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Herpes simplex virus latency-associated transcript is a stable intron

Herpes simplex virus latency-associated transcript is a stable intron

http://www.wikidata.org/entity/Q24564505

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Genome-wide analyses of Epstein-Barr virus reveal conserved RNA structures and a novel stable intronic sequence RNA Molecular biology of pseudorabies virus: impact on neurovirology and veterinary medicine.Spontaneous reactivation of herpes simplex virus type 1 in latently infected murine sensory ganglia MicroRNAs Expressed during Viral Infection: Biomarker Potential and Therapeutic Considerations Regulatory RNAs discovered in unexpected places Bovine Herpes Virus 1 (BHV-1) and Herpes Simplex Virus Type 1 (HSV-1) Promote Survival of Latently Infected Sensory Neurons, in Part by Inhibiting Apoptosis The molecular basis of herpes simplex virus latency A cultured affair: HSV latency and reactivation in neurons Identification of a novel latency-specific splice donor signal within the herpes simplex virus type 1 2.0-kilobase latency-associated transcript (LAT): translation inhibition of LAT open reading frames by the intron within the 2.0-kilobase LAT.Role of chromatin during herpesvirus infections.De novo synthesis of VP16 coordinates the exit from HSV latency in vivo.Herpes simplex virus type 1 (HSV-1)-induced apoptosis in human dendritic cells as a result of downregulation of cellular FLICE-inhibitory protein and reduced expression of HSV-1 antiapoptotic latency-associated transcript sequences.Herpes simplex virus 1 open reading frames O and P are not necessary for establishment of latent infection in mice.A herpes simplex virus type 1 latency-associated transcript mutant with increased virulence and reduced spontaneous reactivation Genetic studies exposing the splicing events involved in herpes simplex virus type 1 latency-associated transcript production during lytic and latent infection.Towards an understanding of the herpes simplex virus type 1 latency-reactivation cycle Epigenetic regulation of latent HSV-1 gene expression.The locus encompassing the latency-associated transcript of herpes simplex virus type 1 interferes with and delays interferon expression in productively infected neuroblastoma cells and trigeminal Ganglia of acutely infected mice.A 2.9-kilobase noncoding nuclear RNA functions in the establishment of persistent Hz-1 viral infection.Alternative splicing of the latency-related transcript of bovine herpesvirus 1 yields RNAs containing unique open reading frames The latency-associated transcript gene enhances establishment of herpes simplex virus type 1 latency in rabbits.Evidence for a bidirectional element located downstream from the herpes simplex virus type 1 latency-associated promoter that increases its activity during latency Herpes simplex virus latency-associated transcript encodes a protein which greatly enhances virus growth, can compensate for deficiencies in immediate-early gene expression, and is likely to function during reactivation from virus latency Small non-coding RNAs encoded within the herpes simplex virus type 1 latency associated transcript (LAT) cooperate with the retinoic acid inducible gene I (RIG-I) to induce beta-interferon promoter activity and promote cell survival.Numerous conserved and divergent microRNAs expressed by herpes simplex viruses 1 and 2.Analysis of protein expression from within the region encoding the 2.0-kilobase latency-associated transcript of herpes simplex virus type 1 Enhancer and long-term expression functions of herpes simplex virus type 1 latency-associated promoter are both located in the same region Three herpes simplex virus type 1 latency-associated transcript mutants with distinct and asymmetric effects on virulence in mice compared with rabbits.Region of herpes simplex virus type 1 latency-associated transcript sufficient for wild-type spontaneous reactivation promotes cell survival in tissue culture The 2.2-kilobase latency-associated transcript of herpes simplex virus type 2 does not modulate viral replication, reactivation, or establishment of latency in transgenic mice Herpes simplex virus type 1 2-kilobase latency-associated transcript intron associates with ribosomal proteins and splicing factors.Detection of the genome and transcripts of a persistent DNA virus in neuronal tissues by fluorescent in situ hybridization combined with immunostaining.Herpes simplex virus type 1 latency-associated transcript expression protects trigeminal ganglion neurons from apoptosis.Herpes simplex virus type 1 latency-associated transcription unit promotes anatomical site-dependent establishment and reactivation from latency.Herpes latency, meningitis, radiculomyelopathy and disseminated infection.A herpes simplex virus type 1 mutant expressing a baculovirus inhibitor of apoptosis gene in place of latency-associated transcript has a wild-type reactivation phenotype in the mouse.HSV-1-based vectors for gene therapy of neurological diseases and brain tumors: part I. HSV-1 structure, replication and pathogenesis.Pseudorabies virus infected porcine epithelial cell line generates a diverse set of host microRNAs and a special cluster of viral microRNAs Herpes keratitis.A gene capable of blocking apoptosis can substitute for the herpes simplex virus type 1 latency-associated transcript gene and restore wild-type reactivation levels

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P2860

Herpes simplex virus latency-associated transcript is a stable intron

http://www.wikidata.org/entity/Q24564505

description

1991 nî lūn-bûn

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1991 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

1991 թվականի փետրվարին հրատարակված գիտական հոդված

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1991年の論文

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1991年学术文章

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1991年学术文章

@zh-cn

1991年学术文章

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1991年学术文章

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1991年学术文章

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1991年學術文章

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name

Herpes simplex virus latency-associated transcript is a stable intron

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Herpes simplex virus latency-associated transcript is a stable intron

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Herpes simplex virus latency-associated transcript is a stable intron

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type

label

Herpes simplex virus latency-associated transcript is a stable intron

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Herpes simplex virus latency-associated transcript is a stable intron

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Herpes simplex virus latency-associated transcript is a stable intron

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prefLabel

Herpes simplex virus latency-associated transcript is a stable intron

@ast

Herpes simplex virus latency-associated transcript is a stable intron

@en

Herpes simplex virus latency-associated transcript is a stable intron

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Herpes simplex virus latency-associated transcript is a stable intron

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P2093

A T Dobson

http://www.w3.org/2001/XMLSchema#string

L T Feldman

http://www.w3.org/2001/XMLSchema#string

M J Farrell

http://www.w3.org/2001/XMLSchema#string

P2860

Primer-directed enzymatic amplification of DNA with a thermostable DNA polymerase

Sequencing end-labeled DNA with base-specific chemical cleavages

SV40-transformed simian cells support the replication of early SV40 mutants

Herpes simplex virus type 1 latency-associated transcripts are evidently not essential for latent infection.

Intragenic pausing and anti-sense transcription within the murine c-myc locus

Herpes simplex virus latent RNA (LAT) is not required for latent infection in the mouse.

RNA from an immediate early region of the type 1 herpes simplex virus genome is present in the trigeminal ganglia of latently infected mice

A novel member of the thyroid/steroid hormone receptor family is encoded by the opposite strand of the rat c-erbA alpha transcriptional unit

The alpha protein ICP0 does not appear to play a major role in the regulation of herpes simplex virus gene expression during infection in tissue culture.

Immediate-early regulatory gene mutants define different stages in the establishment and reactivation of herpes simplex virus latency.

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790-4

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scholarly article

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3574113

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10.1073/PNAS.88.3.790

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3

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88

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1991-02-01T00:00:00Z

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21308947

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1846963

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50899

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