carcinoma in mice
@en
Mammary-specific inactivation ...... sive lobular carcinoma in mice
@nl
type
label
Mammary-specific inactivation ...... sive lobular carcinoma in mice
@ast
Mammary-specific inactivation ...... sive lobular carcinoma in mice
@en
Mammary-specific inactivation ...... sive lobular carcinoma in mice
@nl
prefLabel
Mammary-specific inactivation ...... sive lobular carcinoma in mice
@ast
Mammary-specific inactivation ...... sive lobular carcinoma in mice
@en
Mammary-specific inactivation ...... sive lobular carcinoma in mice
@nl
P2093
P2860
P356
P1476
Mammary-specific inactivation ...... sive lobular carcinoma in mice
@en
P2093
Eline van der Burg
Elly Mesman
Jelle Wesseling
Jos Jonkers
Marten Hornsveld
Patrick W B Derksen
Paul Krimpenfort
Tanya M Braumuller
P2860
P304
P356
10.1242/DMM.006395
P577
2011-01-31T00:00:00Z